A novel mouse model to study the virulence of and host response to Porphyromonas (Bacteroides) gingivalis.
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Porphyromonas gingivalis facilitates the development and progression of destructive arthritis through its unique bacterial peptidylarginine deiminase (PAD)Porphyromonas gingivalis-host interactions: open war or intelligent guerilla tactics?The enduring importance of animal models in understanding periodontal diseasePorphyromonas gingivalis mediates inflammasome repression in polymicrobial cultures through a novel mechanism involving reduced endocytosisMice lacking inducible nitric oxide synthase demonstrate impaired killing of Porphyromonas gingivalisDisruption of heterotypic community development by Porphyromonas gingivalis with small molecule inhibitors.Prevention of Porphyromonas gingivalis-induced oral bone loss following immunization with gingipain R1.A peptide domain on gingipain R which confers immunity against Porphyromonas gingivalis infection in mice.Identification of a gingipain-sensitive surface ligand of Porphyromonas gingivalis that induces Toll-like receptor 2- and 4-independent NF-kappaB activation in CHO cells.Strain-dependent activation of monocytes and inflammatory macrophages by lipopolysaccharide of Porphyromonas gingivalisTetratricopeptide repeat protein-associated proteins contribute to the virulence of Porphyromonas gingivalis.Distribution of Porphyromonas gingivalis strains with fimA genotypes in periodontitis patients.Sequencing of the ribosomal intergenic spacer region for strain identification of Porphyromonas gingivalisPhylogeny of Porphyromonas gingivalis by ribosomal intergenic spacer region analysis.Mechanism and implications of CXCR4-mediated integrin activation by Porphyromonas gingivalis.Review: Pathogen-induced inflammation at sites distant from oral infection: bacterial persistence and induction of cell-specific innate immune inflammatory pathways.The rag locus of Porphyromonas gingivalis might arise from Bacteroides via horizontal gene transfer.Tissue destruction induced by Porphyromonas gingivalis infection in a mouse chamber model is associated with host tumor necrosis factor generationPathogen-mediated proteolysis of the cell death regulator RIPK1 and the host defense modulator RIPK2 in human aortic endothelial cells.Strain-specific colonization patterns and serum modulation of multi-species oral biofilm development.Porphyromonas gingivalis-host interactions in a Drosophila melanogaster model.Porphyromonas gingivalis virulence in a Drosophila melanogaster modelBinding and accumulation of hemin in Porphyromonas gingivalis are induced by hemin.Effects of a Porphyromonas gingivalis infection on inflammatory mediator response and pregnancy outcome in hamsters.Roles of the host oxidative immune response and bacterial antioxidant rubrerythrin during Porphyromonas gingivalis infection.Genotype is an important determinant factor of host susceptibility to periodontitis in the Collaborative Cross and inbred mouse populationsImpact of resolvin E1 on murine neutrophil phagocytosis in type 2 diabetesResistance of a Tn4351-generated polysaccharide mutant of Porphyromonas gingivalis to polymorphonuclear leukocyte killingCharacterization of a Tn4351-generated hemin uptake mutant of Porphyromonas gingivalis: evidence for the coordinate regulation of virulence factors by hemin.Identification of murine protective epitopes on the Porphyromonas gingivalis fimbrillin moleculeThe hemagglutinin genes hagB and hagC of Porphyromonas gingivalis are transcribed in vivo as shown by use of a new expression vectorVirulence characteristics of oral treponemes in a murine model.Porphyromonas gingivalis infection during pregnancy increases maternal tumor necrosis factor alpha, suppresses maternal interleukin-10, and enhances fetal growth restriction and resorption in mice.Genome Sequence of Porphyromonas gingivalis Strain A7436.Use of insertion sequence element IS1126 in a genotyping and transmission study of Porphyromonas gingivalis.Potent In Vitro and In Vivo Activity of Plantibody Specific for Porphyromonas gingivalis FimA.Influence of immunization on Porphyromonas gingivalis colonization and invasion in the mouse chamber model.Phagocytosis of virulent Porphyromonas gingivalis by human polymorphonuclear leukocytes requires specific immunoglobulin G.Antibody-dependent alternate pathway of complement activation in opsonophagocytosis of Porphyromonas gingivalis.The use of rodent models to investigate host-bacteria interactions related to periodontal diseases.
P2860
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P2860
A novel mouse model to study the virulence of and host response to Porphyromonas (Bacteroides) gingivalis.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
A novel mouse model to study t ...... onas (Bacteroides) gingivalis.
@en
type
label
A novel mouse model to study t ...... onas (Bacteroides) gingivalis.
@en
prefLabel
A novel mouse model to study t ...... onas (Bacteroides) gingivalis.
@en
P2093
P2860
P1476
A novel mouse model to study t ...... onas (Bacteroides) gingivalis.
@en
P2093
C W Cutler
D Kapczynski
R R Arnold
P2860
P304
P407
P577
1991-04-01T00:00:00Z