The pathogenic Th17 cell response to major schistosome egg antigen is sequentially dependent on IL-23 and IL-1β - Wikidata - awgldk - TriplyDB

The pathogenic Th17 cell response to major schistosome egg antigen is sequentially dependent on IL-23 and IL-1β

The pathogenic Th17 cell response to major schistosome egg antigen is sequentially dependent on IL-23 and IL-1β

http://www.wikidata.org/entity/Q36843026

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The Differentiation of CD4(+) T-Helper Cell Subsets in the Context of Helminth Parasite Infection Inhibition of Granulomatous Inflammation and Prophylactic Treatment of Schistosomiasis with a Combination of Edelfosine and Praziquantel CD209a expression on dendritic cells is critical for the development of pathogenic Th17 cell responses in murine schistosomiasis Increased IL-17, a Pathogenic Link between Hepatosplenic Schistosomiasis and Amyotrophic Lateral Sclerosis: A Hypothesis.Spatial and temporal transcriptomics of Schistosoma japonicum-induced hepatic granuloma formation reveals novel roles for neutrophils.Inflammasome Activity in Non-Microbial Lung Inflammation.The Dynamics of Treg/Th17 and the Imbalance of Treg/Th17 in Clonorchis sinensis-Infected Mice Concurrent bacterial stimulation alters the function of helminth-activated dendritic cells, resulting in IL-17 induction.Cell-cell interaction with APC, not IL-23, is required for naive CD4 cells to acquire pathogenicity during Th17 lineage commitment.Minor genomic differences between related B6 and B10 mice affect severity of schistosome infection by governing the mode of dendritic cell activation.Expression of Toll-Like Receptors 2 and 4 and Related Cytokines in Patients with Hepatic Cystic and Alveolar Echinococcosis.Induction and regulation of pathogenic Th17 cell responses in schistosomiasis.Contribution of myeloid cell subsets to liver fibrosis in parasite infection.Th2 responses in schistosomiasis.Serum osteopontin is a biomarker of severe fibrosis and portal hypertension in human and murine schistosomiasis mansoni.Ellagic acid reduces murine schistosomiasis mansoni immunopathology via up-regulation of IL-10 and down-modulation of pro-inflammatory cytokines production.Lack of IL-17 signaling decreases liver fibrosis in murine schistosomiasis japonica.Dendritic cell expression of the C-type lectin receptor CD209a: A novel innate parasite-sensing mechanism inducing Th17 cells that drive severe immunopathology in murine schistosome infection.Immunity and Fibrogenesis: The Role of Th17/IL-17 Axis in HBV and HCV-induced Chronic Hepatitis and Progression to Cirrhosis.IL-17 neutralization significantly ameliorates hepatic granulomatous inflammation and liver damage in Schistosoma japonicum infected mice.CD209a Synergizes with Dectin-2 and Mincle to Drive Severe Th17 Cell-Mediated Schistosome Egg-Induced Immunopathology.Low IL-6, IL-10, and TNF-α and High IL-13 Cytokine Levels Are Associated with Severe Hepatic Fibrosis in Schistosoma mansoni Chronically Exposed Individuals.

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P2860

The pathogenic Th17 cell response to major schistosome egg antigen is sequentially dependent on IL-23 and IL-1β

http://www.wikidata.org/entity/Q36843026

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

The pathogenic Th17 cell respo ...... y dependent on IL-23 and IL-1β

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The pathogenic Th17 cell respo ...... y dependent on IL-23 and IL-1β

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JIMMUNOL.1101445

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Journal of Immunology

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The pathogenic Th17 cell respo ...... y dependent on IL-23 and IL-1β

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Arlene H Sharpe

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Bridget M Larkin

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Eduardo Finger

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Krzysztof W Lasocki

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Laura I Rutitzky

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Mara G Shainheit

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Miguel J Stadecker

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Patrick M Smith

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P2860

Stimulation of the intracellular bacterial sensor NOD2 programs dendritic cells to promote interleukin-17 production in human memory T cells

Interleukin-1 in the pathogenesis and treatment of inflammatory diseases

Generation of pathogenic T(H)17 cells in the absence of TGF-β signalling

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

IL-17 and Th17 Cells

STAT3 and NF-kappaB signal pathway is required for IL-23-mediated IL-17 production in spontaneous arthritis animal model IL-1 receptor antagonist-deficient mice

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

Interleukin-12 and the regulation of innate resistance and adaptive immunity

Interleukins 1beta and 6 but not transforming growth factor-beta are essential for the differentiation of interleukin 17-producing human T helper cells

Immunological and inflammatory functions of the interleukin-1 family

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5328-5335

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10.4049/JIMMUNOL.1101445

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10

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187

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Charles A. Dinarello

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2011-10-14T00:00:00Z

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51720163

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22003203

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