Suppression of Enhancer Overactivation by a RACK7-Histone Demethylase Complex
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Acetylation Reader Proteins: Linking Acetylation Signaling to Genome Maintenance and CancerThe KDM5 family is required for activation of pro-proliferative cell cycle genes during adipocyte differentiationDifferent enhancer classes in Drosophila bind distinct architectural proteins and mediate unique chromatin interactions and 3D architectureFolate deficiency facilitates recruitment of upstream binding factor to hot spots of DNA double-strand breaks of rRNA genes and promotes its transcriptionHistone lysine demethylases in mammalian embryonic development.Chromatin and Epigenetics at the Forefront: Finding Clues among PeaksMultivalent Histone and DNA Engagement by a PHD/BRD/PWWP Triple Reader Cassette Recruits ZMYND8 to K14ac-Rich Chromatin.The Emerging Role of Epigenetics in Autoimmune Thyroid Diseases.Enhancer deregulation in cancer and other diseases.Super-Enhancer-Driven Transcriptional Dependencies in Cancer.RNF40 regulates gene expression in an epigenetic context-dependent manner.MyoD- and FoxO3-mediated hotspot interaction orchestrates super-enhancer activity during myogenic differentiation.Loss of Kdm5c Causes Spurious Transcription and Prevents the Fine-Tuning of Activity-Regulated Enhancers in Neurons.Targeting bromodomain and extraterminal proteins in breast cancer.Epigenetic Etiology of Intellectual Disability.A cryptic Tudor domain links BRWD2/PHIP to COMPASS-mediated histone H3K4 methylation.JARID1 Histone Demethylases: Emerging Targets in Cancer.Histone demethylase KDM5A regulates the ZMYND8-NuRD chromatin remodeler to promote DNA repair.Double duty: ZMYND8 in the DNA damage response and cancer.Targeting the Epigenome as a Novel Therapeutic Approach for Breast Cancer.Histone Marks in the 'Driver's Seat': Functional Roles in Steering the Transcription Cycle.An integrated analysis identifies STAT4 as a key regulator of ovarian cancer metastasis.Altered Gene-Regulatory Function of KDM5C by a Novel Mutation Associated With Autism and Intellectual Disability.MYC-driven epigenetic reprogramming favors the onset of tumorigenesis by inducing a stem cell-like state.Esrrb extinction triggers dismantling of naïve pluripotency and marks commitment to differentiationPolycomb complexes associate with enhancers and promote oncogenic transcriptional programs in cancer through multiple mechanismsThe Chromatin Reader ZMYND8 Regulates Igh Enhancers to Promote Immunoglobulin Class Switch RecombinationZMYND8 acetylation mediates HIF-dependent breast cancer progression and metastasisTET2 inhibits tumorigenesis of breast cancer cells by regulating caspase-4Positive Regulation of Transcription by Human ZMYND8 through Its Association with P-TEFb ComplexGenetics and Expression Profile of the Tubulin Gene Superfamily in Breast Cancer Subtypes and Its Relation to Taxane ResistanceKDM5 histone demethylases repress immune response via suppression of STING
P2860
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P2860
Suppression of Enhancer Overactivation by a RACK7-Histone Demethylase Complex
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Suppression of Enhancer Overactivation by a RACK7-Histone Demethylase Complex
@en
type
label
Suppression of Enhancer Overactivation by a RACK7-Histone Demethylase Complex
@en
prefLabel
Suppression of Enhancer Overactivation by a RACK7-Histone Demethylase Complex
@en
P2093
P2860
P1433
P1476
Suppression of Enhancer Overactivation by a RACK7-Histone Demethylase Complex
@en
P2093
Bowen Rong
Hongjie Shen
Lijuan Zheng
Pengyuan Yang
P2860
P304
P356
10.1016/J.CELL.2016.02.064
P407
P50
P577
2016-04-01T00:00:00Z