The Cpx stress response system potentiates the fitness and virulence of uropathogenic Escherichia coli.
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Environmental Regulation of Yersinia PathophysiologyProkaryotic 2-component systems and the OmpR/PhoB superfamilyAn Overview of Two-Component Signal Transduction Systems Implicated in Extra-Intestinal Pathogenic E. coli InfectionsReciprocal regulation of resistance-nodulation-division efflux systems and the Cpx two-component system in Vibrio cholerae.Recent advances in adherence and invasion of pathogenic Escherichia coliPhenotypic heterogeneity enables uropathogenic Escherichia coli to evade killing by antibiotics and serum complement.Dysregulation of Escherichia coli α-hemolysin expression alters the course of acute and persistent urinary tract infectionDynamic interaction between the CpxA sensor kinase and the periplasmic accessory protein CpxP mediates signal recognition in E. coli.Signature-tagged mutagenesis and co-infection studies demonstrate the importance of P fimbriae in a murine model of urinary tract infectionStrengths and Limitations of Model Systems for the Study of Urinary Tract Infections and Related Pathologies.Drug and Vaccine Development for the Treatment and Prevention of Urinary Tract Infections.Similarly Lethal Strains of Extraintestinal Pathogenic Escherichia coli Trigger Markedly Diverse Host Responses in a Zebrafish Model of Sepsis.DamX Controls Reversible Cell Morphology Switching in Uropathogenic Escherichia coliFrom physiology to pharmacy: developments in the pathogenesis and treatment of recurrent urinary tract infectionsSystematic Analysis of Two-Component Systems in Citrobacter rodentium Reveals Positive and Negative Roles in Virulence.The Two-Component System CpxRA Negatively Regulates the Locus of Enterocyte Effacement of Enterohemorrhagic Escherichia coli Involving σ(32) and Lon proteaseBoth MisR (CpxR) and MisS (CpxA) Are Required for Neisseria gonorrhoeae Infection in a Murine Model of Lower Genital Tract Infection.The CpxRA two-component system is essential for Citrobacter rodentium virulence.Innate immune evasion of Escherichia coli clinical strains from orthopedic implant infections.Role of the CpxAR two-component signal transduction system in control of fosfomycin resistance and carbon substrate uptake.Evaluation of CpxRA as a Therapeutic Target for Uropathogenic Escherichia coli Infections.The CpxA/CpxR Two-Component System Affects Biofilm Formation and Virulence in Actinobacillus pleuropneumoniae.Modification in hydrophobic packing of HAMP domain induces a destabilization of the auto-phosphorylation site in the histidine kinase CpxA.Stress-related genes promote Edwardsiella ictaluri pathogenesis.Critical role for a promoter discriminator in RpoS control of virulence in Edwardsiella piscicida
P2860
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P2860
The Cpx stress response system potentiates the fitness and virulence of uropathogenic Escherichia coli.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
The Cpx stress response system ...... ropathogenic Escherichia coli.
@en
type
label
The Cpx stress response system ...... ropathogenic Escherichia coli.
@en
prefLabel
The Cpx stress response system ...... ropathogenic Escherichia coli.
@en
P2093
P2860
P356
P1476
The Cpx stress response system ...... uropathogenic Escherichia coli
@en
P2093
Bijaya K Dhakal
Elizabeth M Ott
Irina Debnath
J Paul Norton
Richard R Kulesus
P2860
P304
P356
10.1128/IAI.01213-12
P407
P577
2013-02-19T00:00:00Z