The c-Jun-induced transformation process involves complex regulation of tenascin-C expression. - Wikidata - awgldk - TriplyDB

The c-Jun-induced transformation process involves complex regulation of tenascin-C expression.

The c-Jun-induced transformation process involves complex regulation of tenascin-C expression.

http://www.wikidata.org/entity/Q36861383

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GATA-6 is a novel transcriptional repressor of the human Tenascin-C gene expression in fibroblasts Astrocyte elevated gene-1: recent insights into a novel gene involved in tumor progression, metastasis and neurodegeneration New insights into the role of nuclear factor-kB in cell growth regulation Transcriptional regulation of tenascin genes Tenascin-C: Form versus function Ets transcription factors cooperate with Sp1 to activate the human tenascin-C promoter Distinct roles of Jun : Fos and Jun : ATF dimers in oncogenesis Annexin II binds progastrin and gastrin-like peptides, and mediates growth factor effects of autocrine and exogenous gastrins on colon cancer and intestinal epithelial cells.Stress-activated protein kinases are negatively regulated by cell density.Function and regulation of AP-1 subunits in skin physiology and pathology.Tenascins and the importance of adhesion modulation.Multiple transcription factor families regulate axon growth and regeneration FosB regulates stretch-induced expression of extracellular matrix proteins in smooth muscle.Role of Annexin-II in GI cancers: interaction with gastrins/progastrins.NF-kappaB is essential for epithelial-mesenchymal transition and metastasis in a model of breast cancer progression.Roles for NF-κB and gene targets of NF-κB in synaptic plasticity, memory, and navigation.Tenascin-C and mechanotransduction in the development and diseases of cardiovascular system The extracellular matrix compartment of neural stem and glial progenitor cells.Tenascin-C induces resistance to apoptosis in pancreatic cancer cell through activation of ERK/NF-κB pathway.The Wilms' tumor gene product represses the transcription of thrombospondin 1 in response to overexpression of c-Jun.Nuclear factor kappa B signaling either stimulates or inhibits neurite growth depending on the phosphorylation status of p65/RelA The intracellular portion of GITR enhances NGF-promoted neurite growth through an inverse modulation of Erk and NF-κB signalling Functional cooperation between JunD and NF-kappaB in rat hepatocytes.Induction of tenascin-C in cardiac myocytes by mechanical deformation. Role of reactive oxygen species.Gastrin induces over-expression of genes involved in human U373 glioblastoma cell migration.Down-regulation of the extracellular matrix protein SPARC in vSrc- and vJun-transformed chick embryo fibroblasts contributes to tumor formation in vivo.

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The c-Jun-induced transformation process involves complex regulation of tenascin-C expression.

http://www.wikidata.org/entity/Q36861383

description

1997 nî lūn-bûn

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P2093

Binétruy B

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Cabon F

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Dejong V

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Gherzi R

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Mercier G

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P2860

Human proto-oncogene c-jun encodes a DNA binding protein with structural and functional properties of transcription factor AP-1

The p65 subunit is responsible for the strong transcription activating potential of NF-kappa B

S26 ribosomal protein RNA: an invariant control for gene regulation experiments in eucaryotic cells and tissues

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

NF-AT-AP-1 and Rel-bZIP: hybrid vigor and binding under the influence

GCN4 protein, a positive transcription factor in yeast, binds general control promoters at all 5' TGACTC 3' sequences

Functional antagonism between c-Jun and MyoD proteins: a direct physical association

jun: oncogene and transcription factor

Human tenascin gene. Structure of the 5'-region, identification, and characterization of the transcription regulatory sequences

Phorbol ester-inducible genes contain a common cis element recognized by a TPA-modulated trans-acting factor

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3202-3209

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10.1128/MCB.17.6.3202

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9154819

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232173

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