Demonstration of an early and a late phase of ischemic preconditioning in mice.
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Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondriaDiscovery of a new function of cyclooxygenase (COX)-2: COX-2 is a cardioprotective protein that alleviates ischemia/reperfusion injury and mediates the late phase of preconditioningTargeted deletion of the A3 adenosine receptor confers resistance to myocardial ischemic injury and does not prevent early preconditioningMicro-ultrasound for preclinical imaging.Gene dosage-dependent effects of cardiac-specific overexpression of the A3 adenosine receptor.The late phase of ischemic preconditioning is abrogated by targeted disruption of the inducible NO synthase gene.Establishment of a novel murine model of ischemic cardiomyopathy with multiple diffuse coronary lesions.Toll-like receptor 4 deficiency: smaller infarcts, but no gain in function.Molecular identification and functional characterization of a mitochondrial sulfonylurea receptor 2 splice variant generated by intraexonic splicing.Peptide-based antibodies against glutathione-binding domains suppress superoxide production mediated by mitochondrial complex I.Disrupting KATP channels diminishes the estrogen-mediated protection in female mutant mice during ischemia-reperfusion.Peripheral nociception associated with surgical incision elicits remote nonischemic cardioprotection via neurogenic activation of protein kinase C signalingEndothelial NOS activity and myocardial oxygen metabolism define the salvageable ischemic time window for ischemic postconditioning.Impairment of pH gradient and membrane potential mediates redox dysfunction in the mitochondria of the post-ischemic heart.An essential role of the JAK-STAT pathway in ischemic preconditioning.Opposing cardioprotective actions and parallel hypertrophic effects of delta PKC and epsilon PKC.Phase I clinical trial for the feasibility and safety of remote ischemic conditioning for aneurysmal subarachnoid hemorrhage.A novel and efficient model of coronary artery ligation and myocardial infarction in the mouse.The NHLBI-sponsored Consortium for preclinicAl assESsment of cARdioprotective therapies (CAESAR): a new paradigm for rigorous, accurate, and reproducible evaluation of putative infarct-sparing interventions in mice, rabbits, and pigsEndoplasmic reticulum stress-dependent activation of ATF3 mediates the late phase of ischemic preconditioningProbenecid as a noninjurious positive inotrope in an ischemic heart disease murine model.Neuroglobin-overexpressing transgenic mice are resistant to cerebral and myocardial ischemia.Aldose reductase decreases endoplasmic reticulum stress in ischemic hearts.Administration of a CO-releasing molecule induces late preconditioning against myocardial infarction.Late preconditioning induced by NO donors, adenosine A1 receptor agonists, and delta1-opioid receptor agonists is mediated by iNOSAdministration of a CO-releasing molecule at the time of reperfusion reduces infarct size in vivo.Mechanism of cyclooxygenase-2 upregulation in late preconditioningCharacterization of a critical role for CFTR chloride channels in cardioprotection against ischemia/reperfusion injury.Endothelial nitric oxide synthase plays an obligatory role in the late phase of ischemic preconditioning by activating the protein kinase C epsilon p44/42 mitogen-activated protein kinase pSer-signal transducers and activators of transcription1/3 paLoss of the miR-144/451 cluster impairs ischaemic preconditioning-mediated cardioprotection by targeting Rac-1.The late phase of ischemic preconditioning induces a prosurvival genetic program that results in marked attenuation of apoptosis.A novel role for mitochondrial sphingosine-1-phosphate produced by sphingosine kinase-2 in PTP-mediated cell survival during cardioprotection.Inhibition of ischemic cardiomyocyte apoptosis through targeted ablation of Bnip3 restrains postinfarction remodeling in mice.Novel cardioprotective strategy combining three different preconditioning methods to prevent ischemia/reperfusion injury in aged hearts in an improved rabbit model.The COX-2/PGI2 receptor axis plays an obligatory role in mediating the cardioprotection conferred by the late phase of ischemic preconditioning.Mesencephalic astrocyte-derived neurotrophic factor protects the heart from ischemic damage and is selectively secreted upon sarco/endoplasmic reticulum calcium depletionThe ClC-3 chloride channels in cardiovascular diseaseCardiomyocyte-restricted overexpression of extracellular superoxide dismutase increases nitric oxide bioavailability and reduces infarct size after ischemia/reperfusion.Roles of the nitric oxide signaling pathway in cardiac ischemic preconditioning against myocardial ischemia-reperfusion injury.Stromal cell derived factor-1 alpha confers protection against myocardial ischemia/reperfusion injury: role of the cardiac stromal cell derived factor-1 alpha CXCR4 axis.
P2860
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P2860
Demonstration of an early and a late phase of ischemic preconditioning in mice.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
1998年论文
@zh
1998年论文
@zh-cn
name
Demonstration of an early and a late phase of ischemic preconditioning in mice.
@en
type
label
Demonstration of an early and a late phase of ischemic preconditioning in mice.
@en
prefLabel
Demonstration of an early and a late phase of ischemic preconditioning in mice.
@en
P2093
P2860
P1476
Demonstration of an early and a late phase of ischemic preconditioning in mice.
@en
P2093
P2860
P304
P407
P433
P577
1998-10-01T00:00:00Z