Alignment of U3 region sequences of mammalian type C viruses: identification of highly conserved motifs and implications for enhancer design
about
Helix-loop-helix transcriptional activators bind to a sequence in glucocorticoid response elements of retrovirus enhancersCellular transcription factor ZASC1 regulates murine leukemia virus transcriptionDEK, an autoantigen involved in a chromosomal translocation in acute myelogenous leukemia, binds to the HIV-2 enhancerSequence specificity of the core-binding factorRetroviral transcriptional regulation and embryonic stem cells: war and peaceVirological events leading to spontaneous AKR thymomasRunx1 binds as a dimeric complex to overlapping Runx1 sites within a palindromic element in the human GM-CSF enhancerMurine helix-loop-helix transcriptional activator proteins binding to the E-box motif of the Akv murine leukemia virus enhancer identified by cDNA cloningThe E47 transcription factor binds to the enhancer sequences of recombinant murine leukemia viruses and influences enhancer functionStructures of endogenous nonecotropic murine leukemia virus (MLV) long terminal repeats in wild mice: implication for evolution of MLVsEstablishment and characterization of molecular clones of porcine endogenous retroviruses replicating on human cells.Sequence-specific and/or stereospecific constraints of the U3 enhancer elements of MCF 247-W are important for pathogenicity.Tandemization of a subregion of the enhancer sequences from SRS 19-6 murine leukemia virus associated with T-lymphoid but not other leukemias.Gene expression profiling of murine T-cell lymphoblastic lymphoma identifies deregulation of S-phase initiating genes.Pathogenicity induced by feline leukemia virus, Rickard strain, subgroup A plasmid DNA (pFRA).Members of the GATA family of transcription factors bind to the U3 region of Cas-Br-E and graffi retroviruses and transactivate their expression.In vivo footprinting of the enhancer sequences in the upstream long terminal repeat of Moloney murine leukemia virus: differential binding of nuclear factors in different cell typesFeline leukemia virus long terminal repeat activates collagenase IV gene expression through AP-1Core-binding factor influences the disease specificity of Moloney murine leukemia virusIncreased induction of osteopetrosis, but unaltered lymphomagenicity, by murine leukemia virus SL3-3 after mutation of a nuclear factor 1 site in the enhancerType B leukemogenic virus has a T-cell-specific enhancer that binds AML-1.Nuclear factors that bind two regions important to transcriptional activity of the simian immunodeficiency virus long terminal repeatIkaros, a lymphoid-cell-specific transcription factor, contributes to the leukemogenic phenotype of a mink cell focus-inducing murine leukemia virusSequence analysis of porcine endogenous retrovirus long terminal repeats and identification of transcriptional regulatory regionsIdentification of homeodomain proteins, PBX1 and PREP1, involved in the transcription of murine leukemia virus.Duplication of U3 sequences in the long terminal repeat of mink cell focus-inducing viruses generates redundancies of transcription factor binding sites important for the induction of thymomasMolecular and phylogenetic analyses of a new amphotropic murine leukemia virus (MuLV-1313)Evaluation of residual promoter activity in γ-retroviral self-inactivating (SIN) vectors.Function of a unique sequence motif in the long terminal repeat of feline leukemia virus isolated from an unusual set of naturally occurring tumors.Increased lymphomagenicity and restored disease specificity of AML1 site (core) mutant SL3-3 murine leukemia virus by a second-site enhancer variant evolved in vivo.PEBP2/PEA2 represents a family of transcription factors homologous to the products of the Drosophila runt gene and the human AML1 gene.The feline leukemia virus long terminal repeat contains a potent genetic determinant of T-cell lymphomagenicityAn SL3-3 murine leukemia virus enhancer variant more pathogenic than the wild type obtained by assisted molecular evolution in vivoPreferred sequences for DNA recognition by the TAL1 helix-loop-helix proteins.Activation of the human T-cell leukemia virus type I enhancer is mediated by binding sites for Elf-1 and the pets factor.Identification of ETS domain proteins in murine T lymphocytes that interact with the Moloney murine leukemia virus enhancer.The R-U5-5' leader sequence of neurovirulent wild mouse retrovirus contains an element controlling the incubation period of neurodegenerative diseaseNuclear factor 1 activates the feline leukemia virus long terminal repeat but is posttranscriptionally down-regulated in leukemia cell linesTwo factors that bind to highly conserved sequences in mammalian type C retroviral enhancers.Characterization of a protein that binds multiple sequences in mammalian type C retrovirus enhancers.
P2860
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P2860
Alignment of U3 region sequences of mammalian type C viruses: identification of highly conserved motifs and implications for enhancer design
description
1990 nî lūn-bûn
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1990年の論文
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1990年論文
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1990年論文
@zh-hant
1990年論文
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1990年論文
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1990年論文
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name
Alignment of U3 region sequenc ...... plications for enhancer design
@ast
Alignment of U3 region sequenc ...... plications for enhancer design
@en
type
label
Alignment of U3 region sequenc ...... plications for enhancer design
@ast
Alignment of U3 region sequenc ...... plications for enhancer design
@en
prefLabel
Alignment of U3 region sequenc ...... plications for enhancer design
@ast
Alignment of U3 region sequenc ...... plications for enhancer design
@en
P2093
P2860
P1433
P1476
Alignment of U3 region sequenc ...... plications for enhancer design
@en
P2093
P2860
P304
P407
P577
1990-02-01T00:00:00Z