Modification of the ATM/ATR directed DNA damage response state with aging and long after hepatocyte senescence induction in vivo
about
Feedback between p21 and reactive oxygen production is necessary for cell senescenceGrowth conditions that increase or decrease lifespan in Saccharomyces cerevisiae lead to corresponding decreases or increases in rates of interstitial deletions and non-reciprocal translocationsIncreased FOXM1 expression can stimulate DNA repair in normal hepatocytes in vivo but also increases nuclear foci associated with senescence.The ATM cofactor ATMIN protects against oxidative stress and accumulation of DNA damage in the aging brain.CHEK2 contribution to hereditary breast cancer in non-BRCA familiesAging is associated with an expansion of CD49fhi mammary stem cells that show a decline in function and increased transformation potentialAdult-onset, short-term dietary restriction reduces cell senescence in mice.Impact of UV Radiation on Genome Stability and Human Health.
P2860
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P2860
Modification of the ATM/ATR directed DNA damage response state with aging and long after hepatocyte senescence induction in vivo
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Modification of the ATM/ATR di ...... e senescence induction in vivo
@en
type
label
Modification of the ATM/ATR di ...... e senescence induction in vivo
@en
prefLabel
Modification of the ATM/ATR di ...... e senescence induction in vivo
@en
P2093
P2860
P1476
Modification of the ATM/ATR di ...... e senescence induction in vivo
@en
P2093
Ayman Isbatan
Guy R Adami
Suchismita Panda
P2860
P304
P356
10.1016/J.MAD.2008.02.014
P577
2008-03-14T00:00:00Z