Activation of PPARα ameliorates hepatic insulin resistance and steatosis in high fructose-fed mice despite increased endoplasmic reticulum stress.
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Integrated physiology and systems biology of PPARαInvolvement of the IRE1α-XBP1 pathway and XBP1s-dependent transcriptional reprogramming in metabolic diseasesMetabolomic Approaches to Explore Chemical Diversity of Human Breast-Milk, Formula Milk and Bovine MilkMetabolic Impact of Light Phase-Restricted Fructose Consumption Is Linked to Changes in Hypothalamic AMPK Phosphorylation and Melatonin Production in Rats.PPARα-independent actions of omega-3 PUFAs contribute to their beneficial effects on adiposity and glucose homeostasisPeriostin promotes liver steatosis and hypertriglyceridemia through downregulation of PPARα.Serum amylase levels are decreased in Chinese non-alcoholic fatty liver disease patientsNeuroendocrine and metabolic components of dopamine agonist amelioration of metabolic syndrome in SHR ratsIdentification of matrine as a promising novel drug for hepatic steatosis and glucose intolerance with HSP72 as an upstream targetCharacterization of the Mouse and Human Monoacylglycerol O-Acyltransferase 1 (Mogat1) Promoter in Human Kidney Proximal Tubule and Rat Liver Cells.Chronic activation of PPARα with fenofibrate reduces autophagic proteins in the liver of mice independent of FGF21.Disparate metabolic response to fructose feeding between different mouse strainsE2F1 mediates sustained lipogenesis and contributes to hepatic steatosis.Zingiber mioga reduces weight gain, insulin resistance and hepatic gluconeogenesis in diet-induced obese miceMolecular mechanisms of lipotoxicity and glucotoxicity in nonalcoholic fatty liver disease.Peroxisome proliferator-activated receptor alpha acts as a mediator of endoplasmic reticulum stress-induced hepatocyte apoptosis in acute liver failure.Current role of fenofibrate in the prevention and management of non-alcoholic fatty liver diseaseZerumbone, a Natural Cyclic Sesquiterpene of Zingiber zerumbet Smith, Attenuates Nonalcoholic Fatty Liver Disease in Hamsters Fed on High-Fat DietBilirubin increases insulin sensitivity in leptin-receptor deficient and diet-induced obese mice through suppression of ER stress and chronic inflammation.Nuclear Receptors Resolve Endoplasmic Reticulum Stress to Improve Hepatic Insulin Resistance.Molecular insights into the mechanisms of liver-associated diseases in early-lactating dairy cows: hypothetical role of endoplasmic reticulum stress.The Role of Nuclear Receptors in the Pathophysiology, Natural Course, and Drug Treatment of NAFLD in Humans.The role of ER stress in lipid metabolism and lipotoxicity.Evaluation of the In Vivo and In Vitro Effects of Fructose on Respiratory Chain Complexes in Tissues of Young Rats.eIF2α phosphorylation is required to prevent hepatocyte death and liver fibrosis in mice challenged with a high fructose diet.Peroxisome proliferator-activated receptors as targets to treat non-alcoholic fatty liver disease.The cardiometabolic benefits of glycine: Is glycine an 'antidote' to dietary fructose?Curcumin Improves Palmitate-Induced Insulin Resistance in Human Umbilical Vein Endothelial Cells by Maintaining Proteostasis in Endoplasmic ReticulumFructose Consumption, Lipogenesis, and Non-Alcoholic Fatty Liver Disease.Evaluation of the effects of fructose on oxidative stress and inflammatory parameters in rat brain.Inhibition of hepatocyte nuclear factor 1b induces hepatic steatosis through DPP4/NOX1-mediated regulation of superoxide.A rich medium-chain triacylglycerol diet benefits adiposity but has adverse effects on the markers of hepatic lipogenesis and beta-oxidation.Fructose metabolism and metabolic disease.Liver function and dysfunction - a unique window into the physiological reach of ER stress and the unfolded protein response.Chinese olive extract ameliorates hepatic lipid accumulation in vitro and in vivo by regulating lipid metabolism.Reduced islet function contributes to impaired glucose homeostasis in fructose-fed mice.Naltrexone changes the expression of lipid metabolism-related proteins in the endoplasmic reticulum stress induced hepatic steatosis in mice.Stress-activated miR-21/miR-21* in hepatocytes promotes lipid and glucose metabolic disorders associated with high-fat diet consumption.Hepatic steatosis risk is partly driven by increased de novo lipogenesis following carbohydrate consumption.Hepatoprotective Effects of MHY3200 on High-Fat, Diet-Induced, Non-Alcoholic Fatty Liver Disease in Rats
P2860
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P2860
Activation of PPARα ameliorates hepatic insulin resistance and steatosis in high fructose-fed mice despite increased endoplasmic reticulum stress.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Activation of PPARα ameliorate ...... endoplasmic reticulum stress.
@en
type
label
Activation of PPARα ameliorate ...... endoplasmic reticulum stress.
@en
prefLabel
Activation of PPARα ameliorate ...... endoplasmic reticulum stress.
@en
P2093
P2860
P50
P356
P1433
P1476
Activation of PPARα ameliorate ...... d endoplasmic reticulum stress
@en
P2093
Ji-Ming Ye
Matthew J Watt
Ruo-Qiong Sun
Zi-Heng Choong
P2860
P304
P356
10.2337/DB12-1397
P407
P577
2013-01-24T00:00:00Z