Glucocorticoids alleviate intestinal ER stress by enhancing protein folding and degradation of misfolded proteins. - Wikidata - awgldk - TriplyDB

Glucocorticoids alleviate intestinal ER stress by enhancing protein folding and degradation of misfolded proteins.

Glucocorticoids alleviate intestinal ER stress by enhancing protein folding and degradation of misfolded proteins.

http://www.wikidata.org/entity/Q36907892

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Ectopic microRNA-150-5p transcription sensitizes glucocorticoid therapy response in MM1S multiple myeloma cells but fails to overcome hormone therapy resistance in MM1R cells Hydrocortisone-induced parkin prevents dopaminergic cell death via CREB pathway in Parkinson's disease model Endoplasmic reticulum stress and oxidative stress in cell fate decision and human disease.Epithelial Sel1L is required for the maintenance of intestinal homeostasis Bitter melon protects against ER stress in LS174T colonic epithelial cells Interferon-γ-Induced Unfolded Protein Response in Conjunctival Goblet Cells as a Cause of Mucin Deficiency in Sjögren Syndrome.High Fat Diets Induce Colonic Epithelial Cell Stress and Inflammation that is Reversed by IL-22 Increased glycogen synthase kinase-3β and hexose-6-phosphate dehydrogenase expression in adipose tissue may contribute to glucocorticoid-induced mouse visceral adiposity Colonic microbiota can promote rapid local improvement of murine colitis by thioguanine independently of T lymphocytes and host metabolism.Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.The Unfolded Protein Response in the Immune Cell Development: Putting the Caretaker in the Driving Seat.The transrepression arm of glucocorticoid receptor signaling is protective in mutant huntingtin-mediated neurodegeneration.Neutralizing IL-23 is superior to blocking IL-17 in suppressing intestinal inflammation in a spontaneous murine colitis model.MUC2 mucin deficiency alters inflammatory and metabolic pathways in the mouse intestinal mucosa.TMEM258 Is a Component of the Oligosaccharyltransferase Complex Controlling ER Stress and Intestinal Inflammation.TNFR1-induced lethal inflammation is mediated by goblet and Paneth cell dysfunction.Colonic thioguanine pro-drug: Investigation of microbiome and novel host metabolism.Lactic Acid Bacteria may Impact Intestinal Barrier Function by Modulating Goblet Cells.A dual role for glucocorticoid-induced leucine zipper in glucocorticoid function: tumor growth promotion or suppression?FKBP11 protects intestinal epithelial cells against inflammation‑induced apoptosis via the JNK‑caspase pathway in Crohn's disease

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Glucocorticoids alleviate intestinal ER stress by enhancing protein folding and degradation of misfolded proteins.

http://www.wikidata.org/entity/Q36907892

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2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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name

Glucocorticoids alleviate inte ...... adation of misfolded proteins.

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type

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Glucocorticoids alleviate inte ...... adation of misfolded proteins.

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Glucocorticoids alleviate inte ...... adation of misfolded proteins.

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Journal of Experimental Medicine

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Glucocorticoids alleviate inte ...... adation of misfolded proteins.

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P2093

Chin Wen Png

http://www.w3.org/2001/XMLSchema#string

Denis I Crane

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Indrajit Das

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Iulia Oancea

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Martina Proctor

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Michael A McGuckin

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Rajaraman D Eri

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Rohan Lourie

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Sumaira Z Hasnain

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Timothy H Florin

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P2860

XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease

The protein disulfide isomerase AGR2 is essential for production of intestinal mucus

Protein-protein interactions and transcriptional antagonism between the subfamily of NGFI-B/Nur77 orphan nuclear receptors and glucocorticoid receptor

Dexamethasone causes sustained expression of mitogen-activated protein kinase (MAPK) phosphatase 1 and phosphatase-mediated inhibition of MAPK p38.

The molecular basis for selective inhibition of unconventional mRNA splicing by an IRE1-binding small molecule

One step at a time: endoplasmic reticulum-associated degradation

SPDEF regulates goblet cell hyperplasia in the airway epithelium

West Nile Virus Infection Activates the Unfolded Protein Response, Leading to CHOP Induction and Apoptosis

Hepatitis C Virus Infection Induces Apoptosis through a Bax-Triggered, Mitochondrion-Mediated, Caspase 3-Dependent Pathway

Discordance of UPR signaling by ATF6 and Ire1p-XBP1 with levels of target transcripts

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1201-1216

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10.1084/JEM.20121268

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6

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210

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2013-05-06T00:00:00Z

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236644139

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23650437

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protein folding

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3674691

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