Nonstructural protein of parvoviruses B19 and minute virus of mice controls transcription.
about
A novel heterogeneous nuclear ribonucleoprotein-like protein interacts with NS1 of the minute virus of miceHuman parvovirus B19.The NS2 proteins of parvovirus minute virus of mice are required for efficient nuclear egress of progeny virions in mouse cells.Parvovirus replicationStructure of the NS1 Protein N-Terminal Origin Recognition/Nickase Domain from the Emerging Human BocavirusBiochemical activities of minute virus of mice nonstructural protein NS1 are modulated In vitro by the phosphorylation state of the polypeptideNuclear export factor CRM1 interacts with nonstructural proteins NS2 from parvovirus minute virus of miceDNA unwinding functions of minute virus of mice NS1 protein are modulated specifically by the lambda isoform of protein kinase C.Severe leukopenia and dysregulated erythropoiesis in SCID mice persistently infected with the parvovirus minute virus of mice.Replicative functions of minute virus of mice NS1 protein are regulated in vitro by phosphorylation through protein kinase C.Gene expression analysis of potential genes and pathways involved in the pathogenic mechanisms of parvovirus B19 in human colorectal cancer.The pathogenesis of infection with minute virus of mice depends on expression of the small nonstructural protein NS2 and on the genotype of the allotropic determinants VP1 and VP2.Genetic diversity within human erythroviruses: identification of three genotypes.Regulation of tumor necrosis factor alpha promoter by human parvovirus B19 NS1 through activation of AP-1 and AP-2.Parvovirus infection-induced cell death and cell cycle arrest.Molecular and functional analyses of a human parvovirus B19 infectious clone demonstrates essential roles for NS1, VP1, and the 11-kilodalton protein in virus replication and infectivity.Human parvovirus B19 NS1 protein aggravates liver injury in NZB/W F1 mice.VP1u phospholipase activity is critical for infectivity of full-length parvovirus B19 genomic clonesCellular microRNA miR-181b inhibits replication of mink enteritis virus by repression of non-structural protein 1 translation.Novel PKCeta is required to activate replicative functions of the major nonstructural protein NS1 of minute virus of mice.Parvovirus infection suppresses long-term repopulating hematopoietic stem cellsCharacterization of erythrovirus B19 genomes isolated in liver tissues from patients with fulminant hepatitis and biliary atresia who underwent liver transplantation.NF-Y controls transcription of the minute virus of mice P4 promoter through interaction with an unusual binding site.The NS1 polypeptide of the murine parvovirus minute virus of mice binds to DNA sequences containing the motif [ACCA]2-3Purification and characterization of the major nonstructural protein (NS-1) of Aleutian mink disease parvovirusMinute virus of mice transcriptional activator protein NS1 binds directly to the transactivation region of the viral P38 promoter in a strictly ATP-dependent manner.Upstream CREs participate in the basal activity of minute virus of mice promoter P4 and in its stimulation in ras-transformed cells.Transcriptional transactivation of parvovirus B19 promoters in nonpermissive human cells by adenovirus type 2ras oncogene-dependent activation of the P4 promoter of minute virus of mice through a proximal P4 element interacting with the Ets family of transcription factors.Transactivation of a cellular promoter by the NS1 protein of the parvovirus minute virus of mice through a putative hormone-responsive element.The cytotoxicity of the parvovirus minute virus of mice nonstructural protein NS1 is related to changes in the synthesis and phosphorylation of cell proteins.Inhibition of parvovirus minute virus of mice replication by a peptide involved in the oligomerization of nonstructural protein NS1.Roles of E4orf6 and VA I RNA in adenovirus-mediated stimulation of human parvovirus B19 DNA replication and structural gene expression.Human parvovirus B19 DNA replication induces a DNA damage response that is dispensable for cell cycle arrest at phase G2/MEx vivo-generated CD36+ erythroid progenitors are highly permissive to human parvovirus B19 replicationThe 3' untranslated region of the B19 parvovirus capsid protein mRNAs inhibits its own mRNA translation in nonpermissive cells.The cytotoxicity of the autonomous parvovirus minute virus of mice nonstructural proteins in FR3T3 rat cells depends on oncogene expressionTargeting of promoters for trans activation by a carboxy-terminal domain of the NS-1 protein of the parvovirus minute virus of miceA putative nucleoside triphosphate-binding domain in the nonstructural protein of B19 parvovirus is required for cytotoxicityIdentification and characterization of novel promoters in the genome of human papillomavirus type 18.
P2860
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P2860
Nonstructural protein of parvoviruses B19 and minute virus of mice controls transcription.
description
1990 nî lūn-bûn
@nan
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
1990年论文
@zh
1990年论文
@zh-cn
name
Nonstructural protein of parvo ...... f mice controls transcription.
@ast
Nonstructural protein of parvo ...... f mice controls transcription.
@en
type
label
Nonstructural protein of parvo ...... f mice controls transcription.
@ast
Nonstructural protein of parvo ...... f mice controls transcription.
@en
prefLabel
Nonstructural protein of parvo ...... f mice controls transcription.
@ast
Nonstructural protein of parvo ...... f mice controls transcription.
@en
P2093
P2860
P1433
P1476
Nonstructural protein of parvoviruses B19 and minute virus of mice controls transcription
@en
P2093
P2860
P304
P407
P577
1990-01-01T00:00:00Z