Compatibility among polymerase subunit proteins is a restricting factor in reassortment between equine H7N7 and human H3N2 influenza viruses.
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Intrasubtype reassortments cause adaptive amino acid replacements in H3N2 influenza genesExperimental Approaches to Study Genome Packaging of Influenza A VirusesReassortment between two serologically unrelated bluetongue virus strains is flexible and can involve any genome segmentPossibility of cross-species/subtype reassortments in influenza A viruses: an analysis of nonstructural protein variations.Avian influenza virus h3 hemagglutinin may enable high fitness of novel human virus reassortantsNaturally occurring mutations in the PA gene are key contributors to increased virulence of pandemic H1N1/09 influenza virus in mice.The contribution of PA-X to the virulence of pandemic 2009 H1N1 and highly pathogenic H5N1 avian influenza viruses.The infection of turkeys and chickens by reassortants derived from pandemic H1N1 2009 and avian H9N2 influenza viruses.Full factorial analysis of mammalian and avian influenza polymerase subunits suggests a role of an efficient polymerase for virus adaptationNuclear import and assembly of influenza A virus RNA polymerase studied in live cells by fluorescence cross-correlation spectroscopy.Postreassortment amino acid substitutions in influenza A viruses.Influenza NA and PB1 Gene Segments Interact during the Formation of Viral Progeny: Localization of the Binding Region within the PB1 Gene.High level of genetic compatibility between swine-origin H1N1 and highly pathogenic avian H5N1 influenza viruses.Atypical characteristics of nucleoprotein of pandemic influenza virus H1N1 and their roles in reassortment restriction.Heterologous Packaging Signals on Segment 4, but Not Segment 6 or Segment 8, Limit Influenza A Virus Reassortment.High genetic compatibility and increased pathogenicity of reassortants derived from avian H9N2 and pandemic H1N1/2009 influenza virusesVirulence and genetic compatibility of polymerase reassortant viruses derived from the pandemic (H1N1) 2009 influenza virus and circulating influenza A viruses.Reassortment between seasonal H1N1 and pandemic (H1N1) 2009 influenza viruses is restricted by limited compatibility among polymerase subunits.Reassortment and mutation of the avian influenza virus polymerase PA subunit overcome species barriersViral reassortment as an information exchange between viral segmentsThe RNA polymerase PB2 subunit of influenza A/HongKong/156/1997 (H5N1) restricts the replication of reassortant ribonucleoprotein complexes [corrected]Mutations in NA that induced low pH-stability and enhanced the replication of pandemic (H1N1) 2009 influenza A virus at an early stage of the pandemic.Molecular determinants of adaptation of highly pathogenic avian influenza H7N7 viruses to efficient replication in the human hostGenetic content of Influenza H3N2 vaccine seedsReassortment between avian H5N1 and human H3N2 influenza viruses creates hybrid viruses with substantial virulence.Genomic and protein structural maps of adaptive evolution of human influenza A virus to increased virulence in the mouse.PB2 and hemagglutinin mutations are major determinants of host range and virulence in mouse-adapted influenza A virusInfluenza virus reassortment occurs with high frequency in the absence of segment mismatch.Positive Selection Drives Preferred Segment Combinations during Influenza Virus Reassortment.Frequency of influenza H3N2 intra-subtype reassortment: attributes and implications of reassortant spread.Amaryllidaceae alkaloids inhibit nuclear-to-cytoplasmic export of ribonucleoprotein (RNP) complex of highly pathogenic avian influenza virus H5N1.Molecular Determinants of Virulence and Stability of a Reporter-Expressing H5N1 Influenza A VirusPA from an H5N1 highly pathogenic avian influenza virus activates viral transcription and replication and induces apoptosis and interferon expression at an early stage of infectionGenetics, Receptor Binding, Replication, and Mammalian Transmission of H4 Avian Influenza Viruses Isolated from Live Poultry Markets in China.The source of the PB1 gene in influenza vaccine reassortants selectively alters the hemagglutinin content of the resulting seed virus.Critical role of segment-specific packaging signals in genetic reassortment of influenza A viruses3D Molecular Modelling Study of the H7N9 RNA-Dependent RNA Polymerase as an Emerging Pharmacological TargetReassortment in segmented RNA viruses: mechanisms and outcomes.Recognition of cap structure by influenza B virus RNA polymerase is less dependent on the methyl residue than recognition by influenza A virus polymerase.Novel insights into bat influenza A viruses.
P2860
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P2860
Compatibility among polymerase subunit proteins is a restricting factor in reassortment between equine H7N7 and human H3N2 influenza viruses.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Compatibility among polymerase ...... human H3N2 influenza viruses.
@en
type
label
Compatibility among polymerase ...... human H3N2 influenza viruses.
@en
prefLabel
Compatibility among polymerase ...... human H3N2 influenza viruses.
@en
P2093
P2860
P356
P1433
P1476
Compatibility among polymerase ...... human H3N2 influenza viruses.
@en
P2093
Chengjun Li
Gabriele Neumann
Masato Hatta
Shinji Watanabe
P2860
P304
11880-11888
P356
10.1128/JVI.01445-08
P407
P577
2008-09-24T00:00:00Z