Glutathione adducts induced by ischemia and deletion of glutaredoxin-1 stabilize HIF-1α and improve limb revascularization. - Wikidata - awgldk - TriplyDB

Glutathione adducts induced by ischemia and deletion of glutaredoxin-1 stabilize HIF-1α and improve limb revascularization.

Glutathione adducts induced by ischemia and deletion of glutaredoxin-1 stabilize HIF-1α and improve limb revascularization.

http://www.wikidata.org/entity/Q36957272

about

Peripheral artery disease, redox signaling, oxidative stress - Basic and clinical aspects Redox regulation of ischemic limb neovascularization - What we have learned from animal studies European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).Glutaredoxin-1 Deficiency Causes Fatty Liver and Dyslipidemia by Inhibiting Sirtuin-1.The Impact of Environmental Factors in Influencing Epigenetics Related to Oxidative States in the Cardiovascular System.Solution structure of the TLR adaptor MAL/TIRAP reveals an intact BB loop and supports MAL Cys91 glutathionylation for signaling.Vascular endothelial growth factor signaling requires glycine to promote angiogenesis.Distinct Shift in Beta-Cell Glutaredoxin 5 Expression Is Mediated by Hypoxia and Lipotoxicity Both In Vivo and In Vitro.Biochemistry of proinflammatory macrophage activation.Oxidative stress and antioxidant treatment in patients with peripheral artery disease.

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P2860

Glutathione adducts induced by ischemia and deletion of glutaredoxin-1 stabilize HIF-1α and improve limb revascularization.

http://www.wikidata.org/entity/Q36957272

description

2016 nî lūn-bûn

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Glutathione adducts induced by ...... mprove limb revascularization.

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Glutathione adducts induced by ...... mprove limb revascularization.

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Glutathione adducts induced by ...... mprove limb revascularization.

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PNAS.1524198113

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Proceedings of the National Academy of Sciences of the United States of America

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Glutathione adducts induced by ...... improve limb revascularization

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Reiko Matsui

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Richard A Cohen

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Soichi Sano

http://www.w3.org/2001/XMLSchema#string

Yasuo Ido

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Yosuke Watanabe

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P2860

Genome engineering using the CRISPR-Cas9 system

Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation

Regulation of PTP1B via glutathionylation of the active site cysteine 215

Dynamic redox control of NF-kappaB through glutaredoxin-regulated S-glutathionylation of inhibitory kappaB kinase beta

Ubiquitination of hypoxia-inducible factor requires direct binding to the beta-domain of the von Hippel-Lindau protein

Normoxic stabilization of hypoxia-inducible factor-1 expression and activity: redox-dependent effect of nitrogen oxides.

Role of gp91phox (Nox2)-containing NAD(P)H oxidase in angiogenesis in response to hindlimb ischemia.

Vascular responses to hypoxia and ischemia.

Reactive oxygen radicals in signaling and damage in the ischemic brain.

Regulation of protein function by S-glutathiolation in response to oxidative and nitrosative stress.

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6011-6016

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21

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113

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Markus Bachschmid

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2016-05-09T00:00:00Z

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27162359

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revascularization

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4889374

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