Histone modifications are responsible for decreased Fas expression and apoptosis resistance in fibrotic lung fibroblasts.
about
Beyond the genome: epigenetic mechanisms in lung remodelingEpigenetics in Brain Tumors: HDACs Take Center StagePhenotypic switching induced by damaged matrix is associated with DNA methyltransferase 3A (DNMT3A) activity and nuclear localization in smooth muscle cells (SMC)MCL-1 is modulated in Crohn's disease fibrosis by miR-29b via IL-6 and IL-8Precision Medicine: The New Frontier in Idiopathic Pulmonary FibrosisRole of epigenetics in pulmonary hypertensionHDAC4: mechanism of regulation and biological functionsInhibition of myocardin-related transcription factor/serum response factor signaling decreases lung fibrosis and promotes mesenchymal cell apoptosis.Integrating omics technologies to study pulmonary physiology and pathology at the systems level.Genetics of allergic diseases.Epigenetics in lung fibrosis: from pathobiology to treatment perspective.Mechanisms of Lung Fibrosis Resolution.Mechanistic links between aging and lung fibrosis.Cigarette smoke induces distinct histone modifications in lung cells: implications for the pathogenesis of COPD and lung cancer.Epigenetics in immune-mediated pulmonary diseases.Recent advances in understanding the pathogenesis of scleroderma-interstitial lung disease.Epigenetic targets for novel therapies of lung diseases.Epigenetics in idiopathic pulmonary fibrosis.The histone deacetylase inhibitor, romidepsin, as a potential treatment for pulmonary fibrosis.Thy-1 interaction with Fas in lipid rafts regulates fibroblast apoptosis and lung injury resolution.Thrombin Induces CCL2 Expression in Human Lung Fibroblasts via p300 Mediated Histone Acetylation and NF-KappaB Activation.Regulation of Gene Expression by Sodium Valproate in Epithelial-to-Mesenchymal Transition.Lung carcinogenesis and fibrosis taken together: just coincidence?Pulmonary fibrosis, part I: epidemiology, pathogenesis, and diagnosis.Ezh2 restricts the smooth muscle lineage during mouse lung mesothelial development."Scar-cinoma": viewing the fibrotic lung mesenchymal cell in the context of cancer biology.Emerging therapies for idiopathic pulmonary fibrosis, a progressive age-related disease.MicroRNA-29c regulates apoptosis sensitivity via modulation of the cell-surface death receptor, Fas, in lung fibroblasts.Loss of CDKN2B Promotes Fibrosis via Increased Fibroblast Differentiation Rather Than Proliferation.Regulation of fibroblast Fas expression by soluble and mechanical pro-fibrotic stimuli.p53 and metabolism: from mechanism to therapeutics.Emerging therapies for idiopathic pulmonary fibrosis, a progressive age-related disease.Epigenetic Regulation of Myofibroblast Phenotypes in Fibrosis
P2860
Q27021767-5D5A3F7B-9D45-41E9-8351-9BF82E7C0202Q28080964-A3C88DA7-A67D-4380-B804-7B784FE72B0FQ28567085-FAB8A451-C343-4DE5-9BD2-D1432CCF87C8Q33581899-34DE9D90-384E-4645-871F-020CB00D1FD1Q33714064-8827ADAA-2E08-4A3A-8A08-07AC9170C61AQ33764713-563B0E28-C69E-470E-BBDA-77D59C4E2B97Q34039987-71D3D9CF-8609-4723-AF9F-B8AF4A15FFA9Q35236478-FE3BB828-DD2B-4BBB-AA84-5A031DA8B122Q35413415-34A9AC26-D58D-4893-80DF-ED4A9D0223B5Q35556785-D7C4A4C8-8C46-4829-A6B0-2B6216849248Q36119163-51ED5035-AA09-46A1-BC00-944BCD17F0D5Q36885732-A51AADFF-E729-48D4-B318-488C0451AD0AQ37366137-56427C98-CE63-43F8-BF77-3C7FF35A898EQ37684600-87D2CE88-E536-402A-8E4C-73F3DDBEC304Q38163413-7AA32031-5A9A-4A91-B9EE-84115CC02489Q38187607-9D484EDE-B300-4737-9E1E-C24C662417A6Q38275276-D69257DE-EA60-4DDA-8915-2ED060D525EFQ38346940-6ED47EF1-ABD9-4DCE-86DF-FDFC1683E2E2Q38688113-793E8DC2-A471-4726-9870-B78A811DEAD7Q38717775-EFCDB44A-11CA-401A-BCC0-A1725CC7F789Q38838136-735BBDA1-AAD2-4D4F-B433-8DA1652C2BF1Q38842811-0923A76C-635E-4D13-854E-C2A3129677BAQ39237924-506068BC-4FDF-46C3-A178-4AFFFC7CFEFCQ40272697-D98DCB33-DEB5-4834-992E-162FE9BC97E5Q42136235-AAEC1838-366C-4BBB-BAA1-06829EB8D06FQ42594342-0275C01A-B3B3-4B0A-BFDA-327D0D30DFC3Q47913152-14D786CC-4C1C-49EB-A4AD-E613903C304EQ48963871-CEC27AC0-25F9-4321-8207-60A679E2161FQ50116500-FB9680B2-A7E0-4590-B67D-8F3A87FC2DDFQ53684491-AC6C126C-E0CD-4C32-9335-4A944571720CQ55002919-121B1B24-EF21-4EEF-91A1-5A33FB2F180DQ55332823-BE652771-1177-4EE1-9F7C-36B242DDDD82Q57147732-8BD86226-E831-484A-8698-3F067628ED2C
P2860
Histone modifications are responsible for decreased Fas expression and apoptosis resistance in fibrotic lung fibroblasts.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Histone modifications are resp ...... in fibrotic lung fibroblasts.
@en
type
label
Histone modifications are resp ...... in fibrotic lung fibroblasts.
@en
prefLabel
Histone modifications are resp ...... in fibrotic lung fibroblasts.
@en
P2093
P2860
P50
P356
P1476
Histone modifications are resp ...... e in fibrotic lung fibroblasts
@en
P2093
A M Scruggs
M Peters-Golden
S Przybranowski
P2860
P356
10.1038/CDDIS.2013.146
P577
2013-05-02T00:00:00Z