Loss of the Kv1.1 potassium channel promotes pathologic sharp waves and high frequency oscillations in in vitro hippocampal slices.
about
Molecular pathophysiology and pharmacology of the voltage-sensing module of neuronal ion channelsRoles of gap junctions, connexins, and pannexins in epilepsyK(+) channelepsy: progress in the neurobiology of potassium channels and epilepsy.In vivo ketogenic diet treatment attenuates pathologic sharp waves and high frequency oscillations in in vitro hippocampal slices from epileptic Kv 1.1α knockout mice.Physiological and pathological functions of mechanosensitive ion channels.Voltage-gated potassium channels at the crossroads of neuronal function, ischemic tolerance, and neurodegeneration.The serotonin 5-HT2C receptor and the non-addictive nature of classic hallucinogens.Revealing neuronal function through microelectrode array recordingsDifferent mechanisms of ripple-like oscillations in the human epileptic subiculumKetone bodies mediate antiseizure effects through mitochondrial permeability transition.Orexin Receptor Antagonism Improves Sleep and Reduces Seizures in Kcna1-null Mice.Targeting deficiencies in mitochondrial respiratory complex I and functional uncoupling exerts anti-seizure effects in a genetic model of temporal lobe epilepsy and in a model of acute temporal lobe seizures.Potassium Channels in Epilepsy.Bexarotene reduces network excitability in models of Alzheimer's disease and epilepsy.Alteration of Neuronal Excitability and Short-Term Synaptic Plasticity in the Prefrontal Cortex of a Mouse Model of Mental Illness.Neocortical pathological high-frequency oscillations are associated with frequency-dependent alterations in functional network topology.A single conserved basic residue in the potassium channel filter region controls KCNQ1 insensitivity toward scorpion toxins.Ion Channels in Genetic Epilepsy: From Genes and Mechanisms to Disease-Targeted Therapies.The Continuing Failure of Bexarotene in Alzheimer's Disease Mice.Respiratory dysfunction progresses with age in Kcna1-null mice, a model of sudden unexpected death in epilepsy.BAD and KATP channels regulate neuron excitability and epileptiform activity.Regulation of brain PPARgamma2 contributes to ketogenic diet anti-seizure efficacy.
P2860
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P2860
Loss of the Kv1.1 potassium channel promotes pathologic sharp waves and high frequency oscillations in in vitro hippocampal slices.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Loss of the Kv1.1 potassium ch ...... n in vitro hippocampal slices.
@en
type
label
Loss of the Kv1.1 potassium ch ...... n in vitro hippocampal slices.
@en
prefLabel
Loss of the Kv1.1 potassium ch ...... n in vitro hippocampal slices.
@en
P2093
P2860
P1476
Loss of the Kv1.1 potassium ch ...... n in vitro hippocampal slices.
@en
P2093
Do Young Kim
Jong M Rho
Kaeli K Samson
Kristina A Simeone
Timothy A Simeone
P2860
P356
10.1016/J.NBD.2013.02.009
P577
2013-03-04T00:00:00Z