The role of cation-dependent chloride transporters in neuropathic pain following spinal cord injury.
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NMDA receptor activity downregulates KCC2 resulting in depolarizing GABAA receptor-mediated currentsExpanding Spectrum of Sodium Potassium Chloride Co-transporters in the Pathophysiology of DiseasesMetaplasticity and behavior: how training and inflammation affect plastic potential within the spinal cord and recovery after injurySerotonergic modulation of post-synaptic inhibition and locomotor alternating pattern in the spinal cordMicroglia and spinal cord synaptic plasticity in persistent painCurrent view on the functional regulation of the neuronal K(+)-Cl(-) cotransporter KCC2Spinal Plasticity and Behavior: BDNF-Induced Neuromodulation in Uninjured and Injured Spinal CordSmall-molecule screen identifies inhibitors of the neuronal K-Cl cotransporter KCC2Folic Acid Modulates Matrix Metalloproteinase-2 Expression, Alleviates Neuropathic Pain, and Improves Functional Recovery in Spinal Cord-Injured Rats.Aquaporins in spinal cord injury: the janus face of aquaporin 4Pathogenesis of spinal cord injury induced edema and neuropathic pain: expression of multiple isoforms of wnk1.Chloride extrusion enhancers as novel therapeutics for neurological diseasesActivation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2.Chloride Homeostasis Critically Regulates Synaptic NMDA Receptor Activity in Neuropathic Pain.BDNF/TRK/KCC2 pathway in nicotine withdrawal-induced hyperalgesiaBilateral transient changes in thalamic nucleus ventroposterior lateralis after thoracic hemisection in the ratMetabolomics uncovers dietary omega-3 fatty acid-derived metabolites implicated in anti-nociceptive responses after experimental spinal cord injury.Differential effects on KCC2 expression and spasticity of ALS and traumatic injuries to motoneurons.GABA and central neuropathic pain following spinal cord injurySpatial and temporal dynamics in the ionic driving force for GABA(A) receptors.In Vivo application of RNAi to study pain.Emerging role of WNK1 in pathologic central nervous system signaling.The multifunctional and multi-system influence of Ghrelin in the treatment of diabetic and spinal cord injury induced Neuropathy.Neuropathic pain: role of inflammation, immune response, and ion channel activity in central injury mechanisms.Spinal cord injury induced neuropathic pain: Molecular targets and therapeutic approaches.The K(+)-Cl(-) Cotransporter KCC2 and Chloride Homeostasis: Potential Therapeutic Target in Acute Central Nervous System Injury.Plasticity in respiratory motor neurons in response to reduced synaptic inputs: A form of homeostatic plasticity in respiratory control?Rehabilitation Strategies after Spinal Cord Injury: Inquiry into the Mechanisms of Success and Failure.The Role of K(+)-Cl(-)-Cotransporter-2 in Neuropathic Pain.Role of Matrix Metalloproteinases 2 in Spinal Cord Injury-Induced Neuropathic Pain.The 5-HT2A receptor potassium-chloride cotransporter 2 signaling pathway in a rat incision pain model.Further optimization of the K-Cl cotransporter KCC2 antagonist ML077: development of a highly selective and more potent in vitro probe.Role of NKCC1 and KCC2 in the development of chronic neuropathic pain following spinal cord injury.Sigma-1 Receptor in Motoneuron Disease.BDNF Induced by Treadmill Training Contributes to the Suppression of Spasticity and Allodynia After Spinal Cord Injury via Upregulation of KCC2.When Pain Hurts: Nociceptive Stimulation Induces a State of Maladaptive Plasticity and Impairs Recovery after Spinal Cord Injury.Pretreatment with AQP4 and NKCC1 Inhibitors Concurrently Attenuated Spinal Cord Edema and Tissue Damage after Spinal Cord Injury in Rats.Acute spinal cord injury (SCI) transforms how GABA affects nociceptive sensitization.Neuropathic pain after chronic nerve constriction may not correlate with chloride dysregulation in mouse trigeminal nucleus caudalis neurons.Metaplasticity within the spinal cord: Evidence brain-derived neurotrophic factor (BDNF), tumor necrosis factor (TNF), and alterations in GABA function (ionic plasticity) modulate pain and the capacity to learn.
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The role of cation-dependent chloride transporters in neuropathic pain following spinal cord injury.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
The role of cation-dependent c ...... following spinal cord injury.
@en
type
label
The role of cation-dependent c ...... following spinal cord injury.
@en
prefLabel
The role of cation-dependent c ...... following spinal cord injury.
@en
P2093
P2860
P356
P1433
P1476
The role of cation-dependent c ...... following spinal cord injury.
@en
P2093
Bradley Allcock
Christopher Baggott
Dandan Sun
Daniel Resnick
Gurwattan Miranpuri
Jessica Tilghman
Samuel W Cramer
Sharad Rajpal
P2860
P2888
P356
10.1186/1744-8069-4-36
P577
2008-09-17T00:00:00Z
P5875
P6179
1017042989