Robust protein nitration contributes to acetaminophen-induced mitochondrial dysfunction and acute liver injury - Wikidata - awgldk - TriplyDB

Robust protein nitration contributes to acetaminophen-induced mitochondrial dysfunction and acute liver injury

Robust protein nitration contributes to acetaminophen-induced mitochondrial dysfunction and acute liver injury

http://www.wikidata.org/entity/Q36923111

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Mitochondrial dysfunction and tissue injury by alcohol, high fat, nonalcoholic substances and pathological conditions through post-translational protein modifications Functional roles of protein nitration in acute and chronic liver diseases Assessment of a Flavone-Polysaccharide Based Prescription for Treating Duck Virus Hepatitis Ethanol and Acetaminophen Synergistically Induce Hepatic Aggregation and TCH346-Insensitive Nuclear Translocation of GAPDH Binge alcohol promotes hypoxic liver injury through a CYP2E1-HIF-1α-dependent apoptosis pathway in mice and humans Zidovudine (AZT) and hepatic lipid accumulation: implication of inflammation, oxidative and endoplasmic reticulum stress mediators.Ethanol extract from portulaca oleracea L. attenuated acetaminophen-induced mice liver injury Epigallocatechin 3-gallate ameliorates bile duct ligation induced liver injury in mice by modulation of mitochondrial oxidative stress and inflammation.Identifying the ionically bound cell wall and intracellular glycoside hydrolases in late growth stage Arabidopsis stems: implications for the genetic engineering of bioenergy crops Increased Sensitivity to Binge Alcohol-Induced Gut Leakiness and Inflammatory Liver Disease in HIV Transgenic Rats.Nitric oxide in liver diseases.Selective chemoprecipitation to enrich nitropeptides from complex proteomes for mass-spectrometric analysis Critical role of c-jun N-terminal protein kinase in promoting mitochondrial dysfunction and acute liver injury Synergistic Interaction of Light Alcohol Administration in the Presence of Mild Iron Overload in a Mouse Model of Liver Injury: Involvement of Triosephosphate Isomerase Nitration and Inactivation Mitochondrial protein adducts formation and mitochondrial dysfunction during N-acetyl-m-aminophenol (AMAP)-induced hepatotoxicity in primary human hepatocytes Role of CYP2E1 in mitochondrial dysfunction and hepatic tissue injury in alcoholic and non-alcoholic diseases.Cytochrome P450-2E1 promotes aging-related hepatic steatosis, apoptosis and fibrosis through increased nitroxidative stress.Mitochondrial dysfunction and cell death in neurodegenerative diseases through nitroxidative stress Protective Activity of Total Polyphenols from Genista quadriflora Munby and Teucrium polium geyrii Maire in Acetaminophen-Induced Hepatotoxicity in Rats.The neuronal nitric oxide synthase inhibitor NANT blocks acetaminophen toxicity and protein nitration in freshly isolated hepatocytes.Glutathione defense mechanism in liver injury: insights from animal models.A novel upregulation of glutathione peroxidase 1 by knockout of liver-regenerating protein Reg3β aggravates acetaminophen-induced hepatic protein nitration.CYP2E1 potentiates binge alcohol-induced gut leakiness, steatohepatitis, and apoptosis.Effects of acetaminophen on mitochondrial complex I activity in the rat liver and kidney: a PET study with 18F-BCPP-BF.Application of urine proteomics for biomarker discovery in drug-induced liver injury.Trifluoperazine inhibits acetaminophen-induced hepatotoxicity and hepatic reactive nitrogen formation in mice and in freshly isolated hepatocytes.Antcin H Protects Against Acute Liver Injury Through Disruption of the Interaction of c-Jun-N-Terminal Kinase with Mitochondria.Mitochondria-targeted ubiquinone (MitoQ) enhances acetaldehyde clearance by reversing alcohol-induced posttranslational modification of aldehyde dehydrogenase 2: A molecular mechanism of protection against alcoholic liver disease.Proteomic analysis of acetaminophen-induced hepatotoxicity and identification of heme oxygenase 1 as a potential plasma biomarker of liver injury.Cytochrome P450-2E1 is involved in aging-related kidney damage in mice through increased nitroxidative stress.Preventive effects of indole-3-carbinol against alcohol-induced liver injury in mice via antioxidant, anti-inflammatory, and anti-apoptotic mechanisms: Role of gut-liver-adipose tissue axis.Targeting mitochondria with methylene blue protects mice against acetaminophen-induced liver injury.Glutathione S-transferase A1 (GSTA1) as a marker of acetaminophen-induced hepatocyte injury in vitro.Apocynum venetum Attenuates Acetaminophen-Induced Liver Injury in Mice.Exogenous exosomes from mice with acetaminophen-induced liver injury promote toxicity in the recipient hepatocytes and mice

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P2860

Robust protein nitration contributes to acetaminophen-induced mitochondrial dysfunction and acute liver injury

http://www.wikidata.org/entity/Q36923111

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2013 nî lūn-bûn

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2013年の論文

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2013年論文

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J.FREERADBIOMED.2013.02.018

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Free Radical Biology and Medicine

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Robust protein nitration contr ...... unction and acute liver injury

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Atrayee Banerjee

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Byoung-Joon Song

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James P Hardwick

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Mohamed A Abdelmegeed

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Sehwan Jang

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P2860

Contribution of human manganese superoxide dismutase tyrosine 34 to structure and catalysis

Crystal structure of the antioxidant enzyme glutathione reductase inactivated by peroxynitrite

Lifetime nonnarcotic analgesic use and decline in renal function in women

Mechanisms of hepatotoxicity

Selective protein arylation and acetaminophen-induced hepatotoxicity.

Updates on acetaminophen toxicity.

Identification of the hepatic protein targets of reactive metabolites of acetaminophen in vivo in mice using two-dimensional gel electrophoresis and mass spectrometry.

Critical role of c-Jun N-terminal protein kinase activation in troglitazone-induced apoptosis of human HepG2 hepatoma cells.

A rapid and selective mass spectrometric method for the identification of nitrated proteins.

Mechanisms of MDMA (ecstasy)-induced oxidative stress, mitochondrial dysfunction, and organ damage.

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211-222

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60

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