C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis - Wikidata - awgldk - TriplyDB

C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis

C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis

http://www.wikidata.org/entity/Q36967242

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Role of endoplasmic reticulum stress in the pathogenesis of nonalcoholic fatty liver disease The stress-regulated transcription factor CHOP promotes hepatic inflammatory gene expression, fibrosis, and oncogenesis Hepatocytes release ceramide-enriched pro-inflammatory extracellular vesicles in an IRE1α-dependent manner.Inactivation of C/ebp homologous protein-driven immune-metabolic interactions exacerbate obesity and adipose tissue leukocytosis.The stress-response sensor chop regulates the function and accumulation of myeloid-derived suppressor cells in tumors CCAAT/enhancer-binding protein homologous (CHOP) protein promotes carcinogenesis in the DEN-induced hepatocellular carcinoma model Mmu-miR-615-3p regulates lipoapoptosis by inhibiting C/EBP homologous protein Ischemia/reperfusion-induced CHOP expression promotes apoptosis and impairs renal function recovery: the role of acidosis and GPR4 Cell death and cell death responses in liver disease: mechanisms and clinical relevance ER stress induces NLRP3 inflammasome activation and hepatocyte death.Regulation of the transcriptome by ER stress: non-canonical mechanisms and physiological consequences.Oxidative stress and calcium dysregulation by palmitate in type 2 diabetes.Pancreatic adaptive responses in alcohol abuse: Role of the unfolded protein response.Pathophysiological mechanisms and therapeutic potentials of macrophages in non-alcoholic steatohepatitis.Lipotoxicity pathways intersect in hepatocytes: Endoplasmic reticulum stress, c-Jun N-terminal kinase-1, and death receptors.Lipotoxic lethal and sublethal stress signaling in hepatocytes: relevance to NASH pathogenesis.Inhibition of sphingosine 1-phosphate signaling ameliorates murine nonalcoholic steatohepatitis.C/EBP homologous protein modulates liraglutide-mediated attenuation of non-alcoholic steatohepatitis.GRP78 and CHOP modulate macrophage apoptosis and the development of bleomycin-induced pulmonary fibrosis.TRAIL receptor deletion in mice suppresses the inflammation of nutrient excess.Multiple programmed cell death pathways are involved in N-methyl-N-nitrosourea-induced photoreceptor degeneration.Mixed-lineage kinase 3 pharmacological inhibition attenuates murine nonalcoholic steatohepatitis.Physiological/pathological ramifications of transcription factors in the unfolded protein response.Unfolded protein response signaling impacts macrophage polarity to modulate breast cancer cell clearance and melanoma immune checkpoint therapy responsiveness.FXR controls CHOP expression in steatohepatitis.Transcription Factor C/EBP Homologous Protein in Health and Diseases.Deletion of endoplasmic reticulum stress-responsive co-chaperone p58IPK protects mice from diet-induced steatohepatitis.Liver function and dysfunction - a unique window into the physiological reach of ER stress and the unfolded protein response.Reduction of Endoplasmic Reticulum Stress Improves Angiogenic Progenitor Cell function in a Mouse Model of Type 1 Diabetes.IL-7 suppresses macrophage autophagy and promotes liver pathology in Schistosoma japonicum-infected mice.

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C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis

http://www.wikidata.org/entity/Q36967242

description

2013 nî lūn-bûn

@nan

2013年の論文

@ja

2013年論文

@yue

2013年論文

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2013年論文

@zh-hk

2013年論文

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2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis

@en

type

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C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis

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C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis

@en

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JBC.M112.442954

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Journal of Biological Chemistry

P1476

C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis

@en

P2093

Amy S Mauer

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Christina R Kobrossi

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Erin M Kropp

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Harmeet Malhi

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JaeSeok Han

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Randal J Kaufman

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Vinna F Clavo

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P2860

Resolution of the two components of macrophage inflammatory protein 1, and cloning and characterization of one of those components, macrophage inflammatory protein 1 beta

Nitric oxide-induced apoptosis in RAW 264.7 macrophages is mediated by endoplasmic reticulum stress pathway involving ATF6 and CHOP

CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription

Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes

CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum

CCL2 recruits inflammatory monocytes to facilitate breast-tumour metastasis

Systematic review: the epidemiology and natural history of non-alcoholic fatty liver disease and non-alcoholic steatohepatitis in adults

Design and validation of a histological scoring system for nonalcoholic fatty liver disease

Systematic review of risk factors for fibrosis progression in non-alcoholic steatohepatitis.

Reduced apoptosis and plaque necrosis in advanced atherosclerotic lesions of Apoe-/- and Ldlr-/- mice lacking CHOP.

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18624-18642

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scholarly article

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10.1074/JBC.M112.442954

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26

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288

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2013-05-17T00:00:00Z

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23720735

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P921

Steatohepatitis

P932

3696637

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