Down-regulation of dendritic cell signaling pathways by Leishmania amazonensis amastigotes. - Wikidata - awgldk - TriplyDB

Down-regulation of dendritic cell signaling pathways by Leishmania amazonensis amastigotes.

Down-regulation of dendritic cell signaling pathways by Leishmania amazonensis amastigotes.

http://www.wikidata.org/entity/Q36973139

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Modulation of Innate Immune Mechanisms to Enhance Leishmania Vaccine-Induced Immunity: Role of Coinhibitory Molecules The Mitogen-Activated Protein Kinase (MAPK) Pathway: Role in Immune Evasion by Trypanosomatids Mechanisms underlying the induction of regulatory T cells and its relevance in the adaptive immune response in parasitic infections Innate immunity against Leishmania infections Purinergic signaling and infection by Leishmania: A new approach to evasion of the immune response Ultradeformable Archaeosomes for Needle Free Nanovaccination with Leishmania braziliensis Antigens Interferon regulatory factor 7 contributes to the control of Leishmania donovani in the mouse liver.Degradation of host sphingomyelin is essential for Leishmania virulence.CXCL10 production by human monocytes in response to Leishmania braziliensis infection.Leishmania interferes with host cell signaling to devise a survival strategy.Leishmania donovani amastigotes impair gamma interferon-induced STAT1alpha nuclear translocation by blocking the interaction between STAT1alpha and importin-alpha5 Impact of Leishmania mexicana infection on dendritic cell signaling and functions Exposure of phosphatidylserine on Leishmania amazonensis isolates is associated with diffuse cutaneous leishmaniasis and parasite infectivity Activation of phosphatidylinositol 3-kinase/Akt and impairment of nuclear factor-kappaB: molecular mechanisms behind the arrested maturation/activation state of Leishmania infantum-infected dendritic cells.Deception and manipulation: the arms of leishmania, a successful parasite Leishmania promotes its own virulence by inducing expression of the host immune inhibitory ligand CD200 Interactions between Neutrophils and Leishmania braziliensis Amastigotes Facilitate Cell Activation and Parasite Clearance The early interaction of Leishmania with macrophages and dendritic cells and its influence on the host immune response.Encephalitozoon intestinalis Inhibits Dendritic Cell Differentiation through an IL-6-Dependent Mechanism Phosphatidylserine exposure on the surface of Leishmania amazonensis amastigotes modulates in vivo infection and dendritic cell function.Role of Toll-like receptor 9 signaling in experimental Leishmania braziliensis infection.Role of natural killer cells in modulating dendritic cell responses to Leishmania amazonensis infection.Altered dendritic cell phenotype in response to Leishmania amazonensis amastigote infection is mediated by MAP kinase, ERK Distinct roles for MyD88 and Toll-like receptor 2 during Leishmania braziliensis infection in mice.Leishmania amazonensis amastigotes trigger neutrophil activation but resist neutrophil microbicidal mechanisms.Tracing immunity to human leishmaniasis.Dysregulation of macrophage signal transduction by Toxoplasma gondii: past progress and recent advances.Cytokines and their STATs in cutaneous and visceral leishmaniasis.Leishmania mexicana promastigotes inhibit macrophage IL-12 production via TLR-4 dependent COX-2, iNOS and arginase-1 expression.Retinoic Acid Regulates Immune Responses by Promoting IL-22 and Modulating S100 Proteins in Viral Hepatitis.Dendritic cells and parasites: from recognition and activation to immune response instruction.Leishmania infantum and Leishmania braziliensis: Differences and Similarities to Evade the Innate Immune System.Leishmania donovani infection drives the priming of human monocyte-derived dendritic cells during Plasmodium falciparum co-infections.Leishmania amazonensis-Induced cAMP Triggered by Adenosine A2B Receptor Is Important to Inhibit Dendritic Cell Activation and Evade Immune Response in Infected Mice.Subversion of Immunity by Leishmania amazonensis Parasites: Possible Role of Phosphatidylserine as a Main Regulator.Leishmania Hijacks Myeloid Cells for Immune Escape.Possible Interrelationship of Inflammatory Cells in Dry Type Cutaneous Leishmaniasis.Leishmania amazonensisimpairs DC function by inhibiting CD40 expression via A2Badenosine receptor activation

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P2860

Down-regulation of dendritic cell signaling pathways by Leishmania amazonensis amastigotes.

http://www.wikidata.org/entity/Q36973139

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Down-regulation of dendritic c ...... mania amazonensis amastigotes.

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Down-regulation of dendritic c ...... mania amazonensis amastigotes.

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Down-regulation of dendritic c ...... mania amazonensis amastigotes.

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J.MOLIMM.2008.04.018

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Molecular Immunology

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Down-regulation of dendritic c ...... mania amazonensis amastigotes.

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Kui Li

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Lijun Xin

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Lynn Soong

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P2860

Interleukin-10 and the interleukin-10 receptor

Evidence for licensing of IFN-gamma-induced IFN regulatory factor 1 transcription factor by MyD88 in Toll-like receptor-dependent gene induction program

Interleukin-10 determines viral clearance or persistence in vivo

IRF-8/interferon (IFN) consensus sequence-binding protein is involved in Toll-like receptor (TLR) signaling and contributes to the cross-talk between TLR and IFN-gamma signaling pathways

IRF family of transcription factors as regulators of host defense

Differential regulation of interleukin (IL)-12 p35 and p40 gene expression and interferon (IFN)-gamma-primed IL-12 production by IFN regulatory factor 1

Subversion mechanisms by which Leishmania parasites can escape the host immune response: a signaling point of view.

Antigen requirements for efficient priming of CD8+ T cells by Leishmania major-infected dendritic cells.

Early enhanced Th1 response after Leishmania amazonensis infection of C57BL/6 interleukin-10-deficient mice does not lead to resolution of infection.

ERK activation following macrophage FcgammaR ligation leads to chromatin modifications at the IL-10 locus.

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3371-3382

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scholarly article

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10.1016/J.MOLIMM.2008.04.018

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12

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45

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2008-06-05T00:00:00Z

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18538399

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2583126

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