Deletions in the genomes of pseudorabies virus vaccine strains and existence of four isomers of the genomes.
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A microfluidic chamber for analysis of neuron-to-cell spread and axonal transport of an alpha-herpesvirusIn vitro analysis of transneuronal spread of an alphaherpesvirus infection in peripheral nervous system neurons.Pseudorabies virus infection alters neuronal activity and connectivity in vitroIntracellular trafficking and localization of the pseudorabies virus Us9 type II envelope protein to host and viral membranes.Efficient axonal localization of alphaherpesvirus structural proteins in cultured sympathetic neurons requires viral glycoprotein E.Neuron-to-cell spread of pseudorabies virus in a compartmented neuronal culture systemA wide extent of inter-strain diversity in virulent and vaccine strains of alphaherpesviruses.Transcriptome signature of virulent and attenuated pseudorabies virus-infected rodent brain.Genome Sequence of the Anterograde-Spread-Defective Herpes Simplex Virus 1 Strain MacIntyre.A deletion at the UL/IR junction reduces pseudorabies virus neurovirulenceGlycoprotein D-independent spread of pseudorabies virus infection in cultured peripheral nervous system neurons in a compartmented system.Use of lambda gt11 to isolate genes for two pseudorabies virus glycoproteins with homology to herpes simplex virus and varicella-zoster virus glycoproteinsThe ability of pseudorabies virus to grow in different hosts is affected by the duplication and translocation of sequences from the left end of the genome to the UL-US junctionHost cell-specific growth advantage of pseudorabies virus with a deletion in the genome sequences encoding a structural glycoproteinNucleotide sequences at recombinational junctions present in pseudorabies virus variants with an invertible L component.Errant processing and structural alterations of genomes present in a varicella-zoster virus vaccine.Proteins specified by the short unique region of the genome of pseudorabies virus play a role in the release of virions from certain cellsGlycoprotein gIII of pseudorabies virus is multifunctionalSequence of the genome ends and of the junction between the ends in concatemeric DNA of pseudorabies virus.Role of glycoprotein gIII of pseudorabies virus in virulenceGenome location and identification of functions defective in the Bartha vaccine strain of pseudorabies virus.Evolution of pseudorabies virions containing genomes with an invertible long component after repeated passage in chicken embryo fibroblasts.Genetic basis of the neurovirulence of pseudorabies virusRole of pseudorabies virus glycoprotein gI in virus release from infected cells.Role of a structural glycoprotein of pseudorabies in virus virulenceIdentification of pseudorabies virus-exposed swine with a gI glycoprotein enzyme-linked immunosorbent assay.Pseudorabies virus variant in Bartha-K61-vaccinated pigs, China, 2012.The highly conserved proline at position 438 in pseudorabies virus gH is important for regulation of membrane fusion.Analysis of viral and cellular factors influencing herpesvirus-induced nuclear envelope breakdownRetrograde, transneuronal spread of pseudorabies virus in defined neuronal circuitry of the rat brain is facilitated by gE mutations that reduce virulence.The Us9 gene product of pseudorabies virus, an alphaherpesvirus, is a phosphorylated, tail-anchored type II membrane protein.A chicken embryo eye model for the analysis of alphaherpesvirus neuronal spread and virulenceRole of pseudorabies virus Us9, a type II membrane protein, in infection of tissue culture cells and the rat nervous system.Insertions in the gG gene of pseudorabies virus reduce expression of the upstream Us3 protein and inhibit cell-to-cell spread of virus infection.The attenuated pseudorabies virus strain Bartha fails to package the tegument proteins Us3 and VP22.Corticosteroids are unable to protect against pseudorabies virus-induced tissue damage in the developing brain.Specific pseudorabies virus infection of the rat visual system requires both gI and gp63 glycoproteins.Pseudorabies virus envelope glycoprotein gI influences both neurotropism and virulence during infection of the rat visual system.Pseudorabies virus avirulent strains fail to express a major glycoprotein.Deletions in vaccine strains of pseudorabies virus and their effect on synthesis of glycoprotein gp63.
P2860
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P2860
Deletions in the genomes of pseudorabies virus vaccine strains and existence of four isomers of the genomes.
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
Deletions in the genomes of ps ...... f four isomers of the genomes.
@en
type
label
Deletions in the genomes of ps ...... f four isomers of the genomes.
@en
prefLabel
Deletions in the genomes of ps ...... f four isomers of the genomes.
@en
P2093
P2860
P1433
P1476
Deletions in the genomes of ps ...... f four isomers of the genomes.
@en
P2093
B Lomniczi
M L Blankenship
T Ben-Porat
P2860
P304
P407
P577
1984-03-01T00:00:00Z