Expression and function of fatty acid amide hydrolase in prostate cancer. - Wikidata - awgldk - TriplyDB

Expression and function of fatty acid amide hydrolase in prostate cancer.

Expression and function of fatty acid amide hydrolase in prostate cancer.

http://www.wikidata.org/entity/Q36901770

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Endocannabinoid system as a regulator of tumor cell malignancy - biological pathways and clinical significance Endocannabinoids as Guardians of Metastasis Cannabinoids, endocannabinoids, and cancer Elevating endocannabinoid levels: pharmacological strategies and potential therapeutic applications High-throughput transcriptomic and RNAi analysis identifies AIM1, ERGIC1, TMED3 and TPX2 as potential drug targets in prostate cancer Ketoconazole inhibits the cellular uptake of anandamide via inhibition of FAAH at pharmacologically relevant concentrations Potential upstream regulators of cannabinoid receptor 1 signaling in prostate cancer: a Bayesian network analysis of data from a tissue microarray.Fatty acid amide hydrolase in prostate cancer: association with disease severity and outcome, CB1 receptor expression and regulation by IL-4 Tumour Cannabinoid CB(1) receptor and phosphorylated epidermal growth factor receptor expression are additive prognostic markers for prostate cancer.The association of N-palmitoylethanolamine with the FAAH inhibitor URB597 impairs melanoma growth through a supra-additive action.A novel activity of microsomal epoxide hydrolase: metabolism of the endocannabinoid 2-arachidonoylglycerol.FAAH inhibition enhances anandamide mediated anti-tumorigenic effects in non-small cell lung cancer by downregulating the EGF/EGFR pathway.Anti-proliferative effect of a putative endocannabinoid, 2-arachidonylglyceryl ether in prostate carcinoma cells.Association between cannabinoid CB₁ receptor expression and Akt signalling in prostate cancer.Regulation of circulating endocannabinoids associated with cancer and metastases in mice and humans.Monoacylglycerol lipase exerts dual control over endocannabinoid and fatty acid pathways to support prostate cancer.The endocannabinoid system and cancer: therapeutic implication.The influence of monoacylglycerol lipase inhibition upon the expression of epidermal growth factor receptor in human PC-3 prostate cancer cells.Cannabinoid receptor type 1 (CB1) activation inhibits small GTPase RhoA activity and regulates motility of prostate carcinoma cells.Prognosis of treatment response (pathological complete response) in breast cancer.Fatty acid amide hydrolase inhibitors confer anti-invasive and antimetastatic effects on lung cancer cells.The endocannabinoid system and sex steroid hormone-dependent cancers.Roles of Eicosanoids in Prostate Cancer.Update on the endocannabinoid system as an anticancer target.The endocannabinoid system in prostate cancer.Delta(9) -tetrahydrocannabinol and cannabidiol as potential curative agents for cancer: A critical examination of the preclinical literature.The evolving role of the endocannabinoid system in gynaecological cancer.Antitumorigenic targets of cannabinoids - current status and implications.Novel transcription-induced fusion RNAs in prostate cancer.Phyto-, endo- and synthetic cannabinoids: promising chemotherapeutic agents in the treatment of breast and prostate carcinomas.Phorbol ester stimulates ethanolamine release from the metastatic basal prostate cancer cell line PC3 but not from prostate epithelial cell lines LNCaP and P4E6.Expression and secretion of N-acylethanolamine-hydrolysing acid amidase in human prostate cancer cells.Effects of tumour necrosis factor α upon the metabolism of the endocannabinoid anandamide in prostate cancer cells.Arachidonic acid pathway members PLA2G7, HPGD, EPHX2, and CYP4F8 identified as putative novel therapeutic targets in prostate cancer.The Endocannabinoid System as Pharmacological Target Derived from Its CNS Role in Energy Homeostasis and Reward. Applications in Eating Disorders and Addiction.Endocannabinoids participate in placental apoptosis induced by hypoxia inducible factor-1.INSIGHT ON THE IMPACT OF ENDOCANNABINOID SYSTEM IN CANCER: A REVIEW.

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P2860

Expression and function of fatty acid amide hydrolase in prostate cancer.

http://www.wikidata.org/entity/Q36901770

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Expression and function of fatty acid amide hydrolase in prostate cancer.

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type

label

Expression and function of fatty acid amide hydrolase in prostate cancer.

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prefLabel

Expression and function of fatty acid amide hydrolase in prostate cancer.

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P1433

International Journal of Cancer

P1476

Expression and function of fatty acid amide hydrolase in prostate cancer

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P2093

Andre Kajdacsy-Balla

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Carol L Williams

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Iffat Choudhry

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Kasem Nithipatikom

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Rebecca Thill

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William B Campbell

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P2860

Cloning of the first sn1-DAG lipases points to the spatial and temporal regulation of endocannabinoid signaling in the brain

Increased metastatic potential in human prostate carcinoma cells by overexpression of arachidonate 12-lipoxygenase

2-Arachidonoylglycerol: a possible endogenous cannabinoid receptor ligand in brain

Identification of an endogenous 2-monoglyceride, present in canine gut, that binds to cannabinoid receptors

Molecular characterization of an enzyme that degrades neuromodulatory fatty-acid amides

Arachidonic acid stimulates prostate cancer cell growth: critical role of 5-lipoxygenase.

Properties of rat liver N-acylethanolamine amidohydrolase.

A new class of inhibitors of 2-arachidonoylglycerol hydrolysis and invasion of prostate cancer cells.

A role for monoglyceride lipase in 2-arachidonoylglycerol inactivation.

Modulators of endocannabinoid enzymic hydrolysis and membrane transport.

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1318-1326

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scholarly article

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10.1002/IJC.23674

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6

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123

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Michael P Endsley

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2008-09-01T00:00:00Z

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5289625

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18566995

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2548421

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