Repulsion by Slit and Roundabout prevents Shotgun/E-cadherin-mediated cell adhesion during Drosophila heart tube lumen formation. - Wikidata - awgldk - TriplyDB

Repulsion by Slit and Roundabout prevents Shotgun/E-cadherin-mediated cell adhesion during Drosophila heart tube lumen formation.

Repulsion by Slit and Roundabout prevents Shotgun/E-cadherin-mediated cell adhesion during Drosophila heart tube lumen formation.

http://www.wikidata.org/entity/Q36790504

about

Tubulogenesis Vascular control of the Drosophila haematopoietic microenvironment by Slit/Robo signalling Multiplexin promotes heart but not aorta morphogenesis by polarized enhancement of slit/robo activity at the heart lumen On the mechanics of cardiac function of Drosophila embryo Distinct functions of the laminin β LN domain and collagen IV during cardiac extracellular matrix formation and stabilization of alary muscle attachments revealed by EMS mutagenesis in Drosophila.Cdc42 and formin activity control non-muscle myosin dynamics during Drosophila heart morphogenesis.A Slit/miR-218/Robo regulatory loop is required during heart tube formation in zebrafish.Ancestral vascular lumen formation via basal cell surfaces Affecting Rhomboid-3 function causes a dilated heart in adult Drosophila.Drosophila neurexin IV interacts with Roundabout and is required for repulsive midline axon guidance Frazzled/DCC facilitates cardiac cell outgrowth and attachment during Drosophila dorsal vessel formation.From cells to organs: building polarized tissue.Integrins are required for cardioblast polarisation in Drosophila.Expression of Slit and Robo genes in the developing mouse heart.Coordinate regulation of stem cell competition by Slit-Robo and JAK-STAT signaling in the Drosophila testis.A hypomorphic lsd1 allele results in heart development defects in mice Preparation of embryos for electron microscopy of the Drosophila embryonic heart tube Beta1 integrin establishes endothelial cell polarity and arteriolar lumen formation via a Par3-dependent mechanism.Slit-Robo signaling induces malignant transformation through Hakai-mediated E-cadherin degradation during colorectal epithelial cell carcinogenesis The Unc-5 Receptor Is Directly Regulated by Tinman in the Developing Drosophila Dorsal Vessel.Fine-Tuning of the Actin Cytoskeleton and Cell Adhesion During Drosophila Development by the Unconventional Guanine Nucleotide Exchange Factors Myoblast City and Sponge.Cellular Mechanisms of Drosophila Heart Morphogenesis.Tinman Regulates NetrinB in the Cardioblasts of the Drosophila Dorsal Vessel Roundabout is required in the visceral mesoderm for proper microvillus length in the hindgut epithelium.Matrix Metalloproteinases are required for membrane motility and lumenogenesis during Drosophila heart development.A genetic screen for mutations affecting gonad formation in Drosophila reveals a role for the slit/robo pathway.The first "Slit" is the deepest: the secret to a hollow heart Remodeling epithelial cell organization: transitions between front-rear and apical-basal polarity N-cadherin acts in concert with Slit1-Robo2 signaling in regulating aggregation of placode-derived cranial sensory neurons.Molecular regulation of lumen morphogenesis.Drosophila models of cardiac disease.Tubulogenesis during blood vessel formation.Vascular lumen formation.A roundabout way to cancer.A holey pursuit: lumen formation in the developing kidney.RhoA regulates actin network dynamics during apical surface emergence in multiciliated epithelial cells.Conserved signaling mechanisms in Drosophila heart development.Slit-2 facilitates interaction of P-cadherin with Robo-3 and inhibits cell migration in an oral squamous cell carcinoma cell line Regulatory subunit I-controlled protein kinase A activity is required for apical bile canalicular lumen development in hepatocytes Slit1b-Robo3 signaling and N-cadherin regulate apical process retraction in developing retinal ganglion cells.

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P2860

Repulsion by Slit and Roundabout prevents Shotgun/E-cadherin-mediated cell adhesion during Drosophila heart tube lumen formation.

http://www.wikidata.org/entity/Q36790504

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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name

Repulsion by Slit and Roundabo ...... la heart tube lumen formation.

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Repulsion by Slit and Roundabo ...... la heart tube lumen formation.

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type

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Repulsion by Slit and Roundabo ...... la heart tube lumen formation.

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Repulsion by Slit and Roundabo ...... la heart tube lumen formation.

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Repulsion by Slit and Roundabo ...... la heart tube lumen formation.

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Repulsion by Slit and Roundabo ...... la heart tube lumen formation.

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Journal of Cell Biology

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Repulsion by Slit and Roundabo ...... la heart tube lumen formation.

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P2093

Edgardo Santiago-Martínez

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Nadine H Soplop

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Rajesh Patel

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Sunita G Kramer

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P2860

Slit proteins bind Robo receptors and have an evolutionarily conserved role in repulsive axon guidance

Regulation of cadherin adhesive activity

Targeted gene expression as a means of altering cell fates and generating dominant phenotypes

Repulsive axon guidance: Abelson and Enabled play opposing roles downstream of the roundabout receptor

slit: an extracellular protein necessary for development of midline glia and commissural axon pathways contains both EGF and LRR domains

VE-cadherin: adhesion at arm's length

slit: an extracellular protein necessary for development of midline glia and commissural axon pathways contains both EGF and LRR domains.

Directed actin polymerization is the driving force for epithelial cell-cell adhesion

Dlg protein is required for junction structure, cell polarity, and proliferation control in Drosophila epithelia

Ena/VASP proteins can regulate distinct modes of actin organization at cadherin-adhesive contacts.

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241-248

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10.1083/JCB.200804120

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2

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182

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2483515

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