Return to the fetal gene program protects the stressed heart: a strong hypothesis.
about
Feeding a protein-restricted diet during pregnancy induces altered epigenetic regulation of peroxisomal proliferator-activated receptor-α in the heart of the offspringCellular and molecular basis of deiodinase-regulated thyroid hormone signalingAnimal Models and "Omics" Technologies for Identification of Novel Biomarkers and Drug Targets to Prevent Heart FailureCardiac energy metabolic alterations in pressure overload-induced left and right heart failure (2013 Grover Conference Series)Epigenetic regulation by histone demethylases in hypoxiaSystems proteomics of healthy and diseased chromatinGenome-Wide Gene Expression Analysis Shows AKAP13-Mediated PKD1 Signaling Regulates the Transcriptional Response to Cardiac HypertrophyHypoxic regulation of hand1 controls the fetal-neonatal switch in cardiac metabolismFetal-adult cardiac transcriptome analysis in rats with contrasting left ventricular mass reveals new candidates for cardiac hypertrophySRC-2 coactivator deficiency decreases functional reserve in response to pressure overload of mouse heartHIF-1alpha Deficiency Attenuates the Cardiomyogenesis of Mouse Embryonic Stem CellsFibroblast growth factor receptor 1 signaling in adult cardiomyocytes increases contractility and results in a hypertrophic cardiomyopathyTargeting myocardial substrate metabolism in heart failure: potential for new therapiesEmerging concepts in the molecular basis of pulmonary arterial hypertension: part I: metabolic plasticity and mitochondrial dynamics in the pulmonary circulation and right ventricle in pulmonary arterial hypertensionMyosin light chain phosphorylation is critical for adaptation to cardiac stress.Diabetic cardiomyopathy: signaling defects and therapeutic approachesNoninvasive Detection of Early Metabolic Left Ventricular Remodeling in Systemic Hypertension.Sequencing of mRNA identifies re-expression of fetal splice variants in cardiac hypertrophy.Engineered Human Muscle Tissue from Skeletal Muscle Derived Stem Cells and Induced Pluripotent Stem Cell Derived Cardiac CellsEmbryonic domains of the aorta derived from diverse origins exhibit distinct properties that converge into a common phenotype in the adultThe right ventricle in pulmonary arterial hypertension: disorders of metabolism, angiogenesis and adrenergic signaling in right ventricular failure.Comparative analysis of mRNA isoform expression in cardiac hypertrophy and development reveals multiple post-transcriptional regulatory modules.Myocardial triglyceride content at 3 T cardiovascular magnetic resonance and left ventricular systolic function: a cross-sectional study in patients hospitalized with acute heart failure.Construction of Defined Human Engineered Cardiac Tissues to Study Mechanisms of Cardiac Cell TherapyReproducible ion-current-based approach for 24-plex comparison of the tissue proteomes of hibernating versus normal myocardium in swine modelsc-Abl tyrosine kinase regulates cardiac growth and development.Common microRNA signatures in cardiac hypertrophic and atrophic remodeling induced by changes in hemodynamic load.Early beneficial effects of bone marrow-derived mesenchymal stem cells overexpressing Akt on cardiac metabolism after myocardial infarction.Compensatory hypertrophy induced by ventricular cardiomyocyte-specific COX-2 expression in miceA novel role for the fifth component of complement (C5) in cardiac physiology.The study of fetal rat model of intra-amniotic isoproterenol injection induced heart dysfunction and phenotypic switch of contractile proteins.Genome-wide mapping of Sox6 binding sites in skeletal muscle reveals both direct and indirect regulation of muscle terminal differentiation by Sox6.Epigenetic coordination of embryonic heart transcription by dynamically regulated long noncoding RNAsA randomized pilot trial of remote ischemic preconditioning in heart failure with reduced ejection fraction.Heterogeneous myocyte enhancer factor-2 (Mef2) activation in myocytes predicts focal scarring in hypertrophic cardiomyopathy.Talin1 has unique expression versus talin 2 in the heart and modifies the hypertrophic response to pressure overload.Small engine, big power: microRNAs as regulators of cardiac diseases and regeneration.Roles and targets of class I and IIa histone deacetylases in cardiac hypertrophy.The cardiac ventricular 5-HT4 receptor is functional in late foetal development and is reactivated in heart failure.Chromodomain helicase binding protein 8 (Chd8) is a novel A-kinase anchoring protein expressed during rat cardiac development.
P2860
Q24635109-681AF36A-92D3-46DF-B86F-F78C2520AA2CQ24648381-D497DB61-2B0B-42B8-86FC-95708BC317F9Q26799838-D646B616-7026-422C-A1B9-CEE8330C29FEQ26825047-8D2CBD3C-A9E2-4CAD-AE39-991E027D594DQ26825704-29F1953D-15B3-4D0D-8A10-61BB41A7BCA1Q27014028-8F1313B8-4584-46AA-AA08-FA3F0496623FQ27302945-1060B0A6-61F0-4DEE-AAC3-CB434A17BCF3Q27316790-91C0B6F0-5639-40F9-A034-3266052BBBE5Q27316925-C8E58B49-8A8F-4AFD-9113-92710DB40825Q27321625-ACD44DC2-02A6-4415-8151-69B7AFAC9801Q27335803-EC7104FB-3CA0-40D6-9FC3-98BDCE030616Q27342077-8D0EEDBB-B2B9-4B7B-A264-6A16E3FBBB11Q27694733-4F097910-4972-47FE-8B5F-ACB8DD61FCDBQ28083183-B763D581-613F-45CE-9E1C-8C8158A81606Q28277846-DF205B94-86DC-4D3B-858B-FDD146A254DAQ28395852-D615461A-5192-497C-9BE9-D5EE52DE0D28Q30278045-043F5D07-9B03-40C2-93FA-02F2D73003D9Q30411046-B8B575EF-F893-426A-A05E-3466E6C716CFQ30575995-12E03FD7-D8BF-4052-9DB2-CCCDD0F28B8BQ30578898-68D037A2-D221-4A70-BF6E-5506893E6F06Q30585014-149AB0CB-4215-4ADF-A7E0-4CF7A2DAB7D5Q31027656-D6DDA3AC-6A61-4021-B660-37E6C3E44EE8Q31043315-AB29FAC8-EDA9-4A07-8274-0A01914CD0BDQ31056955-CC2A8463-6643-4725-9F96-C68B3F859845Q33588975-74CB1F02-041A-4959-9A2C-D9CE282EB204Q33667001-E28C4C2C-1C0D-4E60-A220-1024212FDB0EQ33769163-A2AB2D46-09BB-4D9E-BD47-5D3EC58A4D87Q33862492-0978948C-E5D6-491B-832E-6A0C64990D17Q33905727-4AE796BE-8E9B-4B78-A6E3-D127E9649B9EQ33988617-95802B11-A1C8-4C4B-B9B0-AFD6F86415B4Q34022447-64D8A3C0-EA99-4C5B-A98E-FF1E9A2CD278Q34045389-267B73FE-EC04-488E-BD3D-1EC9CF97D2EDQ34082965-58799655-B60D-4648-8FCC-C61F89FFDC10Q34121775-D08E26C2-F954-46D1-A04D-7A232D25C9A3Q34241524-E2CD3306-77B3-45DD-AB9E-11CF2759E55FQ34319557-328DA7E8-F446-4489-A740-F13A5A803932Q34358827-529A4809-518B-4EEA-9671-B42921C62965Q34383040-B28591ED-512E-49CA-9B0B-BD9D0CE5DF39Q34429191-82B7A2D9-1452-428D-9461-5B72CA3CB48DQ34447042-5AD70677-A587-4A82-B365-53FEB2AC62C3
P2860
Return to the fetal gene program protects the stressed heart: a strong hypothesis.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Return to the fetal gene program protects the stressed heart: a strong hypothesis.
@en
type
label
Return to the fetal gene program protects the stressed heart: a strong hypothesis.
@en
prefLabel
Return to the fetal gene program protects the stressed heart: a strong hypothesis.
@en
P2093
P1476
Return to the fetal gene program protects the stressed heart: a strong hypothesis.
@en
P2093
Christos Kassiotis
Heinrich Taegtmeyer
Mitra Rajabi
Peter Razeghi
P2888
P304
P356
10.1007/S10741-007-9034-1
P577
2007-12-01T00:00:00Z