STING-dependent recognition of cyclic di-AMP mediates type I interferon responses during Chlamydia trachomatis infection.
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Multiple hits, including oxidative stress, as pathogenesis and treatment target in non-alcoholic steatohepatitis (NASH)Crosstalk between Mycobacterium tuberculosis and the host cellStructural insights into recognition of c-di-AMP by the ydaO riboswitchBacterial recognition pathways that lead to inflammasome activationRat and human STINGs profile similarly towards anticancer/antiviral compoundsType I Interferon Induction by Neisseria gonorrhoeae: Dual Requirement of Cyclic GMP-AMP Synthase and Toll-like Receptor 4.Molecular mechanisms and new treatment strategies for non-alcoholic steatohepatitis (NASH)Deletion of the cyclic di-AMP phosphodiesterase gene (cnpB) in Mycobacterium tuberculosis leads to reduced virulence in a mouse model of infection.The cyclic dinucleotide c-di-AMP is an allosteric regulator of metabolic enzyme functionThe DNA sensor, cyclic GMP-AMP synthase, is essential for induction of IFN-β during Chlamydia trachomatis infection.IFI16 is required for DNA sensing in human macrophages by promoting production and function of cGAMPMind-controlled transgene expression by a wireless-powered optogenetic designer cell implant.Detection of cyclic di-AMP using a competitive ELISA with a unique pneumococcal cyclic di-AMP binding protein.Mutation in the C-di-AMP cyclase dacA affects fitness and resistance of methicillin resistant Staphylococcus aureus.STING-dependent type I IFN production inhibits cell-mediated immunity to Listeria monocytogenes.cGAS and Ifi204 cooperate to produce type I IFNs in response to Francisella infection.Analyses of the pathways involved in early- and late-phase induction of IFN-beta during C. muridarum infection of oviduct epithelial cellsIdentification of bromophenol thiohydantoin as an inhibitor of DisA, a c-di-AMP synthase, from a 1000 compound library, using the coralyne assay.A bacterial cyclic dinucleotide activates the cytosolic surveillance pathway and mediates innate resistance to tuberculosis.STIM1 Is a Novel Component of ER-Chlamydia trachomatis Inclusion Membrane Contact SitesIntracellular Concentrations of Borrelia burgdorferi Cyclic Di-AMP Are Not Changed by Altered Expression of the CdaA SynthaseThe PAMP c-di-AMP Is Essential for Listeria monocytogenes Growth in Rich but Not Minimal Media due to a Toxic Increase in (p)ppGpp. [corrected]Structural Studies of Potassium Transport Protein KtrA Regulator of Conductance of K+ (RCK) C Domain in Complex with Cyclic Diadenosine Monophosphate (c-di-AMP)Nanoparticulate STING agonists are potent lymph node-targeted vaccine adjuvants.STING agonist formulated cancer vaccines can cure established tumors resistant to PD-1 blockade.The Characterization of Escherichia coli CpdB as a Recombinant Protein Reveals that, besides Having the Expected 3´-Nucleotidase and 2´,3´-Cyclic Mononucleotide Phosphodiesterase Activities, It Is Also Active as Cyclic Dinucleotide PhosphodiesteraseFunctional analysis of the sporulation-specific diadenylate cyclase CdaS in Bacillus thuringiensisThe Chlamydia trachomatis Inclusion Membrane Protein CpoS Counteracts STING-Mediated Cellular Surveillance and Suicide Programs.Detection of a microbial metabolite by STING regulates inflammasome activation in response to Chlamydia trachomatis infection.Nuclease-Resistant c-di-AMP Derivatives That Differentially Recognize RNA and Protein ReceptorsType I IFN Does Not Promote Susceptibility to Foodborne Listeria monocytogenesChlamydia cell biology and pathogenesisCyclic di-AMP Released from Staphylococcus aureus Biofilm Induces a Macrophage Type I Interferon Response.Cyclic di-AMP impairs potassium uptake mediated by a cyclic di-AMP binding protein in Streptococcus pneumoniae.Outrunning the Red Queen: bystander activation as a means of outpacing innate immune subversion by intracellular pathogens.Cyclic dinucleotides and the innate immune response.DhhP, a cyclic di-AMP phosphodiesterase of Borrelia burgdorferi, is essential for cell growth and virulenceIntracellular STING inactivation sensitizes breast cancer cells to genotoxic agents.Hijacking of Membrane Contact Sites by Intracellular Bacterial Pathogens.Uterotubal junction prevents chlamydial ascension via innate immunity.
P2860
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P2860
STING-dependent recognition of cyclic di-AMP mediates type I interferon responses during Chlamydia trachomatis infection.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
STING-dependent recognition of ...... lamydia trachomatis infection.
@en
type
label
STING-dependent recognition of ...... lamydia trachomatis infection.
@en
prefLabel
STING-dependent recognition of ...... lamydia trachomatis infection.
@en
P2093
P2860
P356
P1433
P1476
STING-dependent recognition of ...... hlamydia trachomatis infection
@en
P2093
Christopher M Waters
Dara L Burdette
Jeffrey R Barker
Raphael H Valdivia
Russell E Vance
Victoria K Carpenter
P2860
P304
P356
10.1128/MBIO.00018-13
P577
2013-04-30T00:00:00Z