Stress fiber-based movement of Shigella flexneri within cells.
about
Common themes in microbial pathogenicity revisitedShigella flexneri enters human colonic Caco-2 epithelial cells through the basolateral poleIdentification and characterization of B-cell epitopes of IpaC, an invasion-associated protein of Shigella flexneri.Directional actin polymerization associated with spotted fever group Rickettsia infection of Vero cells.Shigella flexneri Interactions with the Basolateral Membrane Domain of Polarized Model Intestinal Epithelium: Role of Lipopolysaccharide in Cell Invasion and in Activation of the Mitogen-Activated Protein Kinase ERKPolymorphonuclear leukocyte transmigration promotes invasion of colonic epithelial monolayer by Shigella flexneriNucleotide sequence of the rhamnose biosynthetic operon of Shigella flexneri 2a and role of lipopolysaccharide in virulence.Acute inflammation causes epithelial invasion and mucosal destruction in experimental shigellosisCytoskeletal rearrangements and the functional role of T-plastin during entry of Shigella flexneri into HeLa cells.Bacterial conjugation in the cytoplasm of mouse cells.Movement along actin filaments of the perijunctional area and de novo polymerization of cellular actin are required for Shigella flexneri colonization of epithelial Caco-2 cell monolayersMyosin-cross-reactive epitope of Shigella flexneri invasion plasmid antigen B.Mobilization of F-actin and clathrin during redistribution of Chlamydia trachomatis to an intracellular site in eucaryotic cells.The shigella paradigm and colitis due to enterohaemorrhagic Escherichia coli.Role of lipopolysaccharide in signaling to subepithelial polymorphonuclear leukocytesUnipolar localization and ATPase activity of IcsA, a Shigella flexneri protein involved in intracellular movement.Unipolar reorganization of F-actin layer at bacterial division and bundling of actin filaments by plastin correlate with movement of Shigella flexneri within HeLa cells.Shigella subversion of the cellular cytoskeleton: a strategy for epithelial colonization.Regulation of surface presentation of IcsA, a Shigella protein essential to intracellular movement and spread, is growth phase dependent.Interaction of Ipa proteins of Shigella flexneri with alpha5beta1 integrin promotes entry of the bacteria into mammalian cells.IpaB of Shigella flexneri causes entry into epithelial cells and escape from the phagocytic vacuoleTransfer of zymosan (yeast cell walls) to the parasitophorous vacuoles of macrophages infected with Leishmania amazonensis.rho, a small GTP-binding protein, is essential for Shigella invasion of epithelial cells.
P2860
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P2860
Stress fiber-based movement of Shigella flexneri within cells.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Stress fiber-based movement of Shigella flexneri within cells.
@en
type
label
Stress fiber-based movement of Shigella flexneri within cells.
@en
prefLabel
Stress fiber-based movement of Shigella flexneri within cells.
@en
P2093
P2860
P1476
Stress fiber-based movement of Shigella flexneri within cells.
@en
P2093
Prevost MC
Sansonetti PJ
Vasselon T
P2860
P304
P407
P577
1991-05-01T00:00:00Z