Identification of Aurora-A as a direct target of E2F3 during G2/M cell cycle progression
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Cross-Talk between AURKA and Plk1 in Mitotic Entry and Spindle AssemblyFunctional Significance of Aurora Kinases-p53 Protein Family Interactions in CancerRNAi mediated acute depletion of retinoblastoma protein (pRb) promotes aneuploidy in human primary cells via micronuclei formation.Arsenic treatment increase Aurora-A overexpression through E2F1 activation in bladder cells.Identification of a preneoplastic gene expression profile in tubal epithelium of BRCA1 mutation carriers.HDAC 1/4-mediated silencing of microRNA-200b promotes chemoresistance in human lung adenocarcinoma cells.Aurora kinase A promotes ovarian tumorigenesis through dysregulation of the cell cycle and suppression of BRCA2.Phosphorylation and activation of androgen receptor by Aurora-AE2F inhibition synergizes with paclitaxel in lung cancer cell linesMUC16 induced rapid G2/M transition via interactions with JAK2 for increased proliferation and anti-apoptosis in breast cancer cellsEstrogen-induced aurora kinase-A (AURKA) gene expression is activated by GATA-3 in estrogen receptor-positive breast cancer cells.Aurora A is differentially expressed in gliomas, is associated with patient survival in glioblastoma and is a potential chemotherapeutic target in gliomas.p53 negatively regulates Aurora A via both transcriptional and posttranslational regulation.Silencing of E2F3 suppresses tumor growth of Her2+ breast cancer cells by restricting mitosisTranslational up-regulation of Aurora-A in EGFR-overexpressed cancerMicroarray analysis revealed different gene expression patterns in HepG2 cells treated with low and high concentrations of the extracts of Anacardium occidentale shoots.miR-874 suppresses the proliferation and metastasis of osteosarcoma by targeting E2F3.Low concentration of arsenic-induced aberrant mitosis in keratinocytes through E2F1 transcriptionally regulated Aurora-A.MicroRNA-200b reverses chemoresistance of docetaxel-resistant human lung adenocarcinoma cells by targeting E2F3.The interleukin 6 receptor is a direct transcriptional target of E2F3 in prostate tumor derived cells.E2F3 upregulation promotes tumor malignancy through the transcriptional activation of HIF-2α in clear cell renal cell carcinoma.Human papillomavirus 16 E7 oncoprotein attenuates DNA damage checkpoint control by increasing the proteolytic turnover of claspin.The E2F activators control multiple mitotic regulators and maintain genomic integrity through Sgo1 and BubR1.Transcriptional repression of Aurora-A gene by wild-type p53 through directly binding to its promoter with histone deacetylase 1 and mSin3a.
P2860
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P2860
Identification of Aurora-A as a direct target of E2F3 during G2/M cell cycle progression
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Identification of Aurora-A as a direct target of E2F3 during G2/M cell cycle progression
@en
type
label
Identification of Aurora-A as a direct target of E2F3 during G2/M cell cycle progression
@en
prefLabel
Identification of Aurora-A as a direct target of E2F3 during G2/M cell cycle progression
@en
P2093
P2860
P356
P1476
Identification of Aurora-A as a direct target of E2F3 during G2/M cell cycle progression
@en
P2093
Jin Q Cheng
W Douglas Cress
W Jack Pledger
P2860
P304
31012-31020
P356
10.1074/JBC.M803547200
P407
P577
2008-09-07T00:00:00Z