Calcium sparks and excitation-contraction coupling in phospholamban-deficient mouse ventricular myocytes.
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Proteomic analysis reveals new cardiac-specific dystrophin-associated proteinsPhosphodiesterase 4B in the cardiac L-type Ca²⁺ channel complex regulates Ca²⁺ current and protects against ventricular arrhythmias in miceCa2+ sparks in embryonic mouse skeletal muscle selectively deficient in dihydropyridine receptor alpha1S or beta1a subunits.Phospholamban: a major determinant of the cardiac force-frequency relationshipPhospholemman is a negative feed-forward regulator of Ca2+ in β-adrenergic signaling, accelerating β-adrenergic inotropy.Reduced Ca2+ spark activity after subarachnoid hemorrhage disables BK channel control of cerebral artery tone.Thin filament incorporation of an engineered cardiac troponin C variant (L48Q) enhances contractility in intact cardiomyocytes from healthy and infarcted hearts.Spatial characteristics of sarcoplasmic reticulum Ca2+ release events triggered by L-type Ca2+ current and Na+ current in guinea-pig cardiac myocytes.Measurements of calcium transients in ventricular cells during discontinuous action potential conduction.Sarcoplasmic reticulum calcium load regulates rat arterial smooth muscle calcium sparks and transient K(Ca) currents.cAMP/PKA-dependent increases in Ca Sparks, oscillations and SR Ca stores in retinal arteriolar myocytes after exposure to vasopressin.Functional coupling of calcineurin and protein kinase A in mouse ventricular myocytes.Functional coupling of ryanodine receptors to KCa channels in smooth muscle cells from rat cerebral arteries.Local control of excitation-contraction coupling in human embryonic stem cell-derived cardiomyocytes.Regulation of excitation-contraction coupling in mouse cardiac myocytes: integrative analysis with mathematical modellingCellular and functional defects in a mouse model of heart failure.Phospholamban and cardiac contractility.Reverse mode of the sarcoplasmic reticulum calcium pump and load-dependent cytosolic calcium decline in voltage-clamped cardiac ventricular myocytes.Potentiation of fractional sarcoplasmic reticulum calcium release by total and free intra-sarcoplasmic reticulum calcium concentrationInvited review: significance of spatial and temporal heterogeneity of calcium transients in smooth muscle.Control of sarcoplasmic reticulum Ca2+ release by stochastic RyR gating within a 3D model of the cardiac dyad and importance of induction decay for CICR terminationRegulation of cardiac sarcoplasmic reticulum Ca release by luminal [Ca] and altered gating assessed with a mathematical model.Calcium biology of the transverse tubules in heartThe challenge of molecular medicine: complexity versus Occam's razorRegulation of cardiac excitation-contraction coupling by action potential repolarization: role of the transient outward potassium current (I(to)).Regulation of gastrointestinal motility by Ca2+/calmodulin-stimulated protein kinase II.Acute reversal of phospholamban inhibition facilitates the rhythmic whole-cell propagating calcium waves in isolated ventricular myocytesPhospholamban phosphorylation, mutation, and structural dynamics: a biophysical approach to understanding and treating cardiomyopathy.Chasing cardiac physiology and pathology down the CaMKII cascadeCardiac GPCRs: GPCR signaling in healthy and failing hearts.Twenty years of calcium imaging: cell physiology to dye for.The influence of sarcoplasmic reticulum Ca2+ concentration on Ca2+ sparks and spontaneous transient outward currents in single smooth muscle cells.Evidence for a voltage-dependent enhancement of neurotransmitter release mediated via the synaptic protein interaction site of N-type Ca2+ channels.Intact Heart Loose Patch Photolysis Reveals Ionic Current Kinetics During Ventricular Action PotentialsCa2+ clearance and contractility in vascular smooth muscle: evidence from gene-altered murine models.Transgenic expression of replication-restricted enteroviral genomes in heart muscle induces defective excitation-contraction coupling and dilated cardiomyopathy.Phospholamban knockout breaks arrhythmogenic Ca²⁺ waves and suppresses catecholaminergic polymorphic ventricular tachycardia in mice.Sarcoplasmic reticulum function in smooth muscle.Dynamic local changes in sarcoplasmic reticulum calcium: physiological and pathophysiological roles.Efficacy and potency of class I antiarrhythmic drugs for suppression of Ca2+ waves in permeabilized myocytes lacking calsequestrin.
P2860
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P2860
Calcium sparks and excitation-contraction coupling in phospholamban-deficient mouse ventricular myocytes.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
1997年论文
@zh
1997年论文
@zh-cn
name
Calcium sparks and excitation- ...... nt mouse ventricular myocytes.
@en
type
label
Calcium sparks and excitation- ...... nt mouse ventricular myocytes.
@en
prefLabel
Calcium sparks and excitation- ...... nt mouse ventricular myocytes.
@en
P2860
P1476
Calcium sparks and excitation- ...... ent mouse ventricular myocytes
@en
P2093
E G Kranias
W J Lederer
P2860
P356
10.1111/J.1469-7793.1997.021BI.X
P407
P478
503 ( Pt 1)
P577
1997-08-01T00:00:00Z