Inclusion-body myositis, a multifactorial muscle disease associated with aging: current concepts of pathogenesis.
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How citation distortions create unfounded authority: analysis of a citation networkTargeting protein homeostasis in sporadic inclusion body myositis.Clinical approach to the diagnostic evaluation of hereditary and acquired neuromuscular diseasesIntracellular amyloid formation in muscle cells of Abeta-transgenic Caenorhabditis elegans: determinants and physiological role in copper detoxification.CNBP acts as a key transcriptional regulator of sustained expression of interleukin-6.In AbetaPP-overexpressing cultured human muscle fibers proteasome inhibition enhances phosphorylation of AbetaPP751 and GSK3beta activation: effects mitigated by lithium and apparently relevant to sporadic inclusion-body myositisAutoantibodies produced at the site of tissue damage provide evidence of humoral autoimmunity in inclusion body myositis.Ongoing developments in sporadic inclusion body myositisBeyond the binding site: in vivo identification of tbx2, smarca5 and wnt5b as molecular targets of CNBP during embryonic development.The protein oxidation repair enzyme methionine sulfoxide reductase a modulates Aβ aggregation and toxicity in vivo.In inclusion-body myositis muscle fibers Parkinson-associated DJ-1 is increased and oxidized.Inclusion-body myositis: muscle-fiber molecular pathology and possible pathogenic significance of its similarity to Alzheimer's and Parkinson's disease brains.Idiopathic inflammatory myopathies: current and future therapeutic options.Exercise in inflammatory myopathies, including inclusion body myositis.Secreted protein acidic and rich in cysteine (SPARC) in human skeletal muscle.Vacuolar myopathy in a dog resembling human sporadic inclusion body myositis.Graphical modelling of molecular networks underlying sporadic inclusion body myositis.Overexpression of autophagic proteins in the skeletal muscle of sporadic inclusion body myositis.Expression of DJ-1 in Neurodegenerative Disorders.Endoplasmic Reticulum Stress Induces Myostatin High Molecular Weight Aggregates and Impairs Mature Myostatin Secretion.Synaptic defects associated with s-inclusion body myositis are prevented by copper.Phosphorylated TDP-43 (pTDP-43) aggregates in the axial skeletal muscle of patients with sporadic and familial amyotrophic lateral sclerosis.High Fat With High Sucrose Diet Leads to Obesity and Induces Myodegeneration
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P2860
Inclusion-body myositis, a multifactorial muscle disease associated with aging: current concepts of pathogenesis.
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2007 nî lūn-bûn
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2007年の論文
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2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
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2007年论文
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name
Inclusion-body myositis, a mul ...... rent concepts of pathogenesis.
@en
type
label
Inclusion-body myositis, a mul ...... rent concepts of pathogenesis.
@en
prefLabel
Inclusion-body myositis, a mul ...... rent concepts of pathogenesis.
@en
P1476
Inclusion-body myositis, a mul ...... rent concepts of pathogenesis.
@en
P2093
Valerie Askanas
W King Engel
P304
P356
10.1097/BOR.0B013E3282EFDC7C
P577
2007-11-01T00:00:00Z