Absence of donor Th17 leads to augmented Th1 differentiation and exacerbated acute graft-versus-host disease. - Wikidata - awgldk - TriplyDB

Absence of donor Th17 leads to augmented Th1 differentiation and exacerbated acute graft-versus-host disease.

Absence of donor Th17 leads to augmented Th1 differentiation and exacerbated acute graft-versus-host disease.

http://www.wikidata.org/entity/Q36843812

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Therapeutics for Graft-versus-Host Disease: From Conventional Therapies to Novel Virotherapeutic Strategies Disturbed T Cell Signaling and Altered Th17 and Regulatory T Cell Subsets in the Pathogenesis of Systemic Lupus Erythematosus Inflammatory cytokines in systemic lupus erythematosus The Role of Intestinal Microbiota in Acute Graft-versus-Host Disease The Biology of Chronic Graft-versus-Host Disease: A Task Force Report from the National Institutes of Health Consensus Development Project on Criteria for Clinical Trials in Chronic Graft-versus-Host Disease IL-17A controls IL-17F production and maintains blood neutrophil counts in mice.Current data on IL-17 and Th17 cells and implications for graft versus host disease.Interleukin 17 is not required for autoimmune-mediated pathologic damage during chronic graft-versus-host disease Kinetics of IFN-γ and IL-17 Production by CD4 and CD8 T Cells during Acute Graft-versus-Host Disease.Separating graft-versus-leukemia from graft-versus-host disease in allogeneic hematopoietic stem cell transplantation.An activated Th17-prone T cell subset involved in chronic graft-versus-host disease sensitive to pharmacological inhibition.SOCS3 regulates graft-versus-host disease.IL-17 genetic and immunophenotypic evaluation in chronic graft-versus-host disease.Advances in graft-versus-host disease biology and therapy.Constitutively active Stat5b in CD4+ T cells inhibits graft-versus-host disease lethality associated with increased regulatory T-cell potency and decreased T effector cell responses.A genetic variant in the IL-17 promoter is functionally associated with acute graft-versus-host disease after unrelated bone marrow transplantation.Differential modulation by IL-17A of Cholangitis versus Colitis in IL-2Rα deleted mice.Induction of chimerism permits low-dose islet grafts in the liver or pancreas to reverse refractory autoimmune diabetes.Modeling human gastrointestinal inflammatory diseases using microphysiological culture systems.IL-17RA signaling reduces inflammation and mortality during Trypanosoma cruzi infection by recruiting suppressive IL-10-producing neutrophils Development of a T(H)17 immune response during the induction of murine syngeneic graft-versus-host disease Nuclear factor-kappaB inducing kinase is required for graft-versus-host disease.Immune regulatory cells in umbilical cord blood and their potential roles in transplantation tolerance.Th17/Treg ratio in human graft-versus-host disease.Coevolution of TH1, TH2, and TH17 responses during repeated pulmonary exposure to Aspergillus fumigatus conidia.Decreased levels of circulating IL17-producing CD161+CCR6+ T cells are associated with graft-versus-host disease after allogeneic stem cell transplantation.Altered effector CD4+ T cell function in IL-21R-/- CD4+ T cell-mediated graft-versus-host disease Blockade of osteopontin reduces alloreactive CD8+ T cell-mediated graft-versus-host disease Sequential activation of inflammatory signaling pathways during graft-versus-host disease (GVHD): early role for STAT1 and STAT3.Effector CD4+ T cells, the cytokines they generate, and GVHD: something old and something new.Effects of Pre-conditioning Dose on the Immune Kinetics and Cytokine Production in the Leukocytes Infiltrating GVHD Tissues after MHC-matched Transplantation.B7H1/CD80 interaction augments PD-1-dependent T cell apoptosis and ameliorates graft-versus-host disease A higher risk of acute rejection of human kidney allografts can be predicted from the level of CD45RC expressed by the recipients' CD8 T cells.IL-17A mediates early post-transplant lesions after heterotopic trachea allotransplantation in Mice IL-17 can promote tumor growth through an IL-6-Stat3 signaling pathway.HDAC inhibition and graft versus host disease IL-17A-dependent CD4+CD25+ regulatory T cells promote immune privilege of corneal allografts CD4+ T cells mediate the protective effect of the recombinant Asp f3-based anti-aspergillosis vaccine.IL-17 promotes immune privilege of corneal allografts Interleukin-17A promotes early but attenuates established disease in crescentic glomerulonephritis in mice.

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P2860

Absence of donor Th17 leads to augmented Th1 differentiation and exacerbated acute graft-versus-host disease.

http://www.wikidata.org/entity/Q36843812

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Chia-Lei Lin

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Chunyan Zhang

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Defu Zeng

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Dongchang Zhao

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Fouad Kandeel

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Ivan Todorov

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Tangsheng Yi

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Thomas LeBon

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Ying Chen

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P2860

Interleukin-23 drives innate and T cell-mediated intestinal inflammation

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The orphan nuclear receptor RORgammat directs the differentiation program of proinflammatory IL-17+ T helper cells

Interleukin-23 promotes a distinct CD4 T cell activation state characterized by the production of interleukin-17

Transforming growth factor-beta induces development of the T(H)17 lineage

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

The lineage decisions of helper T cells

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