Tropism of and innate immune responses to the novel human betacoronavirus lineage C virus in human ex vivo respiratory organ cultures - Wikidata - awgldk - TriplyDB

Tropism of and innate immune responses to the novel human betacoronavirus lineage C virus in human ex vivo respiratory organ cultures

Tropism of and innate immune responses to the novel human betacoronavirus lineage C virus in human ex vivo respiratory organ cultures

http://www.wikidata.org/entity/Q36911737

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State of Knowledge and Data Gaps of Middle East Respiratory Syndrome Coronavirus (MERS-CoV) in Humans Emergence of the Middle East respiratory syndrome coronavirus Genetic characterization of Betacoronavirus lineage C viruses in bats reveals marked sequence divergence in the spike protein of pipistrellus bat coronavirus HKU5 in Japanese pipistrelle: implications for the origin of the novel Middle East respirat Critically ill patients with Middle East respiratory syndrome coronavirus infection Coronaviruses and the human airway: a universal system for virus-host interaction studies Animal models of Middle East respiratory syndrome coronavirus infection Evaluation of the human adaptation of influenza A/H7N9 virus in PB2 protein using human and swine respiratory tract explant cultures Rhinovirus C targets ciliated airway epithelial cells.Rapid generation of a mouse model for Middle East respiratory syndrome.The role of laboratory diagnostics in emerging viral infections: the example of the Middle East respiratory syndrome epidemic.A review of treatment modalities for Middle East Respiratory Syndrome.Pathogenic influenza viruses and coronaviruses utilize similar and contrasting approaches to control interferon-stimulated gene responses.Advances in antivirals for non-influenza respiratory virus infections.Treatment outcomes for patients with Middle Eastern Respiratory Syndrome Coronavirus (MERS CoV) infection at a coronavirus referral center in the Kingdom of Saudi Arabia.The emergence of the Middle East respiratory syndrome coronavirus Screening of an FDA-approved compound library identifies four small-molecule inhibitors of Middle East respiratory syndrome coronavirus replication in cell culture.Host species restriction of Middle East respiratory syndrome coronavirus through its receptor, dipeptidyl peptidase 4.Middle East respiratory syndrome coronavirus: another zoonotic betacoronavirus causing SARS-like disease Crystal structure of the Middle East respiratory syndrome coronavirus (MERS-CoV) papain-like protease bound to ubiquitin facilitates targeted disruption of deubiquitinating activity to demonstrate its role in innate immune suppression Role of the spike glycoprotein of human Middle East respiratory syndrome coronavirus (MERS-CoV) in virus entry and syncytia formation.Generation of a transgenic mouse model of Middle East respiratory syndrome coronavirus infection and disease.Cytokine systems approach demonstrates differences in innate and pro-inflammatory host responses between genetically distinct MERS-CoV isolates.Middle East Respiratory Coronavirus Accessory Protein 4a Inhibits PKR-Mediated Antiviral Stress Responses Effective inhibition of MERS-CoV infection by resveratrol Middle East respiratory syndrome coronavirus ORF4b protein inhibits type I interferon production through both cytoplasmic and nuclear targets.Middle East respiratory syndrome coronavirus infection: virus-host cell interactions and implications on pathogenesis Dipeptidyl Peptidase 4 Distribution in the Human Respiratory Tract: Implications for the Middle East Respiratory Syndrome.Middle East Respiratory Syndrome Coronavirus Causes Multiple Organ Damage and Lethal Disease in Mice Transgenic for Human Dipeptidyl Peptidase 4.Association between Severity of MERS-CoV Infection and Incubation Period.Activation of RNase L by Murine Coronavirus in Myeloid Cells Is Dependent on Basal Oas Gene Expression and Independent of Virus-Induced Interferon The structural and accessory proteins M, ORF 4a, ORF 4b, and ORF 5 of Middle East respiratory syndrome coronavirus (MERS-CoV) are potent interferon antagonists The Middle East respiratory syndrome--how worried should we be?Engineering a replication-competent, propagation-defective Middle East respiratory syndrome coronavirus as a vaccine candidate Reverse genetics with a full-length infectious cDNA of the Middle East respiratory syndrome coronavirus.Middle East respiratory syndrome coronavirus (MERS-CoV) causes transient lower respiratory tract infection in rhesus macaques Interferon-β and mycophenolic acid are potent inhibitors of Middle East respiratory syndrome coronavirus in cell-based assays.Evaluation of serologic and antigenic relationships between middle eastern respiratory syndrome coronavirus and other coronaviruses to develop vaccine platforms for the rapid response to emerging coronaviruses.Middle east respiratory syndrome coronavirus 4a protein is a double-stranded RNA-binding protein that suppresses PACT-induced activation of RIG-I and MDA5 in the innate antiviral response.Membrane ectopeptidases targeted by human coronaviruses.Pathogenesis of Middle East respiratory syndrome coronavirus.

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P2860

Tropism of and innate immune responses to the novel human betacoronavirus lineage C virus in human ex vivo respiratory organ cultures

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Journal of Virology

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Tropism of and innate immune r ...... ivo respiratory organ cultures

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Denise I T Kuok

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Joanne H M Fong

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John M Nicholls

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Louisa L Y Chan

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Michael C W Chan

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Renee W Y Chan

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Sudhakar Agnihothram

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Y Guan

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P2860

Coronavirus HKU1 and other coronavirus infections in Hong Kong

Efficient replication of the novel human betacoronavirus EMC on primary human epithelium highlights its zoonotic potential

Immunogenetic mechanisms driving norovirus GII.4 antigenic variation

Time course and cellular localization of SARS-CoV nucleoprotein and RNA in lungs from fatal cases of SARS.

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Exacerbated innate host response to SARS-CoV in aged non-human primates

Induction of proinflammatory cytokines in human macrophages by influenza A (H5N1) viruses: a mechanism for the unusual severity of human disease?

Influenza virus non-structural protein 1 (NS1) disrupts interferon signaling

Identification of a new human coronavirus

Isolation of a novel coronavirus from a man with pneumonia in Saudi Arabia

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6604-6614

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10.1128/JVI.00009-13

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12

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87

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Leo Lit Man Poon

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2013-04-03T00:00:00Z

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23552422

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3676115

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