IL-33 exacerbates antigen-induced arthritis by activating mast cells. - Wikidata - awgldk - TriplyDB

IL-33 exacerbates antigen-induced arthritis by activating mast cells.

IL-33 exacerbates antigen-induced arthritis by activating mast cells.

http://www.wikidata.org/entity/Q36800502

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F-box protein FBXL19-mediated ubiquitination and degradation of the receptor for IL-33 limits pulmonary inflammation IL-33 is processed into mature bioactive forms by neutrophil elastase and cathepsin G The IL-1-like cytokine IL-33 is inactivated after maturation by caspase-1 Hypoxia-Inducible Factor (HIF) as a Target for Novel Therapies in Rheumatoid Arthritis Biomarkers for Bronchopulmonary Dysplasia in the Preterm Infant Interleukin-33 and Mast Cells Bridge Innate and Adaptive Immunity: From the Allergologist's Perspective The role of IL-33 and mast cells in allergy and inflammation A cytokine-centric view of the pathogenesis and treatment of autoimmune arthritis New insights into the dichotomous role of innate cytokines in gut homeostasis and inflammation IL-33 ameliorates Alzheimer's disease-like pathology and cognitive decline.MicroRNA29a regulates IL-33-mediated tissue remodelling in tendon disease Paracrine IL-33 stimulation enhances lipopolysaccharide-mediated macrophage activation A candidate gene approach identifies an IL33 genetic variant as a novel genetic risk factor for GCA Molecular adjuvant interleukin-33 enhances the antifertility effect of Lagurus lagurus zona pellucida 3 DNA vaccine administered by the mucosal route Epithelial-derived IL-33 and its receptor ST2 are dysregulated in ulcerative colitis and in experimental Th1/Th2 driven enteritis Association of increased serum IL-33 levels with clinical and laboratory characteristics of systemic lupus erythematosus in Chinese population.IL-33-induced alternatively activated macrophage attenuates the development of TNBS-induced colitis.Interleukin-33 contributes to both M1 and M2 chemokine marker expression in human macrophages.IL-33 augments substance P-induced VEGF secretion from human mast cells and is increased in psoriatic skin.Effects of interleukin-33 on cardiac fibroblast gene expression and activity.IL-33/ST2-mediated inflammation in macrophages is directly abrogated by IL-10 during rheumatoid arthritis.Genetic variant in IL33 is associated with susceptibility to rheumatoid arthritis Full Length Interleukin 33 Aggravates Radiation-Induced Skin Reaction.Role of IL-33 and its receptor in T cell-mediated autoimmune diseases Synovial fibroblasts promote the expression and granule accumulation of tryptase via interleukin-33 and its receptor ST-2 (IL1RL1)IL-17A expression is localised to both mononuclear and polymorphonuclear synovial cell infiltrates.Our perception of the mast cell from Paul Ehrlich to now.IL-33 is a crucial amplifier of innate rather than acquired immunity Inflammatory bowel disease-associated interleukin-33 is preferentially expressed in ulceration-associated myofibroblasts.Deletion of IL-33R (ST2) abrogates resistance to EAE in BALB/C mice by enhancing polarization of APC to inflammatory phenotype Interleukin-33 primes mast cells for activation by IgG immune complexes IL-33 promotes ST2-dependent lung fibrosis by the induction of alternatively activated macrophages and innate lymphoid cells in mice A novel interleukin 33/ST2 signaling regulates inflammatory response in human corneal epithelium.Associations between genetic polymorphisms in IL-33, IL1R1 and risk for inflammatory bowel disease.Amplification of cytokine production through synergistic activation of NFAT and AP-1 following stimulation of mast cells with antigen and IL-33 Hypoxia-inducible factor-1α and interleukin 33 form a regulatory circuit to perpetuate the inflammation in rheumatoid arthritis.The immune-modulating cytokine and endogenous Alarmin interleukin-33 is upregulated in skin exposed to inflammatory UVB radiation.Novel cytokine signaling pathways in inflammatory bowel disease: insight into the dichotomous functions of IL-33 during chronic intestinal inflammation Measurement of interleukin-33 (IL-33) and IL-33 receptors (sST2 and ST2L) in patients with rheumatoid arthritis.Cytokines in autoimmunity: role in induction, regulation, and treatment.

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P2860

IL-33 exacerbates antigen-induced arthritis by activating mast cells.

http://www.wikidata.org/entity/Q36800502

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

IL-33 exacerbates antigen-induced arthritis by activating mast cells.

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IL-33 exacerbates antigen-induced arthritis by activating mast cells.

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type

label

IL-33 exacerbates antigen-induced arthritis by activating mast cells.

@ast

IL-33 exacerbates antigen-induced arthritis by activating mast cells.

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prefLabel

IL-33 exacerbates antigen-induced arthritis by activating mast cells.

@ast

IL-33 exacerbates antigen-induced arthritis by activating mast cells.

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PNAS.0801898105

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Proceedings of the National Academy of Sciences of the United States of America

P1476

IL-33 exacerbates antigen-induced arthritis by activating mast cells.

@en

P2093

Alasdair R Fraser

http://www.w3.org/2001/XMLSchema#string

Damo Xu

http://www.w3.org/2001/XMLSchema#string

Hui-Rong Jiang

http://www.w3.org/2001/XMLSchema#string

Mariola Kurowska-Stolarska

http://www.w3.org/2001/XMLSchema#string

Nick Pitman

http://www.w3.org/2001/XMLSchema#string

Peter Kewin

http://www.w3.org/2001/XMLSchema#string

Rong Mu

http://www.w3.org/2001/XMLSchema#string

Yubin Li

http://www.w3.org/2001/XMLSchema#string

P2860

IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines

T1/ST2 is preferentially expressed on murine Th2 cells, independent of interleukin 4, interleukin 5, and interleukin 10, and important for Th2 effector function

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

IL-1 receptor accessory protein and ST2 comprise the IL-33 receptor complex

Cytokines in the pathogenesis of rheumatoid arthritis

IL-33, a potent inducer of adaptive immunity to intestinal nematodes

IL-1 receptor accessory protein is essential for IL-33-induced activation of T lymphocytes and mast cells

IL-33 and ST2 comprise a critical biomechanically induced and cardioprotective signaling system

Interleukins 1beta and 6 but not transforming growth factor-beta are essential for the differentiation of interleukin 17-producing human T helper cells

Role of cytokines in rheumatoid arthritis

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10913-10918

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scholarly article

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10.1073/PNAS.0801898105

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31

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105

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Andrew N. J. McKenzie

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2008-07-30T00:00:00Z

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23138101

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18667700

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P932

2491487

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