Surfactant protein (SP)-A suppresses preterm delivery and inflammation via TLR2 - Wikidata - awgldk - TriplyDB

Surfactant protein (SP)-A suppresses preterm delivery and inflammation via TLR2

Surfactant protein (SP)-A suppresses preterm delivery and inflammation via TLR2

http://www.wikidata.org/entity/Q36859373

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Significant roles played by interleukin-10 in outcome of pregnancy SP-R210 (Myo18A) Isoforms as Intrinsic Modulators of Macrophage Priming and Activation Surfactant protein A is expressed in the central nervous system of rats with experimental autoimmune encephalomyelitis, and suppresses inflammation in human astrocytes and microglia.Pathogenesis of idiopathic pulmonary fibrosis: review of recent findings.Differential expression of collectins in human placenta and role in inflammation during spontaneous labor.Future directions of clinical laboratory evaluation of pregnancy.Neonatal mucosal immunology.Pulmonary surfactant protein a is expressed in mouse retina by Müller cells and impacts neovascularization in oxygen-induced retinopathy Altered autophagic flux enhances inflammatory responses during inflammation-induced preterm labor.Platelet-activating factor: a role in preterm delivery and an essential interaction with Toll-like receptor signaling in mice.Fetal-to-maternal signaling to initiate parturition.Notch Signaling in Inflammation-Induced Preterm Labor.Maternal and fetal roles in bacterially induced preterm labor in the mouse.Regulation of apoptosis and innate immune stimuli in inflammation-induced preterm labor.p38 Mitogen activated protein kinase (MAPK): a new therapeutic target for reducing the risk of adverse pregnancy outcomes.The unique immunological and microbial aspects of pregnancy.Surfactant Protein A Enhances Constitutive Immune Functions of Clathrin Heavy Chain and Clathrin Adaptor Protein 2.Role of Notch signaling during lipopolysaccharide-induced preterm labor.

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Surfactant protein (SP)-A suppresses preterm delivery and inflammation via TLR2

http://www.wikidata.org/entity/Q36859373

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2013 nî lūn-bûn

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2013年論文

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2013年论文

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name

Surfactant protein (SP)-A suppresses preterm delivery and inflammation via TLR2

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type

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Surfactant protein (SP)-A suppresses preterm delivery and inflammation via TLR2

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prefLabel

Surfactant protein (SP)-A suppresses preterm delivery and inflammation via TLR2

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JOURNAL.PONE.0063990

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Surfactant protein (SP)-A suppresses preterm delivery and inflammation via TLR2

@en

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Emmet Hirsch

http://www.w3.org/2001/XMLSchema#string

Keith Smart

http://www.w3.org/2001/XMLSchema#string

Varkha Agrawal

http://www.w3.org/2001/XMLSchema#string

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Toll-like receptor signalling

Surfactant protein secreted by the maturing mouse fetal lung acts as a hormone that signals the initiation of parturition

Surfactant protein-A as an anti-inflammatory component in the amnion: implications for human pregnancy

Expression and localization of lung surfactant protein A in human tissues

By binding SIRPalpha or calreticulin/CD91, lung collectins act as dual function surveillance molecules to suppress or enhance inflammation

Fetal macrophages are not present in the myometrium of women with labor at term.

Surfactant protein A inhibits peptidoglycan-induced tumor necrosis factor-alpha secretion in U937 cells and alveolar macrophages by direct interaction with toll-like receptor 2.

TLR-mediated preterm birth in response to pathogenic agents.

Direct binding of Toll-like receptor 2 to zymosan, and zymosan-induced NF-kappa B activation and TNF-alpha secretion are down-regulated by lung collectin surfactant protein A.

Synergy between viral and bacterial toll-like receptors leads to amplification of inflammatory responses and preterm labor in the mouse.

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e63990

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scholarly article

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10.1371/JOURNAL.PONE.0063990

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5

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2013-05-20T00:00:00Z

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236887673

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23700442

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3659120

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