Genetic disruption of Abl nuclear import reduces renal apoptosis in a mouse model of cisplatin-induced nephrotoxicity
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A Role for Tubular Necroptosis in Cisplatin-Induced AKIPathophysiology of cisplatin-induced acute kidney injury.Persistent inhibition of ABL tyrosine kinase causes enhanced apoptotic response to TRAIL and disrupts the pro-apoptotic effect of chloroquine.The kinase ABL phosphorylates the microprocessor subunit DGCR8 to stimulate primary microRNA processing in response to DNA damage.DNA damage response in cisplatin-induced nephrotoxicity.Autophagy in acute kidney injuryThe capable ABL: what is its biological function?DNA damage response in nephrotoxic and ischemic kidney injury.Beneficial Effects of Myo-Inositol Oxygenase Deficiency in Cisplatin-Induced AKI.EnABLing microprocessor for apoptosis.Amelioration of cisplatin-induced acute kidney injury by recombinant neutrophil gelatinase-associated lipocalinDetection of dietary plant-based small RNAs in animals.Dexmedetomidine protects against cisplatin-induced acute kidney injury in mice through regulating apoptosis and inflammation.Exogenous spermidine ameliorates tubular necrosis during cisplatin nephrotoxicity
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P2860
Genetic disruption of Abl nuclear import reduces renal apoptosis in a mouse model of cisplatin-induced nephrotoxicity
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2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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2013年论文
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2013年论文
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name
Genetic disruption of Abl nucl ...... splatin-induced nephrotoxicity
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type
label
Genetic disruption of Abl nucl ...... splatin-induced nephrotoxicity
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prefLabel
Genetic disruption of Abl nucl ...... splatin-induced nephrotoxicity
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P2093
P2860
P356
P1476
Genetic disruption of Abl nucl ...... splatin-induced nephrotoxicity
@en
P2093
P2860
P2888
P304
P356
10.1038/CDD.2013.42
P577
2013-05-10T00:00:00Z