Mice with different susceptibility to tick-borne encephalitis virus infection show selective neutralizing antibody response and inflammatory reaction in the central nervous system.
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The role of viral persistence in flavivirus biologyGene-specific sex effects on eosinophil infiltration in leishmaniasisPolymorphisms in chemokine receptor 5 and Toll-like receptor 3 genes are risk factors for clinical tick-borne encephalitis in the Lithuanian populationIncreased levels of sphingosine-1-phosphate in cerebrospinal fluid of patients diagnosed with tick-borne encephalitisThe expression of the chemokine receptor CCR5 in tick-borne encephalitis.Encephalitic Arboviruses: Emergence, Clinical Presentation, and Neuropathogenesis.Lethal Experimental Tick-Borne Encephalitis Infection: Influence of Two Strains with Similar Virulence on the Immune Response.Chemokine receptors as important regulators of pathogenesis during arboviral encephalitisAnalysis of tick-borne encephalitis virus-induced host responses in human cells of neuronal origin and interferon-mediated protection.The increased concentration of macrophage migration inhibitory factor in serum and cerebrospinal fluid of patients with tick-borne encephalitisEscape of tick-borne flavivirus from 2' -C-methylated nucleoside antivirals is mediated by a single conservative mutation in NS5 that has a dramatic effect on viral fitness.Type I interferon protects mice from fatal neurotropic infection with Langat virus by systemic and local antiviral responses.Interferon gamma modulation of disease manifestation and the local antibody response to alphavirus encephalomyelitis.Analysis of serum levels of cytokines, chemokines, growth factors, and monoamine neurotransmitters in patients with tick-borne encephalitis: identification of novel inflammatory markers with implications for pathogenesis.A tick-borne encephalitis model in infant rats infected with langat virus.A database of human genes and a gene network involved in response to tick-borne encephalitis virus infection.The intrathecal expression and pathogenetic role of Th17 cytokines and CXCR2-binding chemokines in tick-borne encephalitis.A novel locus on mouse chromosome 7 that influences survival after infection with tick-borne encephalitis virus.Virus RNA Load in Patients with Tick-Borne Encephalitis, Slovenia.
P2860
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P2860
Mice with different susceptibility to tick-borne encephalitis virus infection show selective neutralizing antibody response and inflammatory reaction in the central nervous system.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Mice with different susceptibi ...... in the central nervous system.
@en
type
label
Mice with different susceptibi ...... in the central nervous system.
@en
prefLabel
Mice with different susceptibi ...... in the central nervous system.
@en
P2093
P2860
P356
P1476
Mice with different susceptibi ...... in the central nervous system.
@en
P2093
Daniel Růžek
Jan Kopecký
Jarmila Vojtíšková
Jiří Salát
Libor Grubhoffer
Marie Lipoldová
Peter Demant
P2860
P2888
P356
10.1186/1742-2094-10-77
P50
P577
2013-06-27T00:00:00Z
P5875
P6179
1034307715