Mutations in the vaccinia virus A33R and B5R envelope proteins that enhance release of extracellular virions and eliminate formation of actin-containing microvilli without preventing tyrosine phosphorylation of the A36R protein.
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Poxvirus cell entry: how many proteins does it take?A36-dependent actin filament nucleation promotes release of vaccinia virusThe Structure of the Poxvirus A33 Protein Reveals a Dimer of Unique C-Type Lectin-Like DomainsPoxvirus tropismModulation of the myxoma virus plaque phenotype by vaccinia virus protein F11.The formin FHOD1 and the small GTPase Rac1 promote vaccinia virus actin-based motility.Genetically stable and fully effective smallpox vaccine strain constructed from highly attenuated vaccinia LC16m8LC16m8, a highly attenuated vaccinia virus vaccine lacking expression of the membrane protein B5R, protects monkeys from monkeypoxMicroRNA regulation of glycoprotein B5R in oncolytic vaccinia virus reduces viral pathogenicity without impairing its antitumor efficacyThe vaccinia virus O1 protein is required for sustained activation of extracellular signal-regulated kinase 1/2 and promotes viral virulence.Vaccinia Virus LC16m8∆ as a Vaccine Vector for Clinical Applications.The A33-dependent incorporation of B5 into extracellular enveloped vaccinia virions is mediated through an interaction between their lumenal domainsSerro 2 Virus Highlights the Fundamental Genomic and Biological Features of a Natural Vaccinia Virus Infecting Humans.Poxvirus proteomics and virus-host protein interactions.The non-canonical roles of clathrin and actin in pathogen internalization, egress and spread.Rho'ing in and out of cells: viral interactions with Rho GTPase signalingVaccinia virus A34 glycoprotein determines the protein composition of the extracellular virus envelope.Surf and turf: mechanism of enhanced virus spread during poxvirus infection.Interaction between vaccinia virus extracellular virus envelope A33 and B5 glycoproteins.Wiskott-Aldrich syndrome protein is needed for vaccinia virus pathogenesis.Protein B5 is required on extracellular enveloped vaccinia virus for repulsion of superinfecting virions.Loss of Actin-Based Motility Impairs Ectromelia Virus Release In Vitro but Is Not Critical to Spread In Vivo.
P2860
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P2860
Mutations in the vaccinia virus A33R and B5R envelope proteins that enhance release of extracellular virions and eliminate formation of actin-containing microvilli without preventing tyrosine phosphorylation of the A36R protein.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
Mutations in the vaccinia viru ...... orylation of the A36R protein.
@en
type
label
Mutations in the vaccinia viru ...... orylation of the A36R protein.
@en
prefLabel
Mutations in the vaccinia viru ...... orylation of the A36R protein.
@en
P2093
P2860
P1433
P1476
Mutations in the vaccinia viru ...... orylation of the A36R protein.
@en
P2093
Andrea S Weisberg
Brian M Ward
P2860
P304
12266-12275
P356
10.1128/JVI.77.22.12266-12275.2003
P407
P50
P577
2003-11-01T00:00:00Z