Endothelium-specific interference with peroxisome proliferator activated receptor gamma causes cerebral vascular dysfunction in response to a high-fat diet. - Wikidata - awgldk - TriplyDB

Endothelium-specific interference with peroxisome proliferator activated receptor gamma causes cerebral vascular dysfunction in response to a high-fat diet.

Endothelium-specific interference with peroxisome proliferator activated receptor gamma causes cerebral vascular dysfunction in response to a high-fat diet.

http://www.wikidata.org/entity/Q36972995

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Endothelial PPAR-γ Protects Against Vascular Thrombosis by Downregulating P-Selectin ExpressionSignificance Genetic Regulation of Endothelial Vasomotor Function Contrasting vascular effects caused by loss of Bardet-Biedl syndrome genes.Smooth Muscle Peroxisome Proliferator-Activated Receptor γ Plays a Critical Role in Formation and Rupture of Cerebral Aneurysms in Mice In Vivo Endothelial and vascular muscle PPARgamma in arterial pressure regulation: lessons from genetic interference and deficiency.Vascular smooth muscle cell-selective peroxisome proliferator-activated receptor-gamma deletion leads to hypotension Bioinformatic analysis of gene sets regulated by ligand-activated and dominant-negative peroxisome proliferator-activated receptor gamma in mouse aorta.Role of vascular smooth muscle PPARγ in regulating AT1 receptor signaling and angiotensin II-dependent hypertension.Interference with peroxisome proliferator-activated receptor-γ in vascular smooth muscle causes baroreflex impairment and autonomic dysfunction.Genetic interference with peroxisome proliferator-activated receptor γ in smooth muscle enhances myogenic tone in the cerebrovasculature via A Rho kinase-dependent mechanism.Protecting against vascular disease in brain Peroxisome proliferator-activated receptor-γ regulates inflammation and renin-angiotensin system activity in the hypothalamic paraventricular nucleus and ameliorates peripheral manifestations of heart failure.Uncoupling of eNOS causes superoxide anion production and impairs NO signaling in the cerebral microvessels of hph-1 mice.Cullin-3 regulates vascular smooth muscle function and arterial blood pressure via PPARγ and RhoA/Rho-kinase.Interference with PPARγ in endothelium accelerates angiotensin II-induced endothelial dysfunction.Small-molecule inhibitors of signal transducer and activator of transcription 3 protect against angiotensin II-induced vascular dysfunction and hypertension Endothelial PPAR-γ provides vascular protection from IL-1β-induced oxidative stress The adaptation of the cerebral circulation to pregnancy: mechanisms and consequences Dominant negative PPARγ promotes atherosclerosis, vascular dysfunction, and hypertension through distinct effects in endothelium and vascular muscle Microvascular Dysfunction and Cognitive Impairment.Early effects of high-fat diet on neurovascular function and focal ischemic brain injury.PPARgamma in the endothelium regulates metabolic responses to high-fat diet in mice Effect of selective expression of dominant-negative PPARγ in pro-opiomelanocortin neurons on the control of energy balance.Significant synergistic effect of peroxisome proliferator-activated receptor gamma C-2821T and diabetes on the risk of ischemic stroke.Disruption of endothelial peroxisome proliferator-activated receptor-gamma reduces vascular nitric oxide production.Does peroxisome proliferator-activated receptor-gamma (PPAR gamma) protect from hypertension directly through effects in the vasculature?Retinol-binding protein 7 is an endothelium-specific PPARγ cofactor mediating an antioxidant response through adiponectin.Vascular dysfunction in cerebrovascular disease: mechanisms and therapeutic intervention.PPARγ agonist beyond glucose lowering effect.Molecular mechanisms regulating vascular tone by peroxisome proliferator activated receptor gamma.PPARγ Regulation in Hypertension and Metabolic Syndrome.Friend or foe? Telomerase as a pharmacological target in cancer and cardiovascular disease.Cerebral Vascular Disease and Neurovascular Injury in Ischemic Stroke.AT1 receptor signaling pathways in the cardiovascular system.The probiotic Lactobacillus coryniformis CECT5711 reduces the vascular pro-oxidant and pro-inflammatory status in obese mice.A clinical link between peroxisome proliferator-activated receptor γ and the renin-angiotensin system.Restoration of Endothelial Function in Pparα (-/-) Mice by Tempol.Surviving the remodel: the impact of hypertension during pregnancy.A second chance for a PPARγ targeted therapy?PPARγ: no SirT, no service.

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P2860

Endothelium-specific interference with peroxisome proliferator activated receptor gamma causes cerebral vascular dysfunction in response to a high-fat diet.

http://www.wikidata.org/entity/Q36972995

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2008 nî lūn-bûn

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Endothelium-specific interfere ...... n response to a high-fat diet.

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Endothelium-specific interfere ...... n response to a high-fat diet.

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Endothelium-specific interfere ...... n response to a high-fat diet.

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Circulation Research

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Endothelium-specific interfere ...... in response to a high-fat diet

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Andreas M Beyer

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Frank M Faraci

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Henry L Keen

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Mary L Modrick

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Willem J de Lange

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P2860

Dominant negative mutations in human PPARgamma associated with severe insulin resistance, diabetes mellitus and hypertension

Effect of Rosiglitazone on the Risk of Myocardial Infarction and Death from Cardiovascular Causes

Oxidative stress and dysregulation of NAD(P)H oxidase and antioxidant enzymes in diet-induced metabolic syndrome

Increased superoxide and vascular dysfunction in CuZnSOD-deficient mice

PPAR gamma is required for placental, cardiac, and adipose tissue development

The ligands/activators for peroxisome proliferator-activated receptor alpha (PPARalpha) and PPARgamma increase Cu2+,Zn2+-superoxide dismutase and decrease p22phox message expressions in primary endothelial cells.

Conditional disruption of the peroxisome proliferator-activated receptor gamma gene in mice results in lowered expression of ABCA1, ABCG1, and apoE in macrophages and reduced cholesterol efflux.

PPAR-gamma regulates osteoclastogenesis in mice.

Non-DNA binding, dominant-negative, human PPARgamma mutations cause lipodystrophic insulin resistance.

Hypertension and abnormal fat distribution but not insulin resistance in mice with P465L PPARgamma.

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6

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