Systemic inhibition of canonical Notch signaling results in sustained callus inflammation and alters multiple phases of fracture healing.
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Overview of biological mechanisms and applications of three murine models of bone repair: closed fracture with intramedullary fixation, distraction osteogenesis, and marrow ablation by reamingThe wound healing, chronic fibrosis, and cancer progression triadFractures in geriatric mice show decreased callus expansion and bone volumeJagged1 is essential for osteoblast development during maxillary ossification.Transient gamma-secretase inhibition accelerates and enhances fracture repair likely via Notch signaling modulationInactivation of the Progesterone Receptor in Mx1+ Cells Potentiates Osteogenesis in Calvaria but Not in Long Bone.Effects of panax notoginseng saponins on the osteogenic differentiation of rabbit bone mesenchymal stem cells through TGF-β1 signaling pathway.NOTCH signaling in skeletal progenitors is critical for fracture repair.Notch Signaling and the Skeleton.Biological perspectives of delayed fracture healing.Langerhans cell histiocytosis is a neoplasm and consequently its recurrence is a relapse: In memory of Bob Arceci.Analysis of αSMA-labeled progenitor cell commitment identifies notch signaling as an important pathway in fracture healingInhibition of GDF8 (Myostatin) accelerates bone regeneration in diabetes mellitus type 2Jagged1 expression by osteoblast-lineage cells regulates trabecular bone mass and periosteal expansion in mice.Effects of Aging on Fracture Healing.Absence of Notch1 in murine myeloid cells attenuates the development of experimental autoimmune encephalomyelitis by affecting Th1 and Th17 priming.The Tetraspanin TSPAN33 Controls TLR-Triggered Macrophage Activation through Modulation of NOTCH Signaling.Notch pathway is active during osteogenic differentiation of human bone marrow mesenchymal stem cells induced by pulsed electromagnetic fields.Intraoperative delivery of the Notch ligand Jagged-1 regenerates appendicular and craniofacial bone defects.Autocrine Action of Thrombospondin-2 Determines the Chondrogenic Differentiation Potential and Suppresses Hypertrophic Maturation of Human Umbilical Cord Blood-Derived Mesenchymal Stem Cells.Identification of key gene networks associated with fracture healing using αSMA‑labeled progenitor cells.Microenvironmental Regulation of Chondrocyte Plasticity in Endochondral Repair-A New Frontier for Developmental Engineering.
P2860
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P2860
Systemic inhibition of canonical Notch signaling results in sustained callus inflammation and alters multiple phases of fracture healing.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
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2013年论文
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name
Systemic inhibition of canonic ...... le phases of fracture healing.
@en
type
label
Systemic inhibition of canonic ...... le phases of fracture healing.
@en
prefLabel
Systemic inhibition of canonic ...... le phases of fracture healing.
@en
P2093
P2860
P1433
P1476
Systemic inhibition of canonic ...... le phases of fracture healing.
@en
P2093
Andrew M Barr
Joel D Takacs
Julie B Engiles
Michael I Dishowitz
Patricia L Mutyaba
P2860
P304
P356
10.1371/JOURNAL.PONE.0068726
P407
P577
2013-07-03T00:00:00Z