The pathogenic Th17 cell response to major schistosome egg antigen is sequentially dependent on IL-23 and IL-1β
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The Differentiation of CD4(+) T-Helper Cell Subsets in the Context of Helminth Parasite InfectionInhibition of Granulomatous Inflammation and Prophylactic Treatment of Schistosomiasis with a Combination of Edelfosine and PraziquantelCD209a expression on dendritic cells is critical for the development of pathogenic Th17 cell responses in murine schistosomiasisIncreased IL-17, a Pathogenic Link between Hepatosplenic Schistosomiasis and Amyotrophic Lateral Sclerosis: A Hypothesis.Spatial and temporal transcriptomics of Schistosoma japonicum-induced hepatic granuloma formation reveals novel roles for neutrophils.Inflammasome Activity in Non-Microbial Lung Inflammation.The Dynamics of Treg/Th17 and the Imbalance of Treg/Th17 in Clonorchis sinensis-Infected MiceConcurrent bacterial stimulation alters the function of helminth-activated dendritic cells, resulting in IL-17 induction.Cell-cell interaction with APC, not IL-23, is required for naive CD4 cells to acquire pathogenicity during Th17 lineage commitment.Minor genomic differences between related B6 and B10 mice affect severity of schistosome infection by governing the mode of dendritic cell activation.Expression of Toll-Like Receptors 2 and 4 and Related Cytokines in Patients with Hepatic Cystic and Alveolar Echinococcosis.Induction and regulation of pathogenic Th17 cell responses in schistosomiasis.Contribution of myeloid cell subsets to liver fibrosis in parasite infection.Th2 responses in schistosomiasis.Serum osteopontin is a biomarker of severe fibrosis and portal hypertension in human and murine schistosomiasis mansoni.Ellagic acid reduces murine schistosomiasis mansoni immunopathology via up-regulation of IL-10 and down-modulation of pro-inflammatory cytokines production.Lack of IL-17 signaling decreases liver fibrosis in murine schistosomiasis japonica.Dendritic cell expression of the C-type lectin receptor CD209a: A novel innate parasite-sensing mechanism inducing Th17 cells that drive severe immunopathology in murine schistosome infection.Immunity and Fibrogenesis: The Role of Th17/IL-17 Axis in HBV and HCV-induced Chronic Hepatitis and Progression to Cirrhosis.IL-17 neutralization significantly ameliorates hepatic granulomatous inflammation and liver damage in Schistosoma japonicum infected mice.CD209a Synergizes with Dectin-2 and Mincle to Drive Severe Th17 Cell-Mediated Schistosome Egg-Induced Immunopathology.Low IL-6, IL-10, and TNF-α and High IL-13 Cytokine Levels Are Associated with Severe Hepatic Fibrosis in Schistosoma mansoni Chronically Exposed Individuals.
P2860
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P2860
The pathogenic Th17 cell response to major schistosome egg antigen is sequentially dependent on IL-23 and IL-1β
description
2011 nî lūn-bûn
@nan
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
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2011年论文
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2011年论文
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name
The pathogenic Th17 cell respo ...... y dependent on IL-23 and IL-1β
@en
type
label
The pathogenic Th17 cell respo ...... y dependent on IL-23 and IL-1β
@en
prefLabel
The pathogenic Th17 cell respo ...... y dependent on IL-23 and IL-1β
@en
P2093
P2860
P356
P1476
The pathogenic Th17 cell respo ...... y dependent on IL-23 and IL-1β
@en
P2093
Arlene H Sharpe
Bridget M Larkin
Eduardo Finger
Krzysztof W Lasocki
Laura I Rutitzky
Mara G Shainheit
Miguel J Stadecker
Patrick M Smith
P2860
P304
P356
10.4049/JIMMUNOL.1101445
P407
P577
2011-10-14T00:00:00Z