Nitric-oxide synthase knockout modulates Ca²⁺-sensing receptor expression and signaling in mouse mesenteric arteries
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Inhibition of Excessive Cell Proliferation by Calcilytics in Idiopathic Pulmonary Arterial Hypertension.The calcilytics Calhex-231 and NPS 2143 and the calcimimetic Calindol reduce vascular reactivity via inhibition of voltage-gated Ca2+ channels.Stimulation of calcium-sensing receptors induces endothelium-dependent vasorelaxations via nitric oxide production and activation of IKCa channels.L-Arginine Supplementation in Type II Diabetic Rats Preserves Renal Function and Improves Insulin Sensitivity by Altering the Nitric Oxide Pathway.Heteromeric TRPV4/TRPC1 channels mediate calcium-sensing receptor-induced nitric oxide production and vasorelaxation in rabbit mesenteric arteries.Calcimimetic R-568 vasodilatory effect on mesenteric vascular beds from normotensive (WKY) and spontaneously hypertensive (SHR) rats. Potential involvement of vascular smooth muscle cells (vSMCs)
P2860
Nitric-oxide synthase knockout modulates Ca²⁺-sensing receptor expression and signaling in mouse mesenteric arteries
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2013 nî lūn-bûn
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2013年の論文
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name
Nitric-oxide synthase knockout ...... g in mouse mesenteric arteries
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type
label
Nitric-oxide synthase knockout ...... g in mouse mesenteric arteries
@en
prefLabel
Nitric-oxide synthase knockout ...... g in mouse mesenteric arteries
@en
P2093
P2860
P356
P1476
Nitric-oxide synthase knockout ...... g in mouse mesenteric arteries
@en
P2093
Cicely L Williams
Emmanuel M Awumey
Lakeesha E Bridges
P2860
P356
10.1124/JPET.113.205534
P407
P50
P577
2013-05-02T00:00:00Z