cis-acting intron mutations that affect the efficiency of avian retroviral RNA splicing: implication for mechanisms of control.
about
Two distant upstream regions containing cis-acting signals regulating splicing facilitate 3'-end processing of avian sarcoma virus RNA.Mutations in the regions of the Rous sarcoma virus 3' splice sites: implications for regulation of alternative splicing.Multiple regions in the Rous sarcoma virus src gene intron act in cis to affect the accumulation of unspliced RNASelection and characterization of replication-competent revertants of a Rous sarcoma virus src gene oversplicing mutant.U1 small nuclear ribonucleoprotein and splicing inhibition by the rous sarcoma virus negative regulator of splicing element.An exonic splicing silencer downstream of the 3' splice site A2 is required for efficient human immunodeficiency virus type 1 replication.Feedback regulation of human immunodeficiency virus type 1 expression by the Rev protein.The role of overlapping U1 and U11 5' splice site sequences in a negative regulator of splicingJuxtaposition of two distant, serine-arginine-rich protein-binding elements is required for optimal polyadenylation in Rous sarcoma virus.Translation of MMTV Gag requires nuclear events involving splicing motifs in addition to the viral Rem protein and RmREThe Rev protein of human immunodeficiency virus type 1 counteracts the effect of an AU-rich negative element in the human papillomavirus type 1 late 3' untranslated region.Branchpoint and polypyrimidine tract mutations mediating the loss and partial recovery of the Moloney murine sarcoma virus MuSVts110 thermosensitive splicing phenotype.SR protein splicing factors interact with the Rous sarcoma virus negative regulator of splicing elementPossible role of splice acceptor site in expression of unspliced gag-containing message of Moloney murine leukemia virus.The Moloney murine sarcoma virus ts110 5' splice site signal contributes to the regulation of splicing efficiency and thermosensitivity.Genetic selection for balanced retroviral splicing: novel regulation involving the second step can be mediated by transitions in the polypyrimidine tractIdentification of positive and negative splicing regulatory elements within the terminal tat-rev exon of human immunodeficiency virus type 1.Intronic sequences and 3' splice sites control Rous sarcoma virus RNA splicingDistinct RNA sequences in the gag region of human immunodeficiency virus type 1 decrease RNA stability and inhibit expression in the absence of Rev protein.Control of retroviral RNA splicing through maintenance of suboptimal processing signals.A block in mammalian splicing occurring after formation of large complexes containing U1, U2, U4, U5, and U6 small nuclear ribonucleoproteins.Simian immunodeficiency virus displays complex patterns of RNA splicing.Activation of cryptic splice sites in murine sarcoma virus-124 mutantsA temperature-sensitive phenotype of avian myeloblastosis virus: determinants that influence the production of viral mRNAsReversion of thermosensitive splicing defect of Moloney murine sarcoma virus ts110 by oversplicing of viral RNA.Regulation of RNA splicing in gag-deficient mutants of Moloney murine sarcoma virus MuSVts110.Regulation of Rous sarcoma virus RNA splicing and stability.RNA processing control in avian retrovirusesIdentification of cis-acting intron and exon regions in influenza virus NS1 mRNA that inhibit splicing and cause the formation of aberrantly sedimenting presplicing complexes.A C-terminal "Tail" Region in the Rous Sarcoma Virus Integrase Provides High Plasticity of Functional Integrase Oligomerization during Intasome Assembly.cis Elements required for high-level expression of unspliced Gag-containing message in Moloney murine leukemia virusGeneration and role of defective proviruses in cytopathic feline leukemia virus (FeLV-FAIDS) infectionsGenetic determinant of rapid-onset B-cell lymphoma by avian leukosis virus.A naturally arising mutation of a potential silencer of exon splicing in human immunodeficiency virus type 1 induces dominant aberrant splicing and arrests virus production.The tat/rev intron of human immunodeficiency virus type 1 is inefficiently spliced because of suboptimal signals in the 3' splice site.The encephalomyocarditis virus internal ribosome entry site allows efficient coexpression of two genes from a recombinant provirus in cultured cells and in embryos.Influenza A virus utilizes suboptimal splicing to coordinate the timing of infection.
P2860
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P2860
cis-acting intron mutations that affect the efficiency of avian retroviral RNA splicing: implication for mechanisms of control.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
cis-acting intron mutations th ...... ion for mechanisms of control.
@en
type
label
cis-acting intron mutations th ...... ion for mechanisms of control.
@en
prefLabel
cis-acting intron mutations th ...... ion for mechanisms of control.
@en
P2093
P2860
P1433
P1476
cis-acting intron mutations th ...... tion for mechanisms of control
@en
P2093
P2860
P304
P407
P577
1988-08-01T00:00:00Z