Antiangiogenic therapy for normalization of atherosclerotic plaque vasculature: a potential strategy for plaque stabilization. - Wikidata - awgldk - TriplyDB

Antiangiogenic therapy for normalization of atherosclerotic plaque vasculature: a potential strategy for plaque stabilization.

Antiangiogenic therapy for normalization of atherosclerotic plaque vasculature: a potential strategy for plaque stabilization.

http://www.wikidata.org/entity/Q36915918

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Systemic delivery of oncolytic viruses: hopes and hurdles Contact-facilitated drug delivery with Sn2 lipase labile prodrugs optimize targeted lipid nanoparticle drug delivery Role of CX3CR1 receptor in monocyte/macrophage driven neovascularization RANTES/CCL5-induced pro-angiogenic effects depend on CCR1, CCR5 and glycosaminoglycans Antiangiogenesis strategies revisited: from starving tumors to alleviating hypoxia.Perfluorocarbon nanoparticles for physiological and molecular imaging and therapy Perspectives and opportunities for nanomedicine in the management of atherosclerosis.Vascular normalization as a therapeutic strategy for malignant and nonmalignant disease.Normalization of the vasculature for treatment of cancer and other diseases.Theragnostics for tumor and plaque angiogenesis with perfluorocarbon nanoemulsions Perfluorocarbon nanoemulsions for quantitative molecular imaging and targeted therapeutics.Normalizing tumor microenvironment to treat cancer: bench to bedside to biomarkers.VEGF inhibitors in the treatment of cerebral edema in patients with brain cancer.alphaVbeta3-targeted copper nanoparticles incorporating an Sn 2 lipase-labile fumagillin prodrug for photoacoustic neovascular imaging and treatment.The shunt problem: control of functional shunting in normal and tumour vasculature.Intralesional delivery of dendritic cells engineered to express T-bet promotes protective type 1 immunity and the normalization of the tumor microenvironment.Dopamine stabilizes tumor blood vessels by up-regulating angiopoietin 1 expression in pericytes and Kruppel-like factor-2 expression in tumor endothelial cells.Atherosclerosis: from biology to pharmacological treatment.Inflammatory angiogenesis in atherogenesis--a double-edged sword.Atherosclerotic plaque development and instability: a dual role for VEGF.Optical and multimodality molecular imaging: insights into atherosclerosis.The value of multi-slice-computed tomography coronary angiography for risk stratification.Controlling the angiogenic switch in developing atherosclerotic plaques: possible targets for therapeutic intervention.Anti-angiogenic perfluorocarbon nanoparticles for diagnosis and treatment of atherosclerosis.Registration of dynamic contrast-enhanced MRI of the common carotid artery using a fixed-frame template-based squared-difference method.AKT as locus of cancer angiogenic robustness and fragility.Thymosin β4 sustained release from poly(lactide-co-glycolide) microspheres: synthesis and implications for treatment of myocardial ischemia.Update on oncolytic viral therapy - targeting angiogenesis Role of the vasa vasorum and vascular resident stem cells in atherosclerosis.IL-8-mediated angiogenic responses of endothelial cells to lipid antigen activation of iNKT cells depend on EGFR transactivation.Coronary plaque neovascularization and hemorrhage: a potential target for plaque stabilization?Therapeutic potential of novel modulators of neovascularization.Computationally simple analysis of matched, outcome-based studies of ordinal disease states.Characterization of carotid artery plaques with USPIO-enhanced MRI: assessment of inflammation and vascularity as in vivo imaging biomarkers for plaque vulnerability.Mesenteric ischemia in patients with end-stage renal disease: a nationwide longitudinal study.CD163+ macrophages promote angiogenesis and vascular permeability accompanied by inflammation in atherosclerosis.THSD1 preserves vascular integrity and protects against intraplaque haemorrhaging in ApoE-/- mice.Erk5 inhibits endothelial migration via KLF2-dependent down-regulation of PAK1.Multifunctional regulation of angiogenesis by high-density lipoproteins.Improved plaque neovascularization following 2-year atorvastatin therapy based on contrast-enhanced ultrasonography: A pilot study.

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P2860

Antiangiogenic therapy for normalization of atherosclerotic plaque vasculature: a potential strategy for plaque stabilization.

http://www.wikidata.org/entity/Q36915918

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Nature Clinical Practice Cardiovascular Medicine

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Aloke V Finn

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Frank D Kolodgie

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Herman K Gold

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Rakesh K Jain

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Renu Virmani

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P2860

Lessons from sudden coronary death: a comprehensive morphological classification scheme for atherosclerotic lesions.

Cholesterol and phospholipid metabolism in macrophages.

Inhibition of vascular endothelial growth factor (VEGF) signaling in cancer causes loss of endothelial fenestrations, regression of tumor vessels, and appearance of basement membrane ghosts.

Healed plaque ruptures and sudden coronary death: evidence that subclinical rupture has a role in plaque progression.

Angiogenesis inhibitors endostatin or TNP-470 reduce intimal neovascularization and plaque growth in apolipoprotein E-deficient mice.

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491-502

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10.1038/NCPCARDIO0979

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9

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17712362

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atherosclerosis