Recessive constitutive mutant Chinese hamster ovary cells (CHO-K1) with an altered A system for amino acid transport and the mechanism of gene regulation of the A system.
about
The adaptive regulation of amino acid transport system A is associated to changes in ATA2 expression.The role of system A for neutral amino acid transport in the regulation of cell volume.alar4, a constitutive mutant of the A system for amino acid transport, has increased abundance of the Na+,K+-ATPase and mRNA for alpha 1 subunit of this enzyme.Control of A-system amino acid transport by a second regulatory gene R2 in Chinese hamster ovary cells CHO-K1 and the possible connection of this gene with insulin activityEvidence for a regulatory protein involved in the increased activity of system A for neutral amino acid transport in osmotically stressed mammalian cells.SNAT2 amino acid transporter is regulated by amino acids of the SLC6 gamma-aminobutyric acid transporter subfamily in neocortical neurons and may play no role in delivering glutamine for glutamatergic transmission.Evidence for coordinate regulation of the A system for amino acid transport and the mRNA for the alpha 1 subunit of the Na+,K(+)-ATPase gene in Chinese hamster ovary cellsAnalysis of the genes involved in the insulin transmembrane mitogenic signal in Chinese hamster ovary cells, CHO-K1, utilizing insulin-independent mutantsEnhancement in amount of P1 (hsp60) in mutants of Chinese hamster ovary (CHO-K1) cells exhibiting increases in the A system of amino acid transport.Regulatory and molecular aspects of mammalian amino acid transport.Cytoskeletal-dependent activation of system A for neutral amino acid transport in osmotically stressed mammalian cells: a role for system A in the intracellular accumulation of osmolytes.Induction of high affinity glutamate transport activity by amino acid deprivation in renal epithelial cells does not involve an increase in the amount of transporter protein.Two membrane-bound proteins associated with alanine resistance and increased A-system amino acid transport in mutants of CHO-K1.Hyperosmolarity leads to an increase in derepressed system A activity in the renal epithelial cell line NBL-1.Membrane potential and amino acid transport in a mutant Chinese hamster ovary cell line.Up-regulation of system A activity in the regenerating rat liver.Amino acid uptake by liver of genetically obese Zucker rats.Mechanisms implicated in the response of system a to hypertonic stress and amino acid deprivation still can be different.New mutations and phenotypes associated with glutamate and aspartate transport in Chinese hamster ovary (CHO-K1) cells.Distinct sensor pathways in the hierarchical control of SNAT2, a putative amino acid transceptor, by amino acid availability.
P2860
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P2860
Recessive constitutive mutant Chinese hamster ovary cells (CHO-K1) with an altered A system for amino acid transport and the mechanism of gene regulation of the A system.
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
Recessive constitutive mutant ...... ne regulation of the A system.
@en
type
label
Recessive constitutive mutant ...... ne regulation of the A system.
@en
prefLabel
Recessive constitutive mutant ...... ne regulation of the A system.
@en
P2860
P356
P1476
Recessive constitutive mutant ...... ne regulation of the A system.
@en
P2093
Englesberg E
P2860
P304
P356
10.1128/MCB.4.4.799
P407
P577
1984-04-01T00:00:00Z