Progressive metaplastic and dysplastic changes in mouse pancreas induced by cyclooxygenase-2 overexpression. - Wikidata - awgldk - TriplyDB

Progressive metaplastic and dysplastic changes in mouse pancreas induced by cyclooxygenase-2 overexpression.

Progressive metaplastic and dysplastic changes in mouse pancreas induced by cyclooxygenase-2 overexpression.

http://www.wikidata.org/entity/Q36787635

about

Dinosaurs and ancient civilizations: reflections on the treatment of cancer Cyclooxygenase-2 and cancer treatment: understanding the risk should be worth the reward The anti-tumor effect of HDAC inhibition in a human pancreas cancer model is significantly improved by the simultaneous inhibition of cyclooxygenase 2 Neoplasia: the second decade.Diclofenac inhibits tumor growth in a murine model of pancreatic cancer by modulation of VEGF levels and arginase activity Inflammation to cancer: The molecular biology in the pancreas (Review).A triple combination of atorvastatin, celecoxib and tipifarnib strongly inhibits pancreatic cancer cells and xenograft pancreatic tumors Involvement of eicosanoids in the pathogenesis of pancreatic cancer: the roles of cyclooxygenase-2 and 5-lipoxygenase.Downstream of mutant KRAS, the transcription regulator YAP is essential for neoplastic progression to pancreatic ductal adenocarcinoma Risk factors for pancreatic cancer: underlying mechanisms and potential targets.COX-2 overexpression in resected pancreatic head adenocarcinomas correlates with favourable prognosis.Structure-activity studies on therapeutic potential of Thymoquinone analogs in pancreatic cancer Concurrent inhibition of NF-kappaB, cyclooxygenase-2, and epidermal growth factor receptor leads to greater anti-tumor activity in pancreatic cancer.Genetic reduction of insulin-like growth factor-1 mimics the anticancer effects of calorie restriction on cyclooxygenase-2-driven pancreatic neoplasia.Inhibitory effects of prostaglandin E2 on collagen synthesis and cell proliferation in human stellate cells from pancreatic head adenocarcinoma.The role of COX-2 in intestinal inflammation and colorectal cancer Deploying mouse models of pancreatic cancer for chemoprevention studies Smoking accelerates pancreatic cancer progression by promoting differentiation of MDSCs and inducing HB-EGF expression in macrophages.Prostaglandins in cancer cell adhesion, migration, and invasion K-Ras and cyclooxygenase-2 coactivation augments intraductal papillary mucinous neoplasm and Notch1 mimicking human pancreas lesions.Cell intrinsic role of COX-2 in pancreatic cancer development The tobacco carcinogen nitrosamine induces a differential gene expression response in tumour susceptible A/J and resistant C3H mouse lungs.Imaging heterogeneity in the mitochondrial redox state of premalignant pancreas in the pancreas-specific PTEN-null transgenic mouse model Transgenic insulin-like growth factor-1 stimulates activation of COX-2 signaling in mammary glands.Challenges and advances in mouse modeling for human pancreatic tumorigenesis and metastasis Mechanisms of parenchymal injury and signaling pathways in ectatic ducts of chronic pancreatitis: implications for pancreatic carcinogenesis.Transgenic expression of cyclooxygenase-2 (COX2) causes premature aging phenotypes in mice.The War on Cancer rages on.Ras activity in acinar cells links chronic pancreatitis and pancreatic cancer.Combined targeting of STAT3/NF-κB/COX-2/EP4 for effective management of pancreatic cancer.The growing challenge of obesity and cancer: an inflammatory issue.Pharmacogenomics of cyclooxygenases.Cyclooxygenase-2 inhibition inhibits PI3K/AKT kinase activity in epithelial ovarian cancer.Oncogenic Kras is required for both the initiation and maintenance of pancreatic cancer in mice.Loss of Activin Receptor Type 1B Accelerates Development of Intraductal Papillary Mucinous Neoplasms in Mice With Activated KRAS.Chronic inflammation initiates multiple forms of K-Ras-independent mouse pancreatic cancer in the absence of TP53.Pancreatic cancer cell lines can induce prostaglandin e2 production from human blood mononuclear cells.Prostaglandin E2 regulates pancreatic stellate cell activity via the EP4 receptor.Elevated HuR in Pancreas Promotes a Pancreatitis-like Inflammatory Microenvironment that Facilitates Tumor Development.

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P2860

Progressive metaplastic and dysplastic changes in mouse pancreas induced by cyclooxygenase-2 overexpression.

http://www.wikidata.org/entity/Q36787635

description

2008 nî lūn-bûn

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2008年の論文

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2008年论文

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name

Progressive metaplastic and dy ...... clooxygenase-2 overexpression.

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Progressive metaplastic and dy ...... clooxygenase-2 overexpression.

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Progressive metaplastic and dy ...... clooxygenase-2 overexpression.

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Progressive metaplastic and dy ...... yclooxygenase-2 overexpression

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Amy I Pavone

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Claudio J Conti

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Janet Sawicki

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Kaoru Kiguchi

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Mark J McArthur

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Russell D Klein

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Susan M Fischer

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Toru Kawamoto

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P2860

The pancreatic stellate cell: a star on the rise in pancreatic diseases

Sequences 5' of the bovine keratin 5 gene direct tissue- and cell-type-specific expression of a lacZ gene in the adult and during development

Hereditary pancreatitis and the risk of pancreatic cancer. International Hereditary Pancreatitis Study Group

Matrix metalloproteinases: effectors of development and normal physiology

Preinvasive and invasive ductal pancreatic cancer and its early detection in the mouse

Inflammatory mechanisms contributing to pancreatic cancer development.

A pharmacological study of celecoxib and gemcitabine in patients with advanced pancreatic cancer.

TGF alpha transgenic mice. A model of pancreatic cancer development.

Transgenic cyclooxygenase-2 overexpression sensitizes mouse skin for carcinogenesis.

COX-2 and beyond: Approaches to prostaglandin inhibition in human disease.

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782-796

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scholarly article

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10.1593/NEO.08330

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8

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10

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Jennifer K Colby

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23140970

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18670639

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Aizkuņģa dziedzeris

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2481568

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