Successful transfer to sulfonylureas in KCNJ11 neonatal diabetes is determined by the mutation and duration of diabetes. - Wikidata - awgldk - TriplyDB

Successful transfer to sulfonylureas in KCNJ11 neonatal diabetes is determined by the mutation and duration of diabetes.

Successful transfer to sulfonylureas in KCNJ11 neonatal diabetes is determined by the mutation and duration of diabetes.

http://www.wikidata.org/entity/Q36908155

about

Hyperglycaemia induces metabolic dysfunction and glycogen accumulation in pancreatic β-cells.β-Cell dysfunction in diabetes: a crisis of identity?Sulfonylurea challenge test in subjects diagnosed with type 1 diabetes mellitus.Neonatal Diabetes and the KATP Channel: From Mutation to Therapy.First case of neonatal diabetes with KCNJ11 Q52R mutation successfully switched from insulin to sulphonylurea treatment.KATP Channel Mutations and Neonatal Diabetes.Pancreatic β-Cell Electrical Activity and Insulin Secretion: Of Mice and Men.Early transition from insulin to sulfonylureas in neonatal diabetes and follow-up: Experience from China.Neonatal Diabetes Mellitus: An Update on Diagnosis and Management.A case with relapsed transient neonatal diabetes mellitus treated with sulfonylurea, ending chronic insulin requirement.Successful Treatment of Diabetic Ketoacidosis and Hyperglycemic Hyperosmolar Status in an Infant with KCNJ11-Related Neonatal Diabetes Mellitus via Continuous Renal Replacement Therapy

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P2860

Successful transfer to sulfonylureas in KCNJ11 neonatal diabetes is determined by the mutation and duration of diabetes.

http://www.wikidata.org/entity/Q36908155

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Successful transfer to sulfony ...... tion and duration of diabetes.

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type

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Successful transfer to sulfony ...... tion and duration of diabetes.

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Successful transfer to sulfony ...... tion and duration of diabetes.

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Successful transfer to sulfony ...... ation and duration of diabetes

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P2093

Ali J Chakera

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Maggie H Shepherd

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Roisin Finn

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Tarig Babiker

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P2860

Functional analysis of six Kir6.2 (KCNJ11) mutations causing neonatal diabetes.

Reversible changes in pancreatic islet structure and function produced by elevated blood glucose.

Adjacent mutations in the gating loop of Kir6.2 produce neonatal diabetes and hyperinsulinism.

The value of in vitro studies in a case of neonatal diabetes with a novel Kir6.2-W68G mutation.

Age at the time of sulfonylurea initiation influences treatment outcomes in KCNJ11-related neonatal diabetes

The role of the KATP channel in glucose homeostasis in health and disease: more than meets the islet.

Sulfonylurea improves CNS function in a case of intermediate DEND syndrome caused by a mutation in KCNJ11.

Switching from insulin to oral sulfonylureas in patients with diabetes due to Kir6.2 mutations.

Mutations at the same residue (R50) of Kir6.2 (KCNJ11) that cause neonatal diabetes produce different functional effects.

Mutations in KCNJ11, which encodes Kir6.2, are a common cause of diabetes diagnosed in the first 6 months of life, with the phenotype determined by genotype.

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1162-1166

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10.1007/S00125-016-3921-8

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6

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59

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Andrew Tym Hattersley

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Natascia Vedovato

Kashyap A Patel

Sarah E Flanagan

Nicholas J Thomas

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2016-03-31T00:00:00Z

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