Intramyocardial VEGF-B167 gene delivery delays the progression towards congestive failure in dogs with pacing-induced dilated cardiomyopathy. - Wikidata - awgldk - TriplyDB

Intramyocardial VEGF-B167 gene delivery delays the progression towards congestive failure in dogs with pacing-induced dilated cardiomyopathy.

Intramyocardial VEGF-B167 gene delivery delays the progression towards congestive failure in dogs with pacing-induced dilated cardiomyopathy.

http://www.wikidata.org/entity/Q36934608

about

Vascular endothelial growth factor-B in physiology and disease Targeting myocardial remodelling to develop novel therapies for heart failure: a position paper from the Working Group on Myocardial Function of the European Society of Cardiology VEGF gene therapy: therapeutic angiogenesis in the clinic and beyond Ablation of the cardiac-specific gene leucine-rich repeat containing 10 (Lrrc10) results in dilated cardiomyopathy Small and large animal models in cardiac contraction research: advantages and disadvantages Cancer therapy-induced cardiotoxicity: basic mechanisms and potential cardioprotective therapies.Vascular endothelial growth factor-B gene transfer exacerbates retinal and choroidal neovascularization and vasopermeability without promoting inflammation.Resveratrol treatment rescues neurovascular coupling in aged mice: role of improved cerebromicrovascular endothelial function and downregulation of NADPH oxidase.Sizing up models of heart failure: Proteomics from flies to humans.Resveratrol encapsulated in novel fusogenic liposomes activates Nrf2 and attenuates oxidative stress in cerebromicrovascular endothelial cells from aged rats.The vascular marker soluble fms-like tyrosine kinase 1 is associated with disease severity and adverse outcomes in chronic heart failure.Intracoronary Cytoprotective Gene Therapy: A Study of VEGF-B167 in a Pre-Clinical Animal Model of Dilated Cardiomyopathy Disruption of Nrf2 signaling impairs angiogenic capacity of endothelial cells: implications for microvascular aging.AAV9-mediated VEGF-B gene transfer improves systolic function in progressive left ventricular hypertrophy.Progress in gene therapy of dystrophic heart disease.Myocyte repolarization modulates myocardial function in aging dogs Ionizing radiation promotes the acquisition of a senescence-associated secretory phenotype and impairs angiogenic capacity in cerebromicrovascular endothelial cells: role of increased DNA damage and decreased DNA repair capacity in microvascular rad The NO/ONOO-cycle as the central cause of heart failure.VEGF-B gene therapy inhibits doxorubicin-induced cardiotoxicity by endothelial protection.Role of mitochondrial dysfunction and altered autophagy in cardiovascular aging and disease: from mechanisms to therapeutics.Beneficial effects of acute inhibition of the oxidative pentose phosphate pathway in the failing heart.VEGF-B-induced vascular growth leads to metabolic reprogramming and ischemia resistance in the heart.Heart failure associated with sunitinib: lessons learned from animal models.Cardiac gene therapy in large animals: bridge from bench to bedside.Intercellular communication lessons in heart failure.Use of Adeno-Associated Virus Vector for Cardiac Gene Delivery in Large-Animal Surgical Models of Heart Failure.Cardiac AAV9 Gene Delivery Strategies in Adult Canines: Assessment by Long-term Serial SPECT Imaging of Sodium Iodide Symporter Expression.VEGF-C/VEGFR-3 pathway promotes myocyte hypertrophy and survival in the infarcted myocardium.VEGFB/VEGFR1-Induced Expansion of Adipose Vasculature Counteracts Obesity and Related Metabolic Complications.Integrated approaches to spatiotemporally directing angiogenesis in host and engineered tissues.Genetic and ethnic modulation of cardiovascular toxicity of vascular endothelial growth factor inhibitors.Loss of VEGFB and its signaling in the diabetic heart is associated with increased cell death signaling.The Anti-Cancer Multikinase Inhibitor Sorafenib Impairs Cardiac Contractility by Reducing Phospholamban Phosphorylation and Sarcoplasmic Calcium Transients.Vascular Endothelial Growth Factor B and Its Signaling.Cardiotoxicity with vascular endothelial growth factor inhibitor therapy Treatment with placental growth factor attenuates myocardial ischemia/reperfusion injury

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P2860

Intramyocardial VEGF-B167 gene delivery delays the progression towards congestive failure in dogs with pacing-induced dilated cardiomyopathy.

http://www.wikidata.org/entity/Q36934608

description

2010 nî lūn-bûn

@nan

2010年の論文

@ja

2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

2010年论文

@zh

2010年论文

@zh-cn

name

Intramyocardial VEGF-B167 gene ...... nduced dilated cardiomyopathy.

@en

type

label

Intramyocardial VEGF-B167 gene ...... nduced dilated cardiomyopathy.

@en

prefLabel

Intramyocardial VEGF-B167 gene ...... nduced dilated cardiomyopathy.

@en

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CIRCRESAHA.110.220855

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Circulation Research

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Intramyocardial VEGF-B167 gene ...... nduced dilated cardiomyopathy.

@en

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Anna Csiszar

http://www.w3.org/2001/XMLSchema#string

Fabio A Recchia

http://www.w3.org/2001/XMLSchema#string

John G Edwards

http://www.w3.org/2001/XMLSchema#string

Khaled Qanud

http://www.w3.org/2001/XMLSchema#string

Lorena Zentilin

http://www.w3.org/2001/XMLSchema#string

Martino Pepe

http://www.w3.org/2001/XMLSchema#string

Mauro Giacca

http://www.w3.org/2001/XMLSchema#string

Mohammed Mamdani

http://www.w3.org/2001/XMLSchema#string

Serena Zacchigna

http://www.w3.org/2001/XMLSchema#string

Silvia Moimas

http://www.w3.org/2001/XMLSchema#string

P2860

VEGF-B inhibits apoptosis via VEGFR-1-mediated suppression of the expression of BH3-only protein genes in mice and rats

Vascular endothelial growth factor B, a novel growth factor for endothelial cells

VEGF-B is dispensable for blood vessel growth but critical for their survival, and VEGF-B targeting inhibits pathological angiogenesis

The biology of VEGF and its receptors

Mice lacking the vascular endothelial growth factor-B gene (Vegfb) have smaller hearts, dysfunctional coronary vasculature, and impaired recovery from cardiac ischemia

Exogenous nitric oxide reduces glucose transporters translocation and lactate production in ischemic myocardium in vivo

Adeno-associated virus-mediated transduction of VEGF165 improves cardiac tissue viability and functional recovery after permanent coronary occlusion in conscious dogs.

Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy.

A mechanistic role for cardiac myocyte apoptosis in heart failure.

Cause of death: suicide.

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106

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20431055

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