Essential contribution of CCL3 to alkali-induced corneal neovascularization by regulating vascular endothelial growth factor production by macrophages.
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Critical role of SDF-1α-induced progenitor cell recruitment and macrophage VEGF production in the experimental corneal neovascularization.Potential involvement of nitric oxide synthase but not inducible nitric oxide synthase in the development of experimental corneal neovascularizationInhibitory effect of CCR3 signal on alkali-induced corneal neovascularizationADP-ribosylation factor as a novel target for corneal neovascularization regressionTopical administration of orbital fat-derived stem cells promotes corneal tissue regenerationVascular endothelial growth factor expression and inhibition in uveal melanoma cell lines.Mesenchymal Stem Cell Secretome: Toward Cell-Free Therapeutic Strategies in Regenerative Medicine.Otitis Media and Nasopharyngeal Colonization in ccl3-/- Mice.S100A proteins in the pathogenesis of experimental corneal neovascularization.B-cell leukemia/lymphoma 10 promotes angiogenesis in an experimental corneal neovascularization model.
P2860
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P2860
Essential contribution of CCL3 to alkali-induced corneal neovascularization by regulating vascular endothelial growth factor production by macrophages.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Essential contribution of CCL3 ...... tor production by macrophages.
@en
type
label
Essential contribution of CCL3 ...... tor production by macrophages.
@en
prefLabel
Essential contribution of CCL3 ...... tor production by macrophages.
@en
P2093
P2860
P1433
P1476
Essential contribution of CCL3 ...... tor production by macrophages.
@en
P2093
Longbiao Li
Naofumi Mukaida
Peirong Lu
Xueguang Zhang
P2860
P304
P577
2008-09-05T00:00:00Z