NKG2D is critical for NK cell activation in host defense against Pseudomonas aeruginosa respiratory infection.
about
NKG2D ligands as therapeutic targetsImpact of the MICA-129Met/Val Dimorphism on NKG2D-Mediated Biological Functions and Disease RisksNatural killer cells in infection and inflammation of the lungCCR7 deficiency leads to leukocyte activation and increased clearance in response to pulmonary Pseudomonas aeruginosa infectionPseudomonas aeruginosa eliminates natural killer cells via phagocytosis-induced apoptosis.Role of major histocompatibility complex variation in graft-versus-host disease after hematopoietic cell transplantation.Sepsis-induced immunosuppression: from cellular dysfunctions to immunotherapy.Natural Killer Cell Functions during the Innate Immune Response to Pathogenic Streptococci.Reduced mucosal associated invariant T-cells are associated with increased disease severity and Pseudomonas aeruginosa infection in cystic fibrosis.Immune mechanisms and the impact of the disrupted lung microbiome in chronic bacterial lung infection and bronchiectasis.Natural killer cells from patients with chronic rhinosinusitis have impaired effector functionsInnate immune responses to Pseudomonas aeruginosa infection.NKG2D mediates NK cell hyperresponsiveness and influenza-induced pathologies in a mouse model of chronic obstructive pulmonary disease.Autoantibody Profiles in Collagen Disease Patients with Interstitial Lung Disease (ILD): Antibodies to Major Histocompatibility Complex Class I-Related Chain A (MICA) as Markers of ILD.The MICA-129 dimorphism affects NKG2D signaling and outcome of hematopoietic stem cell transplantation.The MICA-129Met/Val dimorphism affects plasma membrane expression and shedding of the NKG2D ligand MICAHigh Mobility Group Box-1 mediates hyperoxia-induced impairment of Pseudomonas aeruginosa clearance and inflammatory lung injury in mice.Sustained CTL activation by murine pulmonary epithelial cells promotes the development of COPD-like disease.NK cells are effectors for resolvin E1 in the timely resolution of allergic airway inflammation.Role of CC chemokine receptor 4 in natural killer cell activation during acute cigarette smoke exposureCandida albicans airway exposure primes the lung innate immune response against Pseudomonas aeruginosa infection through innate lymphoid cell recruitment and interleukin-22-associated mucosal responseNatural killer (NK) and NK-like cells at mucosal epithelia: Mediators of anti-microbial defense and maintenance of tissue integrity.Conventional NK cells can produce IL-22 and promote host defense in Klebsiella pneumoniae pneumonia.The role of natural killer cells in sepsis.Natural killer (NK) cells in antibacterial innate immunity: angels or devils?Effect of Clarithromycin on the Expression of UL16-Binding Protein 2 in Human Cells.
P2860
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P2860
NKG2D is critical for NK cell activation in host defense against Pseudomonas aeruginosa respiratory infection.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
NKG2D is critical for NK cell ...... uginosa respiratory infection.
@en
type
label
NKG2D is critical for NK cell ...... uginosa respiratory infection.
@en
prefLabel
NKG2D is critical for NK cell ...... uginosa respiratory infection.
@en
P2093
P2860
P1476
NKG2D is critical for NK cell ...... uginosa respiratory infection.
@en
P2093
Bryan L Eppert
Daniel J Hassett
Gregory T Motz
Michael T Borchers
Scott C Wesselkamper
P2860
P304
P356
10.4049/JIMMUNOL.181.8.5481
P407
P577
2008-10-01T00:00:00Z