Role of spatial dispersion of repolarization in inherited and acquired sudden cardiac death syndromes. - Wikidata - awgldk - TriplyDB

Role of spatial dispersion of repolarization in inherited and acquired sudden cardiac death syndromes.

Role of spatial dispersion of repolarization in inherited and acquired sudden cardiac death syndromes.

http://www.wikidata.org/entity/Q36857275

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Repolarization Heterogeneity: Beyond the QT Interval CaV1.2 signaling complexes in the heart Ion Channels in the Heart Determinants of incomplete penetrance and variable expressivity in heritable cardiac arrhythmia syndromes Post-translational modifications of the cardiac Na channel: contribution of CaMKII-dependent phosphorylation to acquired arrhythmias Circadian rhythms govern cardiac repolarization and arrhythmogenesis Determinants of beat-to-beat variability of repolarization duration in the canine ventricular myocyte: a computational analysis Mutations in Danish patients with long QT syndrome and the identification of a large founder family with p.F29L in KCNH2.Torsades de pointes during complete atrioventricular block: Genetic factors and electrocardiogram correlates.Isoproterenol disperses distribution of NADPH oxidase, MMP-9, and pPKCepsilon in the heart, which are mitigated by endothelin receptor antagonist CPU0213 Ionic mechanisms for electrical heterogeneity between rabbit Purkinje fiber and ventricular cells.Impact of psychotropic drugs on QT interval dispersion in adult patients.Transmural dispersion of repolarization in failing and nonfailing human ventricle Luminal Ca(2+) content regulates intracellular Ca(2+) release in subepicardial myocytes of intact beating mouse hearts: effect of exogenous buffers Optical mapping of electrical heterogeneities in the heart during global ischemia.Processing and analysis of cardiac optical mapping data obtained with potentiometric dyes Cellular basis for arrhythmogenesis in an experimental model of the SQT1 form of the short QT syndrome Deciphering Arrhythmia Mechanisms - Tools of the Trade Mapping of reentrant spontaneous polymorphic ventricular tachycardia in a Scn5a+/- mouse model.A novel electrocardiographic index for the diagnosis of diastolic dysfunction Arrhythmogenic substrate in hearts of rats with monocrotaline-induced pulmonary hypertension and right ventricular hypertrophy.High yield purification of full-length functional hERG K+ channels produced in Saccharomyces cerevisiae.Resolving the M-cell debate: why and how.Prolonged Tpeak-to-tend interval on the resting ECG is associated with increased risk of sudden cardiac death.How do sex hormones modify arrhythmogenesis in long QT syndrome? Sex hormone effects on arrhythmogenic substrate and triggered activity.Estradiol promotes sudden cardiac death in transgenic long QT type 2 rabbits while progesterone is protective Cardiac repolarization instability during psychological stress in patients with ventricular arrhythmias.Kruppel-like factors in muscle health and disease.Panoramic optical mapping shows wavebreak at a consistent anatomical site at the onset of ventricular fibrillation.Conduction remodeling in human end-stage nonischemic left ventricular cardiomyopathy.Mechanisms of ventricular arrhythmias: a dynamical systems-based perspective.Cardiac arrhythmia mechanisms in rats with heart failure induced by pulmonary hypertension.Diastolic electromechanical coupling: association of the ECG T-peak to T-end interval with echocardiographic markers of diastolic dysfunction.Mechanisms of cardiac arrhythmias and sudden death in transgenic rabbits with long QT syndrome.Ca2+/calmodulin-dependent protein kinase II-dependent remodeling of Ca2+ current in pressure overload heart failure.Genotype- and phenotype-guided management of congenital long QT syndrome.The genetic and clinical features of cardiac channelopathies.The short QT syndrome.Supplemental studies for cardiovascular risk assessment in safety pharmacology: a critical overview.Potassium-channel mutations and cardiac arrhythmias--diagnosis and therapy.

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P2860

Role of spatial dispersion of repolarization in inherited and acquired sudden cardiac death syndromes.

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P2860

Mutations in Kir2.1 cause the developmental and episodic electrical phenotypes of Andersen's syndrome

Ankyrin-B mutation causes type 4 long-QT cardiac arrhythmia and sudden cardiac death

Mutant caveolin-3 induces persistent late sodium current and is associated with long-QT syndrome

Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias

A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndrome

SCN5A mutations associated with an inherited cardiac arrhythmia, long QT syndrome

Mutations in the hminK gene cause long QT syndrome and suppress IKs function

Loss-of-function mutations in the cardiac calcium channel underlie a new clinical entity characterized by ST-segment elevation, short QT intervals, and sudden cardiac death

Ca(V)1.2 calcium channel dysfunction causes a multisystem disorder including arrhythmia and autism

Mutations of the cardiac ryanodine receptor (RyR2) gene in familial polymorphic ventricular tachycardia

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