Excess HB-EGF, which promotes VEGF signaling, leads to hydrocephalus

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EGFR transactivation contributes to neuroinflammation in Streptococcus suis meningitis.VEGF Signaling in Neurological Disorders.

P2860

Q37350200-74C25738-9E2C-4CBE-BCDA-ED107361F024 Q47860045-2B2D64D1-2ABE-41E8-9C2D-DAF6A930B7CA

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Excess HB-EGF, which promotes VEGF signaling, leads to hydrocephalus

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http://www.wikidata.org/entity/Q36950613

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

Excess HB-EGF, which promotes VEGF signaling, leads to hydrocephalus

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type

Item

label

Excess HB-EGF, which promotes VEGF signaling, leads to hydrocephalus

@en

prefLabel

Excess HB-EGF, which promotes VEGF signaling, leads to hydrocephalus

@en

P1476

Excess HB-EGF, which promotes VEGF signaling, leads to hydrocephalus

@en

P2093

Feng C Zhou

http://www.w3.org/2001/XMLSchema#string

Johanna Sandlund

http://www.w3.org/2001/XMLSchema#string

Joon W Shim

http://www.w3.org/2001/XMLSchema#string

Joseph R Madsen

http://www.w3.org/2001/XMLSchema#string

Michael Klagsbrun

http://www.w3.org/2001/XMLSchema#string

Mustafa Q Hameed

http://www.w3.org/2001/XMLSchema#string

P2860

Migration of human monocytes in response to vascular endothelial growth factor (VEGF) is mediated via the VEGF receptor flt-1

Appearance of heparin-binding EGF-like growth factor in wound fluid as a response to injury

EGF converts transit-amplifying neurogenic precursors in the adult brain into multipotent stem cells

VEGF is an essential mediator of the neurogenic and behavioral actions of antidepressants

Estrogen-induced activation of Erk-1 and Erk-2 requires the G protein-coupled receptor homolog, GPR30, and occurs via trans-activation of the epidermal growth factor receptor through release of HB-EGF

EGF receptor transactivation by G-protein-coupled receptors requires metalloproteinase cleavage of proHB-EGF

Hydrocephalus in children

Network of tangential pathways for neuronal migration in adult mammalian brain

Lysophosphatidic acid signaling may initiate fetal hydrocephalus.

High CSF transforming growth factor beta levels after subarachnoid haemorrhage: association with chronic communicating hydrocephalus.

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srep26794

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26794

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/SREP26794

http://www.w3.org/2001/XMLSchema#string

P407

English

P478

http://www.w3.org/2001/XMLSchema#string

P50

Bonnie Blazer-Yost

P577

2016-05-31T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27243144

http://www.w3.org/2001/XMLSchema#string

P921

hydrocephalus

P932

4886677

http://www.w3.org/2001/XMLSchema#string

Excess HB-EGF, which promotes VEGF signaling, leads to hydrocephalus

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P2860

P2860

Excess HB-EGF, which promotes VEGF signaling, leads to hydrocephalus

description

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type

label

prefLabel

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P478

P50

P577

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P407

P478

P50

P577

P698

P921

P932