Chronic interleukin-1 exposure drives haematopoietic stem cells towards precocious myeloid differentiation at the expense of self-renewal
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Stress and Non-Stress Roles of Inflammatory Signals during HSC Emergence and MaintenanceInflamm-Aging of Hematopoiesis, Hematopoietic Stem Cells, and the Bone Marrow MicroenvironmentInhibition of interleukin-1 signaling enhances elimination of tyrosine kinase inhibitor-treated CML stem cells.Fetal and neonatal hematopoietic progenitors are functionally and transcriptionally resistant to Flt3-ITD mutations.Endothelial-specific inhibition of NF-κB enhances functional haematopoiesis.The Role of the Bone Marrow Stromal Compartment in the Hematopoietic Response to Microbial Infections.Applying nanomedicine in maladaptive inflammation and angiogenesis.Rejuvenating Strategies for Stem Cell-Based Therapies in Aging.ISSLS PRIZE IN BASIC SCIENCE 2017: Intervertebral disc/bone marrow cross-talk with Modic changes.Regulation of normal and leukemic stem cells through cytokine signaling and the microenvironment.DNA-damage response in hematopoietic stem cells: an evolutionary trade-off between blood regeneration and leukemia suppression.Mesenchymal Stromal Cell-derived Extracellular Vesicles Promote Myeloid-biased Multipotent Hematopoietic Progenitor Expansion via Toll-Like Receptor Engagement.Inflammatory response of disc cells against Propionibacterium acnes depends on the presence of lumbar Modic changes.Inflammation: a key regulator of hematopoietic stem cell fate in health and disease.Infection-adapted emergency hematopoiesis promotes visceral leishmaniasis.α-Toxin Regulates Local Granulocyte Expansion from Hematopoietic Stem and Progenitor Cells in Staphylococcus aureus-Infected Wounds.Map3k8 controls granulocyte colony-stimulating factor production and neutrophil precursor proliferation in lipopolysaccharide-induced emergency granulopoiesis.Identification of Interleukin-1 by Functional Screening as a Key Mediator of Cellular Expansion and Disease Progression in Acute Myeloid Leukemia.Chronic Infection Depletes Hematopoietic Stem Cells through Stress-Induced Terminal Differentiation.Myeloid progenitor cluster formation drives emergency and leukaemic myelopoiesis.Senescence-Inflammatory Regulation of Reparative Cellular Reprogramming in Aging and Cancer.Inflammatory Signaling Pathways in Preleukemic and Leukemic Stem Cells.Function and regulation of IL-1α in inflammatory diseases and cancer.Alternatively spliced Fibronectin Extra Domain A is required for hemangiogenic recovery upon bone marrow chemotherapy.The DNA Methylcytosine Dioxygenase Tet2 Sustains Immunosuppressive Function of Tumor-Infiltrating Myeloid Cells to Promote Melanoma Progression.IL-1β enables CNS access to CCR2hi monocytes and the generation of pathogenic cells through GM-CSF released by CNS endothelial cells.Circulating Progenitor Cells Correlate with Memory, Posterior Cortical Thickness, and Hippocampal Perfusion.Regulation of myelopoiesis by proinflammatory cytokines in infectious diseases.Senescence and aging: Causes, consequences, and therapeutic avenues.Pharmacodynamic Monitoring Predicts Outcomes of CCR5 Blockade as Graft-versus-Host Disease Prophylaxis.Persistent injury-associated anemia: the role of the bone marrow microenvironment.Modulation of Myelopoiesis Progenitors Is an Integral Component of Trained Immunity.TRAF6 Mediates Basal Activation of NF-κB Necessary for Hematopoietic Stem Cell Homeostasis.Genetic rescue of lineage-balanced blood cell production reveals a crucial role for STAT3 antiinflammatory activity in hematopoiesis.Bone marrow lympho-myeloid malfunction in obesity requires precursor cell-autonomous TLR4.NR4A1 and NR4A3 restrict HSC proliferation via reciprocal regulation of C/EBPα and inflammatory signaling.Analysis of Murine Hematopoietic Stem Cell Proliferation During Inflammation.Biological Age and Circulating Progenitor Cell Levels as Predictors Heart Disease Events.Hematopoietic Stem Cells but Not Multipotent Progenitors Drive Erythropoiesis during Chronic Erythroid Stress in EPO Transgenic Mice."Social Life" of Senescent Cells: What Is SASP and Why Study It?
P2860
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P2860
Chronic interleukin-1 exposure drives haematopoietic stem cells towards precocious myeloid differentiation at the expense of self-renewal
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Chronic interleukin-1 exposure ...... at the expense of self-renewal
@en
type
label
Chronic interleukin-1 exposure ...... at the expense of self-renewal
@en
prefLabel
Chronic interleukin-1 exposure ...... at the expense of self-renewal
@en
P2093
P2860
P50
P356
P1433
P1476
Chronic interleukin-1 exposure ...... at the expense of self-renewal
@en
P2093
Britta Will
Chih Peng Chin
Cristina Mirantes-Barbeito
Eric M Pietras
José-Marc Techner
Larisa V Kovtonyuk
Ranjani Lakshminarasimhan
SiYi Zhang
Ulrich Steidl
P2860
P2888
P304
P356
10.1038/NCB3346
P577
2016-04-25T00:00:00Z