Initiation and termination of duck hepatitis B virus DNA synthesis during virus maturation
about
Identification and characterization of avihepadnaviruses isolated from exotic anseriformes maintained in captivityMolecular biology of hepatitis B virus infectionMechanism of inhibition of duck hepatitis B virus polymerase by (-)-beta-L-2',3'-dideoxy-3'-thiacytidineProperties of monoclonal antibodies directed against hepatitis B virus polymerase protein.Mapping of the hepatitis B virus reverse transcriptase TP and RT domains by transcomplementation for nucleotide priming and by protein-protein interactionEffect of antiviral treatment with entecavir on age- and dose-related outcomes of duck hepatitis B virus infection.Infection of ducklings with virus particles containing linear double-stranded duck hepatitis B virus DNA: illegitimate replication and reversionInteraction between hepatitis B virus core protein and reverse transcriptase.Human hepatitis B virus polymerase interacts with the molecular chaperonin Hsp60.Hepatitis B virus biology.Secretion of genome-free hepatitis B virus--single strand blocking model for virion morphogenesis of para-retrovirus.Conditional replication of duck hepatitis B virus in hepatoma cells.Duck hepatitis B virus virion secretion requires a double-stranded DNA genome.cis-Acting sequences 5E, M, and 3E interact to contribute to primer translocation and circularization during reverse transcription of avian hepadnavirus DNAAnimal models and the molecular biology of hepadnavirus infectionComplementarity between epsilon and phi sequences in pregenomic RNA influences hepatitis B virus replication efficiency.Hepadnavirus Genome Replication and Persistence.Mutations in the epsilon sequences of human hepatitis B virus affect both RNA encapsidation and reverse transcriptionIllegitimate replication of linear hepadnavirus DNA through nonhomologous recombinationNucleotide priming and reverse transcriptase activity of hepatitis B virus polymerase expressed in insect cellsA splice hepadnavirus RNA that is essential for virus replication.Transcomplementation of nucleotide priming and reverse transcription between independently expressed TP and RT domains of the hepatitis B virus reverse transcriptase.Sequence-independent RNA cleavages generate the primers for plus strand DNA synthesis in hepatitis B viruses: implications for other reverse transcribing elements.Expression of functional hepatitis B virus polymerase in yeast reveals it to be the sole viral protein required for correct initiation of reverse transcription.Reverse transcription in hepatitis B viruses is primed by a tyrosine residue of the polymeraseThe carbocyclic analog of 2'-deoxyguanosine induces a prolonged inhibition of duck hepatitis B virus DNA synthesis in primary hepatocyte cultures and in the liverHepadnavirus reverse transcription initiates within the stem-loop of the RNA packaging signal and employs a novel strand transfer.Novel mechanism for reverse transcription in hepatitis B virusesDetection of DNA polymerase activities associated with purified duck hepatitis B virus core particles by using an activity gel assayAvian hepatitis B viruses: molecular and cellular biology, phylogenesis, and host tropism.Preferred translation of human hepatitis B virus polymerase from core protein- but not from precore protein-specific transcript.Identification of a signal necessary for initiation of reverse transcription of the hepadnavirus genomeIdentification and characterization of the woodchuck hepatitis virus origin of DNA replicationSynthesis of hepadnavirus particles that contain replication-defective duck hepatitis B virus genomes in cultured HuH7 cells.cis rescue of a mutated reverse transcriptase gene of human hepatitis B virus by creation of an internal ATG.Molecular analysis of the function of direct repeats and a polypurine tract for plus-strand DNA priming in woodchuck hepatitis virus.Template switches during plus-strand DNA synthesis of duck hepatitis B virus are influenced by the base composition of the minus-strand terminal redundancy.Changing the site of initiation of plus-strand DNA synthesis inhibits the subsequent template switch during replication of a hepadnavirus.Mutations that increase in situ priming also decrease circularization for duck hepatitis B virus.Analysis of duck hepatitis B virus reverse transcription indicates a common mechanism for the two template switches during plus-strand DNA synthesis.
P2860
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P2860
Initiation and termination of duck hepatitis B virus DNA synthesis during virus maturation
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
Initiation and termination of ...... thesis during virus maturation
@en
type
label
Initiation and termination of ...... thesis during virus maturation
@en
prefLabel
Initiation and termination of ...... thesis during virus maturation
@en
P2093
P2860
P1433
P1476
Initiation and termination of ...... thesis during virus maturation
@en
P2093
P2860
P304
P407
P577
1987-12-01T00:00:00Z